You can click on the lower right hand corner
of the video and watch this full screen as it’s HD.
You can click on the lower right hand corner
of the video and watch this full screen as it’s HD.
Unnoticed by the people of Britain, a transformation has been happening beneath our feet. In the first study of its kind, scientists have analyzed the soil the country depends on.
In just the top 8cm (3in) of dirt, soil scientists estimate there are 12.8 quadrillion (12,800 million million) living organisms, weighing 10m tons, and, incredibly, that the number of these invertebrates – some just a hair’s breadth across – which in effect make the soil has increased by nearly 50% in a decade. At the same time, however, the diversity of life in the earth appears to have reduced.
The most likely reason for both the increase in numbers and the decrease in types is the rise of annual temperatures and rainfall over the decade of the study, leading to warmer, wetter summers, said Professor Bridget Emmett, of the UK’s Centre for Ecology and Hydrology (CEH), who led the study. The scientists’ theory is that the warmer, wetter soil encourages most of the bugs to breed faster or for longer, but that more marginal species have been unable to adapt to the new conditions.
They are less certain, however, about whether the changes are a threat or a boon: soil has a relatively high "species redundancy", so there are many species that can do the same job, but all creatures are facing an onslaught of changes such as global warming, pollution and habitat destruction.
In what is thought to be the first national analysis of change in soil bug numbers and types, Emmett’s team extrapolated that there were 1.28 x 10 to the power of 16 individual invertebrates, mainly made up of Oligochaetes (small worms), Collembola (springtails) and Acari (mites).
I got news for them, soil isn’t the only thing with increasing bug counts ….
Internet Price: $29.99
By The Fruit Fly Company
The information above was from this LINK on PETCO and as you see it says they no longer carry the product. However, see this cached page from PETCO … which shows they were selling these springtails only a few days ago? It’s strange that the cached page above is from November of this year and now PETCO no longer even carries the product? I wonder if someone opened up a jar and got more than they bargained for …
I can’t even find this product on the http://www.thefruitflycompany.com/ site either even though that is the website listed on the jar? Perhaps it was just to hard to cultivate them, we’ll probably never know.
I received permission from Dr. Omar M. Amin to republish this article. In some of my future blog posts I will be revisiting the subject of toxicity again. I have made reference to NCS in several posts in the past especially in connection with Toluene and other compounds.
Omar M. Amin, B.Sc., M.Sc., Ph.D.*
*Parasitology Center, Inc., 11445 E. Via Linda, # 2-419, Scottsdale, AZ 85259-2638 USA
Phone: 480-767-2522; Fax: 480-767-5855; E-mail: OmarAmin@aol.com
Web address: http://www.parasitetesting.com/
Neurocutaneous syndrome (NCS), a newly discovered toxicity disorder, is characterized by neurological sensations, pain, depleted energy and memory loss as well as itchy cutaneous lesions which may invite various opportunistic infections. Components in the calcium hydroxide dental sealants Dycal, Life and Sealapex have been identified as sources of the observed symptoms. Sulfonamide and neurological toxicity issues are discussed and three case histories are presented. Additional notes on zinc oxide, Fynal, IRM and Sultan U/P sealers are also included. Diagnostic and management protocols at the Parasitology Center, Inc. (PCI) are proposed.
The original description of the neurocutaneous syndrome (NCS)1 was introductory in nature.1 Examination of many NCS patients and a careful study of their symptoms, exposures, clinical conditions and histories made it possible to identify the underlying cause of the syndrome and proceed with its management.
Patients were personally evaluated and their clinical history, records, symptomology and exposures carefully examined. Specimens provided or collected at the Parasitology Center, Inc. (PCI) were studied. An NCS status was only determined based on symptoms and determination that one or more of the suspect sealers have been used on prior dates. Sensitivity to sulfa and elevated levels of sulfa in the blood were used as a confirmation of sulfonamide toxicity. Continuing patients follow our recommendations for dental rehabilitation, extraction of suspect liner(s), and replacement with ethyltoluene sulfonomide (ETS) and zinc oxide free sealants. A list of vitamin/mineral supplements for patient use during the transitional period and another list of substitute sealants are provided. Patients are followed up to monitor and insure the resolution of symptoms.
The disorder is double faceted with dermatological and neurological symptoms compatible with classical sulfa toxicity. The latter is characterized by changes in blood values, photosensitive reactions, allergic vasculitis sores, bacterial flora changes, and redness of the skin, which may lead to liver and kidney failure.2 The neurological aspects are characterized by pin-prick and/or creeping, painful and irritating movement sensations, often interpreted as parasite movements in various body tissues and/or cavities.. Movement sensations are either unipolar or bipolar and may proceed horizontally or vertically. They may manifest as variably shaped bruises or waves of elevated ripples or channels. In no case was the movement sensation related to parasites1. Neurological symptoms may also include loss of memory, brain fog, lack of concentration and control of voluntary movements.
Fig. 1. Early NCS sores on the thigh of KM. She was born in 1964, treated with Dycal in two teeth in 1982 and in one tooth in 2002. Neurological symptoms in upper quadrant started in 1997. Cutaneous symptoms began in Spring 2002 preceded by extensive treatment with topical sulfa preparations for possible mite infestation. Dycal was removed in December, 2002 and recovery is in progress.
The cutaneous aspects include small itchy sores (Fig.1), inflamed often elevated pimples (Figs.2,3), and fully inflamed and painful open/amorphous mucoid lesions that often enlarge and coalese (Fig.4). Histopathological sections of lesions (Fig.5) show superficial and deep perivascular infiltrate of lymphocytes, accompanied by interstitial deposits of granular mucin material. Eosonophils are usually present within the inflammatory infiltrate and foci of epidermolytic hyporkeratosis are often identified within the epidermis (Fig.5). Lesions may also be on the scalp where they may be associated with infestation of springtails (Collembola). 1 In many cases, lesions are associated with edematous reaction usually in the arms and legs (Fig.6). Blood vessels may also become enlarged and elevated, and head may become hot and turn red. The gum tissue and the teeth and oral mucoid secretions may turn gray and become compromised first and stay compromised the longest. The above creeping sensation is clearly distinguished from these caused by nematodes such as Toxocara canis3 or Dioctophyme sp.4
General symptoms usually include fatigue, compromised immune system, psychological trauma and loss of self- esteem. The depressed immune status in most patients appears to pre-empt them for opportunistic infections.
Fig. 2. Elevated sores on the forehead of KM (Fig.1); note the hot red color of the skin.
Fig. 3. Diffuse NCS sores covering the whole body that was treated with Dycal in 1985 (Case no. 1)
While NCS itself is not a contagious condition, superimposed opportunistic infections on open sores may be. Initial infection with fungus or bacteria appear to attract subsequent infestations with many arthropod species, especially springtails (Collembola: Insecta).1,5,6,7 Black specks associated with such infections appear to be metabolic waste (fecal elements) of these organisms or mycelial masses of certain fungal species. Staphylococcus aureus, S. haemolyticus, Streptomyces spp., Candida albicans and Madurella spp. among others, have been identified from cultured swabs taken from sores of various NCS patients. These opportunistic infections have been shown to aggravate the cutaneous symptoms of NCS patients. The Madurella infections are usually associated with black grains of mycelial masses that may be related to the black specks and fibers observed by some NCS patients. The healing of certain patients lesions9 was observed to be proportional to the exit of remaining fibers from lesions.3 Patients experiencing complete remission remain susceptible to fungal promoting conditions in damp, shaded, moldy places.
Arthropods identified from sores include fleas, caterpillars, wasps, ants, beetles, winged flies, midges, thrips, ticks, mites, spiders, and springtails.1,4 Springtails may have close association with sores in many NCS patients but they, and other opportunistic infections, are not causal factors of NCS sores.
The three major calcium hydroxide sealants causing NCS (Dycal, Life and Sealapex) considered 9 include only about 50% calcium hydroxide in the catalyst (Table1). Of the components common to all three sealants, ethyltoluene sulfonamide as well as zinc oxide are considered most toxic. Toluene is a known potent nerve toxin.10 The sulfonamide component of this compound causes a sensitivity allergic- toxic reaction ultimately manifesting as the vascular mucoid sores characteristic of the NCS, especially in sulfa sensitive patients.
Fig. 4. Mucoid NCS/lesions on the face of MM. She was born in 1950, poisoned with Fynal in six teeth in 1981 and in one tooth in 1986 as well as with Life in two teeth in 1985 and 1988.
Fig. 5. Histopathological section of one of the roughly 300 sores covering the body of SK. She was born in 1956 and reacted with typical NCS symptoms to a zinc oxide cement (combined with Durelon) underneath a total veneer job in 1982. The section shows hyperkeratosis like perivascular dermatitis with eosinophils.
Fig. 6. Cutaneous sores and swelling in the right hand and arm of DB. Born in 1965, DB had 10 amalgam restorations in 1982 and 1983 using Life. She started experiencing symptoms including ulcerated rash all over the body, unilateral edema and pin-prick and subcutaneous movement sensations in 2001-2002. Life is being removed and recovery is in progress.
Zinc oxide was shown to be genotoxic11, cytotoxic12,13, killing microphages14, and causing chronic and fibrous inflammatory reaction15,16 ulcerations16 and osteosclerosis.17 Additionally, the toxic effects of zinc oxide and calcium hydroxide were shown to be similar.18,19 Calcium hydroxide was shown to cause periapical inflammation, typical granuloma and partial lack of healing.20 Titanium dioxide and Barium ions (Table 1) were also shown to provoke strong foreign body and bio-incompatible reactions in live tissue.21,22
Cytotoxicity of Dycal, Life and Sealapex was clearly demonstrated invivo and invitro in various tissues.23 Sealapex was shown to cause severe inflammatory infiltration15,24,25 and edema25 accompanied by subcutaneous tissue necrosis15,26 and progressive differentiation and reaction of monocytes, macrophages and epithelial cells27. The final phase of the inflammation is characterized by an intense granulomatus reaction especially in epithelial cells causing various intensities of irritation.28The cytotoxicity29,30 and neurotoxicity31 of Sealapex was well demonstrated in various mammalian systems.
As with Sealapex, Dycal was also shown to cause hemorrhage and acute to consistent inflammatory cells16,32,33 necrosis,16,32,33 tissue loss,33 karyorrhexis,16 neurotoxicity.34 and formation of serous exudates.16 Life has been the least researched sealant. It, however, has the same toxic ingredients, i.e., ethyltoleune sulfonamide and zinc oxide, as Sealapex and Dycal and has been associated with classical NCS symptoms in some of our patients, e.g., DB (Fig.6) and MM (Fig.4).
Sealants not containing ethyltoluene sulfonamide but including zinc oxide and eugenol have also been associated with NCS cases.These include Fynal(>75% zinc oxide), IRM and Sultan U/P (<50% zinc oxide). Fynal was associated with the cases of MM (Fig.4). Similarly, IRM (by Dentsply caulk) and Sultan U/P (by Sultan Chemists) were associated with classical NCS symptoms in some of our patients.
A white female born in 1951. In 1985 she underwent dental repairs, which included the use of Dycal in 20 teeth. The lady is allergic to sulfonomides, with IGE values reaching 5000. Every dental treatment was followed by aggressive skin reactions of allergic and toxicological nature (Fig.3). All tests for parasites were negative. Her symptoms fulminated into full blown typical sulfa toxicity reactions including oozing skin and nasal sores with bloody scabs and smelly discharge and an infection with S. aureus ( Fig.7). Other symptoms included loss of memory, kidney pain and urgency, sensitivity to light and electricity fields, pin-prick and moving sensations under the skin, and swelling. After each treatment, the white female felt totally knocked out with breathing and talking difficulties. She subsequently developed intestinal problems and her skin sores flared up with unbearable and unresolved itching. Photosensitive reactions presented as blotchy skin ( Fig.7) with severe burning sensations in the face, throat and chest.
Dycal was removed in 1991-1992 and initially replaced with Harvard cement. The lady was confined to bed with whole body musculo-skeletal system pain, bowel disturbances and signs of polyneuropathy. Shortly after the removal of the Dycal in February 1992, most of her sores and rashes disappeared and she could tolerate sunlight (Fig.8).
Case # 2.
Born in Chicago in 1965, JM was a healthy active Caucasian woman until she started experiencing her first symptoms in 1991. By then, she already had 17 fillings. No sealants were used in one filling; Dycal was used in the other 16. Her earliest symptoms appeared as skin break outs on the face and neck, which was recurrent over the following 9 years, accompanied by body tremors, sleeplessness and joint pain with occasional vomiting of black bile. Thrush appeared in the mouth and around the lips. Pain at the teeth roots persisted throughout the nineties associated with rapid major decay. A sensation of prickling pain with a pressure and movement under the skin, urticaria and skin ulcerations would last for weeks or months. JMs body showed random swelling with red marks in serpentine-like shapes. The swellings eventually bottlenecked at the knees and ankles. The chest burned and hurt with strange fits of coughing. JM then started losing hair as she experienced night fevers and sweats, and peeling of the skin.
During the early 1990s JM was medicated with various antibiotics, antiparasitics and herbal remedies. She experienced some anti-inflammatory relief and occasional temporary clearing of ulcers after which ulcers returned and lasted longer. In 1998, massive ulcers appeared on JMs face at the nasiolobial area and on the skin ( Fig.9). A CBC in 1999 was unremarkable except for a high level of Alpha 1- Globulin of 0.5 (Normal range 0.2-0.4) and low levels of IgA of 99 (normal range 60-400) and IgG of 724(normal range 700-1500). The right ocular cavity was severely painful and JM was beginning to lose her eyesight.
A major dental repair was completed in 2001 when Dycal was removed from all 16 teeth. Initially, JM experienced a few episodes of sickness, sweats, and vomiting. After the fourth visit, her eyebrow area had a dramatic reduction in swelling, sensation of movement and in the red-hot congestion of her face. JMs teeth were subsequently rebuilt with gold onlays section by section. By the end of the total repair, Nov.2001, JM has regained her normal skin (Fig.10) with no movement sensations or pain anywhere in her body. This state of total resolution has lasted to date without regression or relapses.
Fig. 9. Case no. 2 (JM) before treatment; note the lesion
Fig. 10. JM after recovery.
LG, a medium- built white American born in 1957, was in perfect health until September 18, 1998 when she had a filling in her tooth no. 18 using Dycal as a liner. She experienced severe headache within 2 hours. By 6:00 pm she was vomiting and delirious with the headache persisting. Her blood pressure then was monitored at 169/108 and remained high for the following three years despite repeated attempts to control it with Atenenol and Diazide. LG never experienced high blood pressure or headaches before. An MRI scan was negative. In 1999 LGs health deteriorated progressively with arthritis- like symptoms in her back, heart palpitations, mitral valve prolapse, fatigue, abnormal pap-smears including pre-cancerous cell abnormalities, night sweats, missed periods, and severe depression. By March 2001, LG, who normally weighed 120 lbs has lost 20 lbs.
In April 2001 lesions started appearing on LGs face, which quickly became red-hot. Her legs became swollen and painfully burning. By May 2001, LG had several open lesions (6 mm to 2 cm in diameter) with some surrounding erythema, on her face and scalp. Her cheek pulsated as the facial lesions seemed to track to the chin (Fig.11) where the most fulminating lesion was; nearest to her teeth. The face was burning hot. Springtails (Collembola) and fibers were recovered from these sites. At that time, she showed low lymphocytes of 15.0% (normal 20-43%), high granulocytes of 77.1% (normal 51-74%) and high rheumatoid factor of 22.6 (normal <20 IU/ml). She also tested negative for all communicable diseases then. Her weight dropped to 92 lbs as she started experiencing movement sensations under the skin of her arms, face and scalp. Grayish pustular secretions oozed and moved down from the bloody lesions on the scalp and face. The lesion then extended to her legs.
In January 2002, LG was diagnosed with NCS by OMA. She was allergic to sulfa and sulfonamide compounds. Following our protocol, LG had the filling and the Dycal liner removed from tooth #18 in April 2002. These were replaced with Starflow and Aria (a combination of Bisgma, Tegdma, Lidma and catalysts). Our recommended vitamin supplementation program was initiated then. By May 2002, all symptoms were resolved (Fig.12). Constitutional and neurological functions as well as psychological, emotional and energy levels were restored to normalcy.
Fig. 11. Case no. 3 (LG) before treatment.
Fig. 12. LG after recovery; note the return of the natural
The toxicity of Dycal, Life and Sealapex has been well demonstrated in invivo and invitro studies of various animal and human models by many workers. The toxicity assumed cytotoxic, genotoxic, neurotoxic, phototoxic, necrotic, and inflammatory manifestations compatible with the pathology and symptoms observed in NCS patients. Ethyltoluene sulfonamide, common to all three sealants, is considered the primary cause of the NCS. The toluene component, a known nerve toxin, is believed to be responsible, at least in part, for the neurological symptoms. Neurological abnormalities are related to nerve damage associated with vasomotoric reactions due to a direct influence on the peripheral nerve endings.35 The sulfonamide component is the cause of the cutaneous symptoms, especially in sulfa-sensitive patients who usually had elevated sulfonamide/sulfa levels in blood tests and allergy to sulfa in skin sensitivity tests. The relationship between sulfonamide and phototoxicity has been well established.29 Resolving the symptoms (effect) by removing the sealants (cause) in patients undergoing treatments, confirms this cause-effect relationship.
The nature of causation of NCS precludes contagious transmission. Any similarities of symptoms among partners within the same household are traceable to the transmission of opportunistic infections, especially fungi.
It is recommended not to rehabilitate more than two or three teeth per month. The patient is given a list of vitamins and other supplements to take during the procedure and for the following few weeks until symptoms are completely resolved. After reaching the state of normalcy, the patient may still retain some sensitivity to moldy places lacking sun and fresh air circulation.
After additional test results become available and a satisfactory diagnosis of an NCS case is made at the Parasitology Center, Inc. (PCI), arrangements for dental rehabilitation are made and patient prognosis is monitored.
I am grateful to Marie Erixon, Nordea, Sweden for her contributions to the better understanding of issues related to NCS.
1. Amin OM. Neuro-cutaneous Syndrome (NCS); a new disorder. Explore 2001; 10: 55-56.
2. Ockert K. Filling caused serious reactions. Trandlakartidningen 1994; 86: 470. (in Swedish).
3. Garcia LS. Diagnostic Medical Parasitology. Wash, DC: Am Soc Microbiol Press, 2001.
4. Urano Z, Hasegawa H, Katsumata T, Toriyama K, Aoki Y. Dioctophymatid nematode larva found from human skin with creeping eruption. J Parasitol 2001;87: 462-465.
5. Amin OM. Facial cutaneous dermatitis associated with arthropod presence. Explore 1996; 7: 62-64.
6. Frye FL. In search for the haphazardly elusive: a follow-up report on an investigation into the possible role of collembolans in human dermatitis. Vet Invert Soc Newsletter 1997; 13: 10-13.
7. Janssens F. Checklist of the Collembola: Collembola in association with man. http//www.collembola.org/publicat/sidney.htm 1999-2003; 10pp, and per comm..
8. Mahon CR, Manuselis G Jr. Diagnostic Microbiology. Philadelphia: WB Saunders Co, 1995.
9. Draheim RN, Murray AJ. Compressive strength of two calcium hydroxide bases. J Prothet Dent 1985; 54: 365-366.
10. Burry MB. Neurodevelopmental toxicity of toluene.M. Sc. Thesis: Seattle, Univ Wash, 2001.
11. Tai KW, Huang FM, Huang MS, Chang YC. Assessment of the genotoxicity of resin and zinc-oxide eugenol-based root canal sealers, using an in vitro mammalian test system. J Biomed Mater Res 2002; 59: 73-77.
12. Wright KJ, Barbosa SV, Araki K, Spangberg LS. In vitro antimicrobial cytotoxic effects of Kri 1 paste and zinc oxide eugenol used in primary tooth pulpectomies. Pediatr Dent 1994; 16: 102-106.
13. Pissiotis E, Spangberg LS. Toxicity of pulpisad using four different cell types. Int Endod J 1991; 24: 249-257.
14. Sadeghein A, Bolhari B, Sarafnejad A. A comparison of the effects of three endodontic sealers on adherence of mouse peritoneal macrophages. J Calif Den Assoc 2001; 29: 673-677.
15. Soares I, Goldberg F, Massone EJ, Soares IM. Periapical tissue response to two calcium hydroxide-containing endodontic sealers. J Endod 1990; 16: 166-169.
16. McShane CJ, Stimson PG, Bugg JL, Jennings RE. Tissue reactions to Dycal. J Dent Childr 1970; 37: 466-474.
17. Erausquin J. Periapical tissue reaction to root canal fillings with zinc, titanium, lead, and aluminum oxides. Oral Surg Oral Med Oral Pathol 1970; 30: 545-554.
18. Berman DS. Pulpal healing following experimental pulpotomy. Brit Dent J 1958; 105: 7-16.
19. Berman DS, Massler M. Experimental pulpotomies in rat molars. J Dent Res 1958; 37: 229-242.
20. Weinstein R, Goldman M. Apical hard-tissue deposition in adult teeth of monkeys with use of calcium hydroxide. Oral Surg Oral Med Oral Pathol 1977; 43: 627-630.
21. Bennatti-netto C, Bramante CM, Ber-Bert A, Lia RCC. Reacao do tecido conjuntivo subcutaneo de rato ante a implantacao dos materials components do cimento AH-26. Rev Bras Odontol 1982; 39: 11-20.
22. Smith JW, Leeb IJ, Torney DL. A comparison of calcium hydroxide and barium hydroxide as agents for inducing apical closure. J Endod 1984; 10: 64-70.
23. Topalian M. Effecto Citotoxico de los cementos selladores utilizados en endodoncia sobre et Tejido periapical. Endodocia-Caracas 2002, http://www.carlosboveda.com; 48pp, and per comm.
24. Buntak-Kobler D, Prpic-Mehicic, Najzar-Fleger D, Katunaric M, Talan-Hranilovic J, Suman L. Cytotoxicity of Ca(OH)2 endodontic sealers on connective, muscle and bone tissues. Acta Stomatol Croat 1993; 27: 175-180.
25. Sonat B, Dalat D, Gunhan O. Periapical tissue reaction to root fillings with Sealapex. Intern Endod J 1990; 23: 46-52.
26. Bezerra LA, Leonardo MR, Faccioli MR, Faccioli LH, Figueiredo F. Inflammatory response to calcium hydroxide based root canal sealers. J Endod 1997; 23: 86-90.
27. Tronstad L, Barnett F, Flax M. Solubility and biocompatibility of calcium hydroxide-containing root canal sealers. Endod Dent Traumatol 1988; 4: 152-159.
28. Zmener O, Guglielmotti MB, Cabrini RL. Biocompatibility of two calcium hydroxide-based endodontic sealers: a quantitative study in the subcutaneous connective tissue of the rat. J Endod 1988; 14: 229-232.
29. Beltes B, Koulaouzidou E, Kotoula V, Kortsaris AH. In vitro evaluation of the cytotoxicity of calcium hydroxide-based root canal sealers. Endod Dent Traumatol 1995; 11: 245-249.
30. Guertsen W, Leinenbach F, Krage T, Leyhausen G. Cytotoxicity of four root canal sealers in permanent 3T3 cells and primary human periodontal ligament fibroblast cultures. Oral Surg Oral Med Oral Pathol Oral Radiol 1998; 85: 592-597.
31. Serper A, Ucer O, Onur R, Etikan I. Comparative neurotoxic effects of root canal filling materials on rat sciatic nerve. J Endod 1998; 24: 592-594.
32. Good DL. Effects of materials used in pediatric dentistry on the pulp: a review of the literature. J Calif Dent Assoc 1999; 27: 861-867.
33. Heys DR, Heys RJ, Cox CF, Avery JK. The response of four calcium hydroxides on monkey pulp. J Oral Pathol 1980; 9: 372-379.
34. Norrsells N. Aven svenska tandlakare tillats nu sedan EU-intradet att anvanda den effektiva N2-metoden for rotfyllig. Med denna metod kan 500 miljoner kr sparas arligen at patientena och lidandet minskas. Endod Sverige 2002; 5p.
35. Hensten-Pettersen A. Skin and mucosal reactions associated with dental materials. Eur J Oral Sci 1998; 106: 707-712.
See also: Amin, O. M. 2004. Dental Sealant Toxicity: Neurocutaneous Syndrome (NCS), a dermatological and neurological disorder. Holistic Dental Association Journal (No. 1, Jan.): 1-15 http://www.holisticdental.org/.
See also: Amin, O. M. 2004. On the diagnosis and management of neurocutaneous syndrome, a toxicity disorder from dental sealants. California Dental Association Journal 32 (9): 657-663.
This is taken from a comment left by Rob on my Morgellons and More Cases of Collembola Infesting Man post.
Quote from Mr. Common Sense …
I worked at a certain company when I came down with this, I believe at least 15 or so people had it there (by the time I realized what I had). When I first got this real bad I sat in a conference room chair (30 year old cloth chairs) and the crawling was unbelievable, I didn’t know what to to do so I just sat there. I was just learning of my condition at that time. What ever was causing the crawling sensation was definitely in the chair, but others had sat there time after time and felt nothing.
Now, I still know some of those people as I have moved on from that place. When we meet and shake hands my hands burn, itch, crawl for at least 3 or 4 days maybe more and it will move on to my entire body.
When I read this . I had to read it 2 or 3 times. This was exactly what I Experience. About 2 years ago I came down with this Unknown Burning on the back of the hands and sometimes feet Pin pricks itch, and crawling experience. It was a Horrible time. It lasted about 6 months after trying everything from tea tree oil. to Anti parasite herbs. Cleaning my whole house out. After some time it went away. I thought I got it from a friend. She was a Backpacker from Japan. When she went back to Japan she left some clothes and books in a cupboard . I never touched them until last week when I decided to throw them out . That’s when it all started again. First i noticed a burning of the back of the hands and then pin pricks all over my body again. I thought OH NO here we go again. I cant believe that they can live off books and clothes for nearly 2 years. I don’t want to go through this all over again.
This has been my experience too. There is something, some external component to this that is responsible for most of the crawling and biting. However, and let me be clear on this, this is only happening to us because of the devastating process going on internally which is very real and serious, Morgellons is not external, it is internal. Now, even years removed from that company there are people that I still know that if I get around or they physically touch me in any way the crawling will start back up (yet they feel nothing). But now I can quickly beat it back, probably by that very same evening. Some of these folks you can tell are annoyed by something on their skin but they haven’t a clue what is going on, but many don’t feel this yet they carry whatever it is or cause the reaction in me for whatever reason. I know I didn’t give Morgellons to them, some had it bad long before I got there (one even had big lesions). Also, this happens to me when I get around pets (sorry pet owners) as I think they are major carriers of what causes the crawling and biting sensations. However, let’s examine what might be causing this.
First, Rob’s comments are in no way novel. I have heard this story so many times it should not be news to you. The forums are full of such reports. Scabies? In Rob’s case, no way, in 4 days Scabies die off the host. To handle clothes that have been stored for months and years and then become “re-infected” as it were is not possible with Scabies. This has happened to me too, that is, bringing out old clothing and getting the crawlies back. As fall approaches and I begin to rotate in new clothing you can bet they will be washed at least twice with Dr. Bonners before I wear them. No, this is definitely not scabies.
I think this (not Morgellons, but the crawling sensation) could only be one of two things.
It’s either an Arthropod such as a mite or Collembola (springtail) or an extreme allergy to Arthropods. When a person first gets Scabies it could take a month or two before they actually begin to notice them. And believe it or not, a person will usually only have a few dozen at most on their body (though it can feel like millions on you). However, once free of them if a person becomes re-infected they will usually notice the re-infection immediately because their body now reacts so negatively to the presence of Scabies, a histamine reaction of sorts. This is the same thing that is happening to us, only it cannot be Scabies in our case they would not live off the host for months and even years at a time.
Could it be a fungus? Well, in my opinion I find it hard to believe that fungus living on stored clothing that has been washed could be the culprit, but Arthropods have been proven to survive washing cycles, and even drying cycles.
Did you know still today, by far, the #1 search from Google and other search engines that bring folks to this blog are searches regarding Collembola infestations of humans? That’s right, people everywhere seem to be searching on this.
Here’s how I think I eliminated the crawling from my life (though, as I have stated, I can still get the feeling back but it’s usually gone within 24 hours).
I live out of a single laundry basket, wash my clothes in Dr. Bonners Peppermint Soap and shower with the Sulfur Soap which is a powerful and known Arthropod killer. The first time I ever experience a crawling free day was using Trisha’s soaps, especially the green stuff you rub on after the shower. I think it literally gummed up the Arthropods such that they couldn’t move. However, also very effective is the sulfur soap on my Poor Mans Protocol. Key to this though long term is correcting your terrain. Did you know an acidic PH lies behind most allergies?
When I did have this really bad and worked at the company where I know for a fact others also had this very badly they only had to get within say three feet of me and I would feel a sort of “extreme crawling or electrical feeling” like I was standing near a high voltage generator. It was unmistakable and very real. Then, when I left that company and went to another company I noticed that upon entering a conference room everyone almost immediately (when I was within their line of site – came around the corner into the room) reached up and itched their noses. This happened to at least 50% of those in the room and I could be 20 feet from them. This has been reported by others many times as well. I remember a guy on LymeBusters who had written on a piece of paper “You will be itching your nose” and showed it to every doctor he went to see. This would freak them out because it always happened. I always wondered what could travel so fast from me to them that could cause this?
This of course no longer happens for me. I was horrified to go to the weekly status meeting, the effect I had on the room was profound but nobody had a clue what was going on.
Just a side note, whenever I go into a Wallmart I get the same feeling I get from being around animals, the crawling starts right up, then home for a quick shower with my sulfur soap and all is well. I have a good idea what is going on here, but will save that for another post. I am curious to hear your comments, especially if you have experienced similar things.
These photos were taken by a follow Morgellons sufferer after undergoing Chemo/Radiation treatment around the date of June 30, 2009. She is a dear person to me and well known in the Morgellons community. Here is a quote from her regarding the posting her photos (which she gave me permission to use in this post). I’m merely going to post them here so they never go away, but trust me, we are coming back to this topic in a later post. One of the springtails (Collembola) even lived for an hour after coming out of her scalp.
Please go have a look, I was so shocked to have so many come out of my hair because I don’t even feel movement there anymore. I also have several lesions on my head after my hair came out.
No doubt you can make out the spring tails (Collembola) and what appear to be fungal gnats of some sort. Again, I am only posting these for posterity but trust me, we are coming back to this subject.
Our prayers are you dear sufferer ….
First, I would like to thank Sidney for providing a link to this information. Sidney, you’ve been a big help with my research, thank you. I’m merely going to copy the contents of two articles in here in case they are lost to time. This way we will always have a copy. The 2nd article is an incredibly large report that was posted in six segments in the comments section of the end of the first report, I find it very interesting, so be sure to scroll through this entire post. There is a reason these soil based pests are in us folks, I’m getting to that in another post.
This information was taken from:
Frederic L. Frye, DVM, MSc, CBiol, FIBiol,
It has been known for over a century that springtail insects (Apterygota) can live freely or in association with a variety of biological systems that include terrestrial, freshwater- and shoreline-marine aquatic. Once believed to be entirely saprophagous and/or phytophagous facultative (or obligate) detritovores, springtails have been found in the following circumstances: feeding upon nesting ants, flies, and conspecific dead springtails (Dasgupta and Dasgupta, 1995); infesting bumblebees (Husband and Brown, 1976); infesting insect eggs (Tosi, 1977; Tosi and Sartini, 1983; Waldorf 1971); parasitizing cabbage moth larvae (Griffiths, 1985); infesting hermit crabs (Van Goetham and Hanssens, 1982); living in the gut of an avian ectoparasitic insect, Myrsidea sp., which is a Mallophagan louse (Oniki and Butler, 1990); parasitizing salamanders and toads (Dasgupta and Dasgupta, 1990); associated with nesting mammals (Dasgupta and Dasgupta, 1995; Hrivnak, 1981; 1983; Nosek and Vysotskaya, 1976; Roy and Dasgupta, 1971; Szabo, 1977); and attracted to and actively feeding on moist blood films from red pandas, Ailurus fulgens, (Dasgupta and Dasgupta, 1995). There have been a few reports of springtails infesting humans (Dasgupta and Dasgupta, 1995; Hunter, Frye and Schwartzwelder, 1960; Scott, Wiseman, and Stojanovich, 1962). Collembola can utilize phoresy as a means for transporting themselves from one habitat (or host) to another, particularly from birdnest to birdnest (Oniki and Butler, 1990). Thus, mounting evidence supports the contention that, although they usually are thought to be merely ubiquitous and harmless saprophytes, springtails can alter their behavior and become parasitic without undergoing evolutionary structural alterations to their feeding anatomy. While not limited to springtails, a change in fundamental feeding behavior is unusual and noteworthy.
Recently, I was contacted by an official of the National Pediculosis Society who was investigating the possibility that all members of a family that was living in Iowa and suffering from very sever and intensely pruritic mutifocal head, trunk, and pubic dermatitis were infested with parasitic collembolan (springtail) insects. After extensive diagnosis and treatment of their intese pruritis, each was told that he/she was suffering from "delusional parasitosis" and was referred for psychiatric evaluation. The adult female in the family is a registered obstetrical nurse and perceived that these lesions could be attributed to an organic rather than a psychological aetiology. She then commenced a search for "unconventional" professional assistance to help determine the cause for this condition. Interestingly, the rationale for contacting me may have been erroneous because: (1) although I am the senior author of a paper documenting the parasitism of a dog by human crab lice, Phthirus pubis, (Frye and Furman, 1968), I possess no special knowledge of springtails; and (2) although there is a textbook on tropical medicine edited by someone with an identical surname as mine, I am not the author. However, this approach was serendipitous because I began my veterinary career as a chronic disease epidemiologist and I am now a comparative pathologist with an admittedly ecclectic curiousity.
Affected patients complained of slightly raised rec, black, and/or white lesions that itched, and it felt as if something was crawling beneath the skin. When scraped or rubbed with vinegar or antifungal cream, some areas produced short red or white "fibres" that moved. In a few instances, entire creatures with "scorpion-like tails that curved downward" were teased out. In some instances, the patients reported seeing small, round, white "nit"-like objects adhering to their head hairs.
Routine tests and biopsies were negative for the presence of Herpesviral dermatitis, mite infestation (scabies), and head- and bodylouse infestation (pediculosis). However, the white nit-like objects that adhered to individual head hairs probably were the eggs of headlice. Some skin biopsies revealed the presence of arthropod insects that were characterized as collembolas. Because of their similar small size, it is unlikely that the springtails used the headlice as phoretic transport vehicles to move between hosts. Therefore, it appears that some patients were simultaneously infested with both springtails and headlice.
A literature search revealed an expanding source of information that documents that springtails have become true parasites of a variety of animal hosts even though they lack specialized feeding structures. Once the Centers for Disease Control in Atlanta, Georgia began to investigate pruritic dermatitis of unknown pathogenicity, they found 123 cases. These cases included 122 people from 22 states in the U.S. and 1 person who was thought to have been infested in South Korea; all of these cases occurred from 1990 to 1995. The largest number were from California. Most were originally believed to be instances of scabies or pediculosis and, accordingly, were treated initially with prescription and/or over-the-counter proprietary insecticidal preparations. Most of these cases were traced to (or at least thought to be related to) a variety of outdoor occupations, contacts with domestic and wild animals, gardening activities, exposure to various fungi, etc. Patients ranged from young children to elderly adults. The sex ratio was 59% female:43% male. In one-third of the households, more than one inhabitant was affected by "atypical" pruritic dermatitis for which mites, lice, or other common arthropods could NOT be implicated as the aetiologic agent(s).
Whether the immune status of the affected individuals played a major role in promoting springtail parasitism or whether insecticide resistance was a component in these cases is entirely conjectural; however, both factors have been suggested as possibilities. In all of these cases, the patient’s serological status with respect to HIV infection is unknown because of privacy protection; the liberal use of lindane and other potent organochlorine-containing acaricidal or insecticidal preparations was documented, but their actual role as promoters of immunoincompetence or chemical resistance is unknown.
To the date of this writing, the taxa that have been reported to induce intense pruritic dermatitis in man are: Entomobryo nivalis and E. tenuicauda (Hunter, et al, 1960; Scott, et al, 1962); Hypogastrura menubrialis and Sora buski (Megnin, 1839; Freche and Beille, 1896). In addition, Orchesella albosa var. oinslferi was recorded infesting humans without causing pruritis (Scott, et al, 1962).
In summary, these cases of collemboliasis may be only medical curiousities , or they may be the first "sentinel" instances of a significant emerging parasitism.
Dasgupta, R. and Dasgupta, R. (1990). Collembolan Insects as Potential Parasites. Trans. R. Soc. Trop. Med. Hyg., 84:438.
Dasgupta, R. and Dasgupta, R. (1995). A Treatise on Zoophily in Collembola with a Summary of Knowledge on Origin and Evolution of Parasitism in Saprophagous Forms of Animals. J. Bengal Natural History (New Series) 1(2):53-60.
Freche, L. and Beille, L. (1896). Un Parasitisme Accidentale de L’Homme. Comp. Rend. Acad. Sci. Paris, 73-80.
Griffiths, G.C.D. (1985). Hypogastrurua succinea (Collembola:Hypogastruridae) Dispers by Adults of the Cabbot Maggot, Delia radicum (Diptera:Anthomytidae), Infected with the Parasitic Fungus Strongwellsea castrans (Zygomycetes:Entomophthoracea). Canadian Entomol., 8:1063-1064.
Hrivnak, L. (1981). Qualitative and Quantitative Analyses of Collembolan Fauna in the Nests of Small Mammals in the Hight Tatra Mountains. Biologica, Bratislava, 36(5):363-372.
Hrivnak, L. (1983). Signs of Collembola in the Nests of Small Mammals in the High Tatras and Hyposometric Distribution of Individual Collembolan Species. Biologica, Bratislava, 38(6):555-567.
Hunter, G.W., Frye, W.W. and Schwartzwelder, J.C. (1961). A MANUAL OF TROPICAL MEDICINE). Philadelphia, PA; W.B. Saunders Co. xxx+892 pp.
Husband, R.W. and Brown, T.M. (1976). Insects Associated with Michigan Bumble-Bees (Bombus sp.). Great Lakes Entomol., 91:57-62.
Mitchell, W.G. and Turner, E.C., Jr. (1969). Arhtropod Parasites on the Starling (sturnus vulgaris) in Southwest Virginia. J. Econ. Entomol., 62(1):195-197.
Nosek, J. and Vysotskaya, S.O. (1976). Protura from Nests of Small Mammals in the Trans-Capethian Oblast. Entomol. Obzr., 55(4):808-813.
Oniki, Y, and Butler, J.F. (1989/1990). The Presence of Mites and Insects in the Gut of two Species of Chewing Lice, (Myrsidea sp. Mallophaga). Accident or Predation? Rev. Bras. Biol., 49(4):1013-1016.
Roy, P. and Dasgupta, B. (1971). Behavior of Chrysomys megacephala (Fahr) and Hemispyrellia ligurriens (Wied.) as Parasites of Living Animals under Experimental Conditions. S. Afr. J. Med. Sci., 36:85-91.
Scott, H.G., Wiseman, J.S.,, and Stojanovich, C.J. (1962). Collembola Infesting Man. Ann. Entomological Soc. America, 55(4):428-430.
Szabo, I. (1977). Examination of the Fauna in the Nests of Mammals and Birds in Hungary. Allantani Kozl., Budapest, 64(1-4):153-158.
Tosi, L. (1977). Alimenti Animali Neili Diete di Alcune Species di Collemboli, Richerce Preliminary sul Cannibalismo in Sinella caeca (Schott). Ateneo Paramense. Acta. Nat., 13:445-455.
Tosi, L. and Sartini, M. (1983). Interactions Between Social and Feeding Behaviour in Sinella caecs (Collembola). Boll. Zool. Naples, 50:189-195.
Waldorf, E. (1971). Selective Egg Cannibalism in Sinella curviseta (Collembola, Entomohryidae). Ecology, 52:673-675.
Vol. 53, February, 1951, No. 1.
Unusual Scalp Dermatitis in Humans Caused by the Mite, Dermatophagoides (Acarina, epidermoptidae)
By Jay Traver, Department of Zoology, University of Massachusetts, Amherst
The writer and two other members of her immediate family, all of us adult females, have been for the past several years the unwilling hosts to the mite, Dermatophagoides scheremetewskyi Bogdanow. Since the published reports on this mite as a parasite of humans are not numerous, it seems desirable to present an account of the activities of the mite from first hand information. Symptoms, treatment employed in the attempt to control or eradicate the mite, reaction of certain members of the medical profession to this problem, and the present status of the situation are therefore set forth.
It is to Dr. Edward Baker of the Division of Insect Identification, Bureau of Entomology and Plant Quarantine, Washington, D.C., that we owe the identification of this mite, and the synonymy herewith is presented. It is to him also that the writer owes her contact with Dr. Gaines W. Eddy of the same Bureau in Washington, who has suggested the use of certain new synthetic organic compounds as scabicides in the treatment of this mite. Had it not been for Dr. Baker’s interest in this problem, expressed in personal correspondence, it is quite possible that no attempt would have been made to publish this personal account of an infestation with the mite Dermatophagoides.
Personal Observation of the Activity of the Mite.
Small itching red papules on the scalp were noted as early as 1934. This condition persisted without much change in spite of sporadic efforts to control it, until the spring and early summer of 1943. At this time, the sensations as of some arthropod crawling, scratching and biting became very pronounced, and occurred over wide areas of the scalp. The idea that Pediculus humanus capitus might be the causative agent was not borne out since at no time was it possible to ‘comb out’ a louse nor to locate nits on the hairs. Further, the infestation did not yield to treatment known to be effective against pediculosis. The itching and crawling sensations were most pronounced between 10 p.m. and the early hours of the morning.
By the middle of August, 1943, the annoyance had become excessive and more strenuous efforts were made to clear up the infestation and to locate the causative agent. Three areas of the scalp were principally involved, a space as large as the palm of the hand above and behind each ear, and an even larger area on the top of the head in the frontal region. The sensations as of something biting, scratching, and crawling from place to place were now almost continuous, becoming apparent as early as 10 a.m. and continuing all day and far into the night, increasing in intensity from 11 p.m. onward. Sound sleep was quite impossible. The principal areas involved were also painful and swollen, and as was discovered later, the epidermis over each of them was extremely thickened. On the suggestion of a druggist, a soap containing 1% mercuric iodide was employed as a shampoo. This seemed to irritate the mites, which became very active after the use of this soap. Many of them began to move down out of the scalp, and some of the thickened epidermis began to slough off.
Those that continued down on to the body soon became embedded in itching red papules reminiscent of trombiculid infestations. Treated with strong sulphur ointment, they apparently did no further damage. They could be found on the shoulders, under the arms, beneath the breasts, on chest and both upper and lower back, occasionally around the umbilicus. The sensations of crawling and biting which were felt on legs and feet, in the latter case often on but seldom between the toes, indicated that some of the mites had migrated to the lower extremities.
Others of the mites, however, moved down on to the face, invading eyes, ears and nostrils. It was the mites in these locations that did the most damage. Both eyes became so badly swollen that it was impossible to move the eyeballs; to look to right or left it was necessary to move the entire head. Invasion of the eyes was not confined to the period immediately following the use of the mercuric iodide soap, but continued for many days thereafter, this invasion occurring mostly between midnight and 3 a.m. The victim of their attack would have fallen into a light sleep, when a sharp pin-prick-like sensation in one eye, followed by an immediate response on the part of the already badly swollen eye would drive sleep away quite effectively. The movements of a mite that had entered under the eyelid could be felt as it crawled slowly about, then began to ‘dig in’ at which moment the eye suddenly became even more swollen than before. An almost continuous flow of lachrymal secretion seemed to attract the mites and made vision difficult. At no time, however, was there evidence of the formation of pus in the affected eyes.
Invasion of the nostrils produced quite distressing symptoms, as of something crawling and scratching in the mucus membrane; of then this accompanied by a distinct irritation of the throat, trachea and bronchi. Early invasions of the ears seemed confined to the region of the pinna, in the folds of which the mites burrowed, producing the usual itching red papules. Their presence in the ears was the cause of some concern on my part, as it is well known that certain species of mites may cause great damage in the ears of small animals. Apparently we may consider ourselves most fortunate that his particular mite did not choose to invade the external auditory meatus, from which location it might easily have pierced the eardrum. The difficulty of controlling such an infestation in the ear, had it occurred, is at once apparent, since as yet we have found no drug that is completely effective against the mites. Even had such a drug been known, its use in the ears might have been inadvisable. Unfortunately for humans, medical doctors are much less versed in dealing with such situations than are the veterinarians who care for our dogs and cats.
In addition to the trouble caused directly by the mites, a certain amount of allergic reaction and probably also of secondary bacterial or fungicidal reaction were also present. It is difficult if not impossible to determine how much additional injury and discomfort may have resulted from these secondary infections.
The most acute phase of the infestation yielded to control measures within six weeks. Henceforth, the symptoms previously mentioned became less sever and somewhat chronic, exhibiting an increase in activity more or less periodically every two and one-half to three months. This condition has continued up to the present time. Even this summer (1950) live mites have been taken from all three members of the family. July and August are their periods of greatest activity.
In spite of my optimism in regard to the limited activity of those mites that invaded the ears, there is still a question as to whether or not the difficulty I am now having with my ears, and the rather sudden onset of deafness on the part of the other infested members of my family, may have been aggravated at least by the presence of the mites. Likewise, I wonder if the chronic sinusitis to which I have been heir for some time may also have been increased by those mites which entered the nostrils.
The much-thickened epidermis had sloughed off from the three most heavily infested areas of the scalp before the end of the the period of acute symptoms, and since that time no such unusual thickening in these or other parts of the body has been noted. Itching red papules continue to occur irregularly on the scalp, the pinna of the ears, on the face, and occasionally on neck chest and back. Such areas frequently become covered with small incrustations. The mites succeeded in establishing themselves temporarily on one writst, between the third and fourth finger of one hand, just below one ear, on the ventral surface of one knee, and just below the hair line above the left temple. Applications of 2 to 5% aqueous solution of gentian to each of these affected areas except the one above the temple, repeated daily for a week or ten days, finally cleared up each of these sites of infestation, some of which were reminiscent of the behavior of scarcoptes scabiei. The network of red lines and itching swellings above the left temple seems also to have been brought under control, largely through the use of sulphur ointment and lysol. There still remain infested areas on the face, in the upper corner of the lower lip and in the right eyebrow, which have not yet been brought under control. Tunnels or burrows, presumably in the dermis, could be seen on the wrist, in the soft skin between the fingers, on the knee, below the ear, above the temple, and on the lower lip. On wrist and knee, these burrows took the form of one or more or less straight central tunnel, visible as a slightly raised reddish line, from which extended at right angles, several shorter lateral tunnels. Above the temple, as noted, a network of raised red lines with small swellings here and there, indicated the presence of the mites. In other areas mentioned, the tunnels were more or less U-shaped, somewhat wider at one end, and apparently without lateral branches. Inasmuch as it has never been possible to locate a mite in the sloughed-off epidermis or in the small incrustations which form over infested areas, it seems probable that the burrows are actually in the dermis. Further evidence for this belief is the fact that live mites have been captured from the deeper regions underlying such an incrustation when the latter had been removed. Certainly, during the period of acute symptoms, when mites could be felt running about below the thickened epidermal masses, and causing their host almost to tear off bits of the scalp in an effort to get at them, none were even inconvenienced by any substance used against them until the mercuric iodide soap was employed. One of the most annoying runways occupied by the mites, the entire length of the right eyebrow, with extensions to and from the adjacent hairline, does not show on the surface any indication of its presence save a slight swelling. Even when such a burrow does not show externally, however, its presence it readily ascertainable by reason of the activity of the mites, which habitually travel from one area to another along certain well-defined highways. If these highways were in the epidermis, it should seemingly have been possible to capture mites in them, as the mites moved from place to place.
Symptoms experienced by other members of the family.
It was not until some time after my acute symptoms had subsided that other members of the family began to feel the effects of the mites. Whether they were less susceptible to the mites, from whatever source these were originally obtained, or whether they were infested from my infestation, we shall never know. As I had not at any time discussed the details of my symptoms with either of them, it is evident that they did not develop symptoms ‘in sympathy’ with mine. Only now and then, one of them would mention her particular symptoms as of the moment. One has had more difficulty with mites invading the pinna of the ear than I have had. These mites, when driven from the ear by the use of sulphur ointment or some other substance, moved down on to the body and became embedded there, particularly on the chest, shoulders and lower back. This member has also had much trouble with mites invading the nostrils. The other member of the family had more difficulty with mites embedded on the neck and shoulders. At no time have either of them suffered acutely from the presence of the mites.
The medical profession.
Aid was of course sought from the medical profession, but very little help in treatment for the symptoms was forthcoming from this source. A reliable oculist who was consulted during the period of my acute symptoms reported the finding of some foreign objects embedded in the eye, but as these were apparently not in the cornea, he thought they would do no serious damage. A dermatologist, recommended by a local physician, was apparently convinced without more than a very casual examination, that the patient’s symptoms were largely imaginary, those that did exist having been caused by an ill-advised attempt on the part of the patient to rid herself of something that was not there. Not until three days after my visit to his office, however, did he tell me this. Even he could not fail to note the greatly swollen eyes and the three areas of thickened epidermis on the scalp. He X-rayed the eyes and the affected scalp areas, and recommended several days’ stay in the hospital, ostensibly for further treatment of the eyes. Actually, such treatment consisted solely of applications of hot boric acid compresses to the eyes, and the use of cold boric acid as an eye wash treatment which I could have rendered myself at home fully as well. After the third day in the hospital, he turned me over to a neurologist for treatment of my ‘psychoneurotic’ condition. The patient, however, succeeded in convincing the neurologist that she had no need of his services. The local physician now suggested the use of sulphur ointment on the scalp, such as had been used effectively by the patient against mites embedded elsewhere on the body. She also suggested the use of sulfathiazole, which seemed to aid materially in reducing the allergic reactions and possible secondary infections. The complete amazement of this physician, when she was later shown some of the first mites to be captured, indicated full well that she, too, had accepted at face value the dermatologist’s diagnosis of ‘Just imagination.’ Hence any real aid in the treatment of the scabitic condition, other than the suggested use of sulphur on the scalp, came from the patient herself or from parasitologists. It is doubtful if the X-raying of eyes and scalp had much effect on the mites, although it may certainly have aided materially in clearing up secondary infections.
To date, no treatment applied has been able to bring about complete eradication of the mites. However, many substances used on the scalp and elsewhere on the body have afforded welcome even though temporary relief from the annoying sensation produced by the mites. It would seem that a certain number of the substances employed have actually killed some or all of the adult mites, but have had no effect on the ova. Any substance used on the scalp was applied with sterile cotton or with a pipette, usually being then well rubbed in with the finger tips. Twenty-four to 48 hours was the usual length of time between the application of any one substance and its removal by means of a shampoo, although some substances were left on the scalp for at least a week. The scalp was always shampooed as a preliminary to the application of any substance, and no other substance was used until the preceding one had been thoroughly washed out. After the use of certain of these compounds, a period of two to three months often elapsed before renewed symptoms indicating the presence of the mites were apparent. On circumstantial evidence this might be taken to indicate that the length of time required for the maturation and hatching of a young mite from the ovum is approximately two to three months. There is always the possibility, however, that the young mites may not make their presence felt at once, and that the growth period is really somewhat shorter. Attempts at control have continued actively from 1943 up to the present time.
Many substances employed as a possible means of control proved useless for that purpose. Among these may be mentioned: DDT powder, 10% used as for pediculosis; kerosene emulsion with vinegar; Seargant’s Mange Cure, which had been of value in treating mange on two pet cats some years before, but was of no help in the present situation; ethyl, normal butyl and isopropyl alcohols, used separately and in varying strengths from 70 to 100%; ether, applied to the scalp a few drops at a time; 3% phenol, also as used for pediculosis.
The following chemicals have proved of value in bringing relief, sometimes of a very temporary nature, from the activities of the mite:
Sulphur ointment, 15% USP, applied to the scalp or other parts of the body. This could be left on for several days after application. It is one of the most helpful substances employed to date. In the early treatment of mites on the body, a stronger preparation of sulphur was used, made by mixing flowers of sulphur with Crisco or Spry. The exact percent of the sulphur so used was never determined. At one time only during the period of acute symptoms was any sensitivity to sulphur noticed. The ointment used at that time was made up in lanolin. This sensitivity soon disappeared and no indication of it has been felt since that time. Mites treated with sulphur seem to die in situ.
Gentian violet, 2 to 5% aqueous solution. This, if left on any affected part of the body for a week or 10 days, and if constantly augmented by additions of the same every few days, proved completely effective in eradicating the small colonies already mentioned, on wrist, knee, fingers and below one ear. Unfortunately it was never feasible to leave this dye on the scalp for more than two days at the most, as it is by no means desirable to go about with one’s scalp and hair painted purple. There is also some question as to whether or not gentian violet would prove as effective over areas in which the mites were more deeply located or where they had been "at home" for a considerable period of time.
Lysol, a relatively strong solution (4 teaspoonfuls to the pinte, or even somewhat stronger) has proved to be one of the most effective methods of killing the adult mites. It can be used, at this strength, on small areas only. It has the disadvantage of possessing a strong odor and is also hard on the skin. The epidermis usually peels off from any area so treated some time after the use of this solution. In a weaker solution it has been used over the entire scalp following a shampoo, usually being rinsed off in a few hours. It is also by the use of lysol solution that some of the mites have been captured, as noted below.
Zemo, both regular and extra strength solutions, has proved of value in reducing the irritation caused by the mites’ activities, and may even have killed some of the adult mites.
Germicidal soap, 1% mercuric-iodide, used as a shampoo, appears to be quite effective against the mites, but does not eliminate all of them, nor does it appear to have any effect upon the ova. This soap is used quite regularly as a shampoo, and since no ill effects have followed its use except at one period of acute symptoms, it seems evident that this soap cannot have been the causative substance which precipitated the acute attack. No evidence of sensitivity to this soap has been noted. It often causes the mites to migrate, however, which is not desirable if the migration is toward eyes or ears.
Zinc oxide ointment. This has been used at times alone or in combination with sulphur ointment, as is sometimes recommended, to alleviate itching on areas of the face and neck. Painted around each eye, it served to prevent the entrance of many mites into the eyes, as they could be felt moving up to this barrier but going no further.
In addition, certain other compounds known to be of value in the control of sarcoptic and other forms of mange, have been employed. Of these, the first two were suggested by Dr. Gaines W. Eddy, who has recently published the results of a series of experiments with some new synthetic organic compounds as scabicides (Jour. Investig. Dermatology 12(2): 117-123, 1949). He likewise sent me samples of several of these substances, enough for two or more treatments with each. It seems quite possible at the present writing that one or both of these new chemicals may eventually prove effective in the eradication of our mites.
Benzyl salicylate. An aqueous solution of salicylic acid and benzyl ester 10%, in 2% of Tween 20, is the first of these two drugs. Samples of the Tween 20 were kindly donated by the Atlas Powder Company of Wilmington, Del. This has been used on my own scalp for several different doses during the past two months. After each such use, the symptoms were temporarily eliminated, but migration of the mites was quite noticeable. It has recently been employed in the treatment of one of the members of my family, but living mites are still present on her scalp.
2-phenylcyclohexanol 10%, and Tween 20, 2% as an aqueous solution, is the second chemical recommended by Dr. Eddy. The Dow Chemical Company of Midland, Mich., was kind enough to provide me with samples of this compound. It has recently been used on my own scalp. As yet, the results are inconclusive, but again the symptoms are temporarily alleviated. Migration of mites occurs after its use. Ultimate success might be achieved by the frequent repetition of this substance to the scalp and other affected areas, if used repeatedly over a sufficient period of time so as to kill all newly hatched larvae before they reach maturity.
Benzyl benzoate. This has been employed in two forms: first as a liquid, and second as an emulsion, the Wellcome brand, which was much used by British scientists during World War II in the treatment of human scabies. Some relief was obtained from its use, particularly when the emulsion was employed. It seemed especially effective against those mites which had located in the pinna of the ears. Migration of the mites after the use of this compound was most pronounced. Recently, the emulsion has been used on small areas of the scalp at double the strength recommended. Some migration has been caused, but many of the mites seem to have been killed in situ. As it is probably unwise to use benzyl benzoate at double strength except on very limited areas, and as it fails to effect complete cure even when so used, this drug cannot be relied upon to control the mite under consideration.
Tetraethylthiuram monosulphide. Another remedy much used, and with good success according to the literature on the subject, against human and animal scabies. Used as an alcoholic solution as recommended, it has not proved as useful in the treatment of our mite as have several other substances. It is stated to give excellent control against Sarcoptes and Notoedres, but it seems not to be the answer when used against Dermatophagoides.
Gamma isomer of hexachlorocyclohexane. This was used in the form of an ointment sold under the commercial name of KWELL. Ticks and mites are reported to be killed readily by this substance, but not so the mite Dermatophagoides. It was left on the scalp for several days before removal with a shampoo, and almost immediately thereafter the mites could be felt moving about in their accustomed places.
40% aqueous hypo (sodium hyposulfite). This was allowed to dry on the scalp, and was followed by a second application, which was also allowed to dry. Sometimes hypo was used alone, or again was followed by 5% HCl after the second application was thoroughly dry. Both methods often brought relief for several days, even for a week or more. But again the ova were evidently not destroyed. Although this treatment has been repeated many times during the past four years, the mites are still with us. However, it did give temporary relief, and caused only a moderate amount of migration on the part of the mites.
Considering the above list of chemicals, many of which are purported to eradicate several species of mites commonly infesting man and domestic animals, it would seem that the mite presently under consideration is much more difficult to deal with than are such other genera as Sarcoptes, Notoedres, Psorptes and Chorioptes. Whether or not this is due to an inherent ability of Dermatophagoides to withstand strong chemicals or whether, as it seems more likely, this mite inhabits the dermis and is therefore less subject to contact with chemicals which are employed against it, we cannot say. At any rate, we have first-hand information that this mite is not easily eradicated.
Capturing the Mite.
This was a tedious process, and the number of mites actually captured is surprisingly low. This does not mean, to me at least, that there were not many more mites present at any given time, which could not be captured by any method employed.
My first attempt at locating the causative agent by examining sloughed-off epidermis and incrustations from infested areas, after treating these with KOH, was completely negative. Hence it followed that similar material which was collected from my scalp and body and sent away to two different parasitologists for examination also yielded negative results. Both of the parasitologists were interested in the case, and anxious to help if possible, but they were located many miles away from the individual who had the infestation.
During the period of the acute symptoms, my eyes were so badly swollen that it would have been impossible for me to recognize a mite had I been able to capture it. Furthermore, during that summer I did not have a micrscope at my disposal. A few months after the acute symptoms had subsided, however, I captured two very small Hymenoptera which had been felt crawling about on the scalp. These have been identified by Mr. C. F. W. Muesebeck, Chief of the Division of Insect Identification Bureau of Entomology and Plant Quarantine, in Washington, as members of the Trichogrammatidae, know to be egg parasites. There will never be any certainty as to whether or not these Trichogrammatidae bore any relationship to the mite infestation, but of course there is always at least a possibility that they were parasitic upon the ova of Dermatophagoides. The technic, if one may call it that, of their capture was the same as that which was soon successful in the capture of the first mite found. It consisted of holding a bit of sterilized cotton, dipped in lysol solution, firmly in place for several minutes directly over a spot where a mite or some other arthropod could be felt moving about. Success by this method was never very great, but at least it was thus that most of the few mites taken directly from the scalp have been captured. If successful, the mite still alive and moving sluggishly, would be found adhering to the cotton when the latter was removed from the scalp.
A rather amazing number of other arthropods have likewise been captured from the scalp in the same fashion. Among these were: several Hymenoptera of a considerably larger size than the Trichogrammatidae; an oribatid mite; small portions of the skeleton of an unknown arthropod bearing feathered hairs similar to those of a trombiculid mite; what appears to be the cast skin of a small spider; an apparent jassid; and of course, many small gnats and other Diptera, among these being the psychodids and ceratopogonids. Pollen of various sorts was also found commonly. Perhaps a study of the flora and fauna of the human scalp in summer might be interesting.
A modification of the method of capture indicated above also yielded several mites of Dermatophagoides. Immediately following a shampoo, especially when the mercuric iodide soap had been employed for that purpose, mites could be felt moving and scratching at various places on the scalp. Sometimes, following such activity, there would occur a sharp pin-prick-like sensation in a different spot than the original one, and the appearance at that spot of one of the customary itching red papules which often increased in size later. This I interpret to mean that the mite, disturbed but not killed by the substance used in the shampoo, had come up from its "den" in the dermis, and when on the surface of the skin had moved to a new location, when again it burrowed in, perhaps using a hair follicle as its point of entrance. If now the saturated cotton was applied before the second burrowing-in took place, there was a good chance of capturing the mite.
Another method of capture that has been met with greater success is as follows. After treatment of the scalp or face with sulphur or indeed with almost any of the chemicals listed as being of value, mites could sometimes be found the following morning on the pillow of the individual using this treatment. These mites, collected by means of a bit of cotton moistened with water, were usually dead when taken, but recently several live, active impregnated females have been captured in this manner.
At one time only did I succeed in "digging out" a mite from my own scalp, by means of an exploratory fingernail applied to the area beneath an incrustation, the latter having been forcibly removed. The mites had been felt moving about under this incrustation. When thus captured, the mite was quite alive and healthy, and waved its legs lustily as it was being examined under the microscope.
It is perhaps worthy of note that even a small mite, when moving about actively in or on one’s scalp feels "as large as a cat." A peculiar sensation as of pressure sometimes occurs synchronously with the activities of a mite which has been annoyed by treatment with some chemical. This has been especially noticeable after the use of sulphur ointment. Then there is another peculiar sensation sometimes felt: a "streaming" activity, as though many mites, perhaps small ones, were leaving some common center, due to the use on the scalp of some chemical distasteful to them, and "fanning out" in various directions from this center. In fact, some mites may later be located in red papules that had not been there before, on face, neck or shoulders, following such a streaming. I venture to preduct that if any dermatologist should become the host of this mite, he would not diagnose his case as "Just imagination."
Possible Source of the Infestation.
The source of the infestation is completely unknown. Two possible sources are: (1) two pet cats of the household, the last one of which died at least ten years ago; these cats suffered from what we called "mange" for which ailment we treated them, with some degree of success, with Seargant’s Mange Cure; and (2) a neighbor’s doc, which for years suffered from some sever skin infection which several good veterinarians tried in vain to bring under control, so that the dog had finally to be destroyed. It is possible that our cats acquired their infestation from the dog, and that we acquired ours from either the cats or the dog.
At the time of the most acute symptoms, in the summer of 1943, I tended toward the belief that the infestation had been acquired, in my case, from a woodchuck which I had used to obtain material for some histological preparations. That the woodchuck did have ectoparasites, I know for a certainty, as I not only removed from its body some lice (apparently Enderleinellus marmotae) but also some immature or nymphal Ixodes. I noted some of the latter climbing my arm as I worked on the woodchuck, and later captured one nymphal Ixodes, probably cookei, from my own scalp. However the fact that I had noticed itching papules in my scalp for some years previous to this time, leads me to believe now that the mite Dermatophagoides did not come to me from the woodchuck. This mite has been rather recently reported from rats, but I have had no occasion to work with rats other than to kill an occasional white rat for histological purposes. The same mite has been reported from the skin of birds. But I have had occasion to come in contact with birds only to pick off a few ectoparasites from such dead specimens as may have been brought into the laboratory, and most of this has been done since 1943.
The fact that the infestation has lasted for so many years seems to indicate that the mite can adjust itself readily to life on or in the human skin, and maintain itself there once it is well established.
This mite belongs to the family Epidermoptidae, which may be easily distinguished from its nearest ally, the family Psoroptidae, by the fact that the females of the Epidermoptidae possess suckers on all the tarsi, whilst females of the family Psoroptidae have long whiplike setae on the third tarsi.
Dermatophagoides Bogdanow, 1864
Dermatophagoides Bogdanow, 1864. Bull. Soc. Imp. Nat. Moscou 37 (1): 341-348, pl. 7, figs. 1-2.–Vitzhum, 1929. Tierreich Mitteleuropas, Acar, Bd. 3, 1f. 3,102–Sasa, 1950. Japanese Journal of Experimental Medicine 20:519-525.
Pachylichus Canestrini, 1894. Prospetto del’ Acarofauna Italiana, fasc. 6:824-829, pl. 77, figs 1-4.–Berlese, 1897. Acari, Myriopoda, Scorpiones, Cryptostigmata, Sacroptidae, fasc. LXXXII, no 12.–Oudemans, 1904, Ent. Bericht. 20:190-195 (synonomy with Dermatophagoides.)
Mealia Berlese, 1897. Acari, Myriopoda, Scorpiones, Cryptostigmata, Sarcoptidae 1:104.–Berlese, 1898. Idem., fasc. LXXXIX, no. 10, and XCII, no. 3 and no. 4–Canestrini and Kramer, 18999, Das Tierreich, Demodicidae and Sarcoptidae, 137-138.–Trouessart, 1901, Bull. Soc. Zool. France XXVI:82-84.
Viscopteres Sasa, 1947. Nisshin Igaku 34(3):167-170.
Type: Dermatophagoides scheremetewskyi Bogdanow, 1864. Bogdanow described two forms of mites. The first of these, designated D. scheremetewskyi, is a female, depicted in dorsal and ventral views in figs. 1a and 1b. It occurred on the surface of the skin of humans suffering from the itch. The mite of the second type, found on a single occasion on a child suffering from herpes farinosus, and designated by Bogdanow as "Acarus de l’Herpes farinosus," is depicted in figs. 2a and 2b. It is a male, very probably that of D. scheremetewskyi as Bogdanow himself suggested; certainly it belongs in the same genus as the previously described female mite. Bogdanow compares his new genus with Dermatophagus, saying that it has "une grande resemblance ave selui des acriens due genre Dermatophagus, decrit en detail par Furstenberg."
He notes two differences between Dermatophagoides and Dermatophagus: (1) the conformation of two lyre-shaped chitinous arcs near the genital opening of the female; and (2) differences in the termini of the third legs of the females. (Dermatophagus Furstenberg, 1861, is a synonym of Chorioptes Gervais, according to Ewing, 1929.)
Dr. Menber, an M.D., reports on the finding of a species of parasitic mite infesting the human skin and causing a sever dermatitis. This mite he describes and figures, as: "Acarus incapsulator or Sarcoptes trichogenetos (Boeking)." The mite is said to have been found also in capsules beneath the skin of the same patient and was thought to be related to or be the causative agent of trichinosis, hence the specific name applied to it.
North American records are: Texas; in a house in Kentucky; from a rat in Florida; from a house in Chester, N.H., causing an itch; and from New York. These, combined with the European distribution, indicate a rather widespread range and eventually the mite should be found throughout most of the world.
Several other species have been placed in Dermatophagoides, but much work is needed before the exact status of the various species can be determined with certainty. Dr. Manabu Sasa, of the Institute of Infectious Diseases, University of Tokyo, Japan (The Japanese Journal of Experimental Medicine, vol. 20, pp. 519-525, 1950), discuss the genus Dermatophagoides in Japan. D. saitoi (Sasa) was taken in sputum of a patient with typical Loeffler’s syndrom, D. takeuchii was found in urine, and an undertermined species was obtained from a patient with chronic bronchial asthma.
Generic characters. Body oval. Dorsal shield present. Prominent transverse groove anterior to middle of body, the two hind pairs of legs posterior to this groove. Sexual dimorphism evident, especially noticeable in legs of male; however, all legs in both sexes are well developed. Posterior end of body more or less rounded, not lobed in either sex. A pair of copulatory suckers present on anal plate of male. Genital openings of female between third coxae; surrounded by a crescentic anterior and two curved lateral sclerotized supports. Corresponding opening of male somewhat more posterior, lying between fourth coxae. Penis short, conical. Epimera of first legs not united. All tarsi in both sexes with short terminal stalks (peduncles) which bear cup-shaped suckers. Claws, where present, small and not easily discernible. Two pairs of long, strong posterior setae, the outer pair longer. One pair of vertical setae, also long and strong, arise from anterior portion of dorsal shield. Chelicerae powerful, terminating in paired chelate cutting surfaces which are dentate on the inner margins. Legs I and II directed forward; legs III and IV directed backward.
Dermatophagoides scheremetewskyi Bogdanow
Dermatophagoides scheremetewsky Bogdanow, 1864. Bull. Soc. Imp. Nat. Moscou 37(1):341-348.
Mealia pteronyssina Berlese, 1897. Acari, Myripoda, Scorpiones, Cryptostigmata, Sarcoptida 1:104.
Acarus incapsulator Menger, 1896. Texas Medical News, San Antonio, Texas, Sept. 24:1-20 (attributed to Boeking).
Sarcoptes, trichogenetos Menger, 1896. Texas Medical News, San Antonio, Texas, Sept. 25:1-20 (attributed to Boeking).
Eleven females and nine males, all adults, were available for study. Three of the females, taken alive, were observed before being killed. On larva and two nymphs are also present in the available material. Mites were mounted (a) in clarite, after clearing in xylol, or (b) in modified Berlese’s medium, usually direct from 35% alcohol. Some mites were cleared in KOH previous to mounting; others, including most of those mounted by the second method, did not undergo such preliminary treatment. Male mites are almost transparent, but female mites, unless cleared, show well only such structures as are on the surface which is uppermost. Certain morphological features are seen more clearly in mites mounted by the first method; others, by the second.
Size. Female: 10 specimens measured. Average length, 355.2 u, from tips of mouthparts to tip of abdomen; average width 196.8u. Longest, 400u; shortest 320u; widest 240u; narrowest 160u. Male: 8 specimens measured. Average length 296u; av width 180u. Longest: 352u; shortest 256u; widest 208U; narrowest, 128u.
Mouthparts. See Plate III, figs. 15, 16, 17, 18 and 19. These are quite complicated, heavily sclerotized, and interpretation of some of th parts is difficult. Two large chelicerae are present, each bearing distally a pair of cutting surfaces, the inner margins of which are dentate or serrate, as indicated in figures 15, 16, 17 and 18. From ventral view, two large triangular or conical structures are seen; from beneath these, the cutting surfaces are visible. Perhaps these represent a bilobed hypostome or rostrum. These structures are shown in the ventral views of male and female mites, Pl. I, figs. 3 and 4. Viewed from above, a very similar pair of triangular structures lying directly beneath the basis capituli almost obscures the cutting surfaces, which can be seen only by focussing downward. These are perhaps the basal portions of the chelicerae, on which the cutting surfaces are bone. It appears, then, that there are two sets of sclerotized, conical sturcutres, on pair dorsal, the other ventral, with the cutting surfaces between them, and attached to the uppermost pair of sclerotized cones. A pair of well-developed palps is present, on palp lying on each side of the conical structures. Each is four-segmented, if the basal segment <CLIP from this page (15) to page 22>
One mite was kept alive for two days, at the end of which period it was as lusty and active as when first caught. Immersion in water, in which the cotton was kept moist, did not seem to disturb the mite. Occasionally a mite would leave the cotton and try to crawl about on the floor of the watch glass, but made little progress.
One mite was killed in warm, not hot, water in which it died very quickly. A second was immersed in lysol solution of the strength used for treatment of the scalp. For 30 seconds it swam actively, then became quiescent. At 45 seconds, no movement could be detected. It is thus evident that the mites are susceptible and easily killed, if the killing substance can be brought into contact with them. But when in or under the epidermis, they are well protected.
Opportunity is taken here to mention a similar situation in which another mite, Bdellonyssus sylviarum (C.&F.), family Laelaptidae (Dermanyssinae) was the causative agent. Several years ago a Mrs. X wrote me in regard to a difficulty which she and other members of her family had been experiencing. They had been suffering considerably from the attacks of an unknown "something" which was present on their scalps and bodies, and which they had been unable to eradicated. Mrs. X, on the advice of her physician, had consulted a dermatologist, who told her she was merely suffering from a "phobia" and to go home and forget it. Two days later, Mrs. X was fortunate enough to capture two dead mites that fell from her scalp. These she took to the dermatologist, who now became interested in her case, and prescribed some lotion as a treatment. Meantime he sent the two mites off for identification, and in due time they came to me. I believed them to be Bdellonyssus sylviarum, but sent them to Dr. Edward W. Baker for confirmation. He concurred in the above identification.
I have had some further correspondence with Mrs. X on this subject. The last letter, written several months ago, indicated that she was still suffering from the effects of the parasites, and that nothing prescribed by doctor or dermatologist had been of permanent value in controlling her trouble. On the theory that English sparrows nesting under the eaves might have brought the mites into their home, the family had had the entire house fumigated, repainted and repapered and still Mrs. X suffered from the mites. The other members of her family had meantime recovered from their unpleasant experience with the arthropods.
Here, then, is another instance in which humans, attacked by mites other than Sarcoptes, have received scant attention from the medical profession. Had Mrs. X not been fortunate enough to capture the two mites, she might really have come to believe that she did have a "phobia," whatever that expression may be taken to mean. And even with the causative agent at hand, and identified, the dermatologist was unable to treat the infestation successfully.
A certain parasitologist, in conversation on the subject of mites, has told me that he has had several experiences similar to the above, in which persons fairly frantic from the attacks of some unknown creature had been unable to obtain aid from their doctors, and had written him or come to him for help.
What, on the other hand, could a medical adviser or dermatologist prescribe, in the present state of our lack of knowledge, in such a case? So little is known of the activity and behavior of many mites that may occasionally infest man, and so little also as to the effective methods of treatment. This is, I believe, a subject which is in great need of further investigation. I suggest that the medical profession might do well to take the lead in such a study.
The mite dermatophagoides scheremetewskyi Bogdanow, a member of the family Epidermoptidae, is herewith reported as attacking humans, infesting the scalp as well as other parts of the body. Its behavior is not similar to that of such mites as Sarcoptes. It is believed that the mites burrow down into the dermis, in which they make their runways, and that they occasionally come to the surface from these runways. In general, movements of the mites from place to place are believed to occur within the shelter of these runways.
To date, no treatment employed against the mite has been completely effective, in the sense of killing the ova as well as the adult mites. Nor has repeated re-application of any of the substances used, proved effective in eradication. Many different substances known to be highly effective against Sarcoptes and Notoedres have been employed as scabicides over the past seven years, in attempts to bring about control or eradication of the mites. Of these, the old standby, sulphur ointment 15%, is the one to which we return again and again, after other substances have failed. Yet it, too, has been unable to eradicate these arthropods. Three compounds which have been most recently used, benzyl salicylate, 2-phenylcyclohexanol, and benzyl benzoate used at double strength, may perhaps be the answer to our problem, but it is still too soon to know. Certain other substances known to be effective against sarcoptic scabies have proved of little value in the treatment of Dermatophagoides. Among these are tetraethylthiurum monosulfide, and benzyl benzoate used at the usual strength.
The infestations here reported have lasted ofver a period of at least seven years, and have occurred on three adult members of the same family. The source from which the infestation was originally obtained is unknown. If, as we surmise, infested cats or a dog suffering from mange may have been the source or sources, there has been no chance for reinfestation for some years, as these animals have long since died. But there is no certainty that the "mange" from which they suffered was produced by the same causative agent as that which has infested us. It seems probable that the human infestation has lasted for this period of time due to continued self-reinfection of each individual concerned, since the ova are apparently never destroyed by any treatment we have employed. It should be noted that the mites seem to thrive on cleanliness, if weekly, sometimes semi-weekly, shampoos during the entire period of time can be used as a criterion. The mites, be it recalled, occur principally on the scalp.
It is to be hoped that members of the medical profession may soon come to realize that it is possible for humans to be infested with mites other than Sarcoptes, give the patient the benefit of the doubt and try to determine the real cause of the ailment. It is not too easy for a person infested with said mites to make a diagnosis himself, nor to prescribe treatment if he is fortunate enough to locate the causative agent. The medical adviser and the dermatologist, on the other hand, are in a position to add much valuable information in regard to the habits and activities of this and other human-infesting mites, as well as to determine methods of control or eradication, if only they are willing to do so. We can but echo Bogdanow, when he states: "Il serait a desirer que les observations prochains de M. Scheremetewsky et des autres dermatologues nous donnent plus des details sure les acaricides, parasites de la surface de la peau de l’homme."
Had it not been that the writer of this article was (1) unwilling to accept the dermatologist’s verdict of "Just imagination";(2) had sufficient knowledge of parasitology and access to enough literature on this subject so that she was able to proceed "on her own" in quest of the invading organisms; and (3) knew how to preserve and mount the mites when found, this mite would in all probability not have been located, and the difficulties experienced would never have been attributed to their real cause. It is quite possible that the mite Dermatophagoides infests humans more often than anyone has known, but that its attacks are usually of a minor order, so that the itching and other annoyances caused by its presence may be attributed by the human host to (1) "imagination," as the doctors would have us believe; (2) "summer heat," since the mites are most active during hot weather; or perhaps (3) "dandruff." A wide-open field exists for anyone enterprising enough to do some real investigation on this subject.
The presence of the mite Bdellonyssus sylvarium as a causative agent of dermatitis in the human scalp and on the human body is likewise reported. At last reports, the unfortunate host of this mite was still suffering from its depredations. In this case, the diagnosis has been: "Just a phobia." Here is another field in which to date there has been no competition in the matter of studying the mite as an occasional human parasite.
I really do feel that Collembola do play a role in our itching and crawling sensations. In fact, I believe they are responsible for the majority of my crawling and biting sensations at this point. If I am correct, and I’m not sure I am, are they here to feed on fungi and or bacteria in my body? As I stated in my very first post “It would seem the skin and body of the Morgellons sufferer has become like a rotting log, or the very least as favorable a home to both soil bacteria and soil based pests that they are both perfectly at home living in us”. However, not covered in this post are the fungi and bacteria that are found in the gut of the Collembola, could they set this whole thing in motion in us? Some are quite pathogenic. Just thinking out loud here.
I have a FIR (Far Infrared) heating pad that I purchased through Dr. Staninger and when I break out in these red pimple like things the FIR heatpad makes them go away and my skin returns to healthy looking very quickly, but it wasn’t cheap, around $350.00 if I recall. I don’t urge you to run out and buy one as it might not do the same for you and I know how much so many of you have already spent on things that haven’t worked. I have been considering lately as to whether or not the heating pad is killing them.
This post is a follow up to my first post regarding Collembola titled “Collembola – A Major Role in Morgellons Despite the Disinformation” and I hope you find this post interesting as well. I have found even more interesting information regarding Collembola. Here is a quote from http://www.earthlife.net/insects/collembo.html
They are generally small and some species of Neelidae (Collembola) are among the smallest hexapods in the world at just over 0.2 mm long (about the same size of the period at the end of this sentence) while the largest Collembola are in the family Uchidanuridae which can reach 10 mm in length. Most species live for a year or less, however some live considerably longer and the record for long life in the laboratory is 67 months for a specimen of Pseudosinella decipiens.
Most Collembola feed on the fungi and bacteria found in rotting organic matter but many arboreal (living in trees) and epidaphic (living on the surface of the soil) species also feed on algae. Some feed on other plant materials and in some places particularly Australia Sminthurus viridus is a pest of lucerne crops. A few other species are carnivorous feeding on Nematodes and other Collembola.
Note that above text mentions that some species of Collembola can be as small as .2 mm. Just so we are clear on things .2 mm is very small, .2 mm = 0.000787 of an inch !!!!! There are an estimated 30,000 to 100,000 undiscovered species of Collembola.
The smallest insects are fairy flies, which are insects that parasitize other insects’ eggs by laying their eggs inside them. Fairy flies are only 1/5 of a millimeter in length. Many beetles are less than one millimetre in length, and the North American Feather-winged Beetle Nanosella fungi, at 0.25mm, is a serious contender for the title of smallest insect in the world. Other insect orders which contain extremely small members are the Diptera (True Flies) and the Collembola (Springtails). The “feather-winged” beetles and the “battledore-wing fairy flies” are smaller than some species of protozoa (single cell creatures). Megaphragma caribea a hymenopteran parasite from Guadeloupe, measuring out at a huge 0.17 mm long is in contention for the smallest insect.
See http://www.entomon.net/smallest-fastest-insects-alive.htm for more information. We are talking about flies here that you probably couldn’t see with your naked eye, amazing.
My first post talked about the National Pediculosis Association’s (NPA) study where they identified Collembola in 18 of 20 individuals studied, and also the 1955 cases is Sweden (there were many), and finally the 80 year old Woman in Romania. I would like to thank Sidney, one of our fellow readers and somebody that knows a lot about this subject. She pointed out to me that there were some mistakes in the identification in the samples of the 80 year old woman. She has first hand knowledge of this case.
When Neculai removed the Collembola and Rotifers from the body of the 80 year old Romanian woman please bear in mind Neculai was not an entomologist, but a veterinarian pathologist and head of the department of veterinary medicine at his university. This was not a case of “sample contamination.” The Collembola was removed from the Romanian woman’s tissue by a needle aspiration biopsy. The corrections I mentioned in my email refer to the PUPA and LARVAL stage which Collembola do not have. Rotifers were also found within the Romanian woman’s skin (actual photos shown left), along with bundles of fibers.
I didn’t include the pictures of the fibers that Sidney sent to me but suffice it to say they look like our typical fiber bundles. So, some of the images that were put forth as various stages of the adult Collembola on my first post were actually found to be Rotifers. If you really want to know more about Rotifers see this article “Epizoic and parasitic rotifers”. There is a interesting quote from the parasitic rotifer article (but you should click on the link and read the entire abstract).
There appear to be few records of epizoic or parasitic rotifers among vertebrates, apart from Encentrum kozminskii on carp, Limnias ceratophylli on the Amazonian crocodile, Melanosuchus niger, and an unidentified Bdelloid apparently living as a pathogenic rotifer in Man.
So, I’ll bet you can sense yet another blog post coming on Rotifers in the future and you would be correct, but for this post we are going to stay on the topic of Collembola.
Let’s start with Dr. Omar, M. Amin, Ph.D., founder of PCI, who is a Professor of Parasitology and who works with something he calls Neuro-cutaneous Syndrome (NCS) which to me at least, seems to be exactly the same thing as Morgellons, though he might differ with me on that. The photo shown on the left is of a springtial (Collembola) that was taken from an actual patient. If you really want to dive into some of Dr. Amin’s information see this PAGE and follow the links on NCS. And if you hit this PAGE look at Table #2 and notice how many were documented to have springtails (another name for Collembola).
Here is a quote regarding the Collembola shown above from Dr. Amin’s site.
Scalp lesions also occur in patients with neurological symptoms and are usually associated with arthropod infestation. JH (a tall, healthy, well-nourished, middle aged white American female from Arizona) had a number of such lesions (Fig. 4) from which springtails (Collembola: Insecta: Arthropoda) (Fig. 5) were collected by myself in December, 1995. There is only two other published reports of springtails from humans (Hunter et al., 1960; Scott et al., 1962).
Very interesting. Dr. Amin mentions that there are only two other published reports of springtails in humans. However, he doesn’t mention any of the cases in my first blog post, nor did I mention the cases he is referring to (Hunter et al., 1960; Scott et al., 1962). But as you are about to find out, we are just getting started. We are very grateful for your work Dr. Amin, you provide an amazing amount of information to aid in our search on your site.
Pescott, R.T.M. (1942:68-69) Australia:
“In 1939, specimens of springtails were received from a Melbourne specialist who stated that they were causing skin troubles on a female patient. The insect in question was the species Entomobrya multifasciata Tull., a European species originally described in 1871, but which is now cosmopolitan in its distribution. Womersley (3) records it as being common in cultivated areas in the Australian States. The symptoms of this case were as follows : the patient experienced a sharp biting sensation, followed by intolerable itching. There were few marks on the body with an occasional excoriated papule, while the irritation was distributed fairly generally over the trunk and limbs, but was most marked around the waist. Several specimens of the insect responsible for the condition were found on the patient’s body. She received no active treatment, but her clothes and bedclothes were sterilised and this was sufficient to destroy the insect and thereby remove the irritation.
On considering the origin of this infection, it appeared that the patient had recently moved into a new house where the garden was in the process of being made. The insects had apparently migrated to the patient when the grass, weeds and soil outside were disturbed.”
“In 1941, specimens of another springtail were received from a military hospital in Victoria, where skin irritations were occuring among the nursing staff. The species concerned was Entomobrya tenuicauda Schott., a native insect originally described in 1917 from Queensland, later recorded by Womersley (3) from Western Australia and Tasmania, and now from Victoria. In this instance, the presence of the insect produced on several nurses raised lumps very similar to mosquito bites, and which later were very irritable. In one instance there was also a good deal of reddening of the calf of the leg. These conditions lasted for somewhat less than twenty-four hours in each case, but reoccurred the next day, probably from more ‘bites’. On analyzing this case, it appears certain that the insects were introduced into the hospital with flowers, and from there moved on the affected nurses during their normal routine duties.”
Womersley suggested that the easily detached, long ciliated hairs of Entomobrya species undoubtedly would cause skin irritations. Pescott concludes that severe skin irritation can be caused by certain species of Collembola: “Typical symptoms are a biting sensation, followed by intense irritation and the production of small pimple-like bodies.“
Mackie, T.T., Hunter, G.W. & Brooke Worth, C. (1945:541-542) Australia:
“The Collembola are primarily phytophagous and are not usually thought of as medically important insects. Two Australian species, however, Entomobrya multifasciata Tullb. and E. tenuicauda Schott have recently been recorded as attacking man, the patients complaining of a sharp, biting sensation followed by irritation and papules similar to mosquito bites, with pruritus.”
Cited from Scott, H.G., Wiseman, J.S. & Stojanovich, C.J. (1962:430):
“Entomobrya nivalis (cosmopolitan) and Entomobrya tenuicauda (Australasian) have been reported as causing a pruritic dermatitis in man.”
Cited from Ebeling, W. (1975):
“They [Collembola] have never been incriminated in the transmission of any human disease, but Entomobrya nivalis L., a cosmopolitan species, has been reported to cause an itching type of dermatitis in man, …”
Martini, M. (1952:354) cited from Bryk, F. (1955:1824) :
“Very discomforting mosquito-like skin irritations attributable to collembolans of the genus Entomobrya attempting to bite. “
Mertens, J. in Christiansen, K. (1998 in 2001:in litt.) Belgium:
” Several years ago our Faculty of Medicine once offered me ‘strange small insects’, which were considered as being responsible for causing allergic reactions on the skin of a woman. Those insects were Seira domestica. I could prove that the scales of Seira on the cushioned seats caused the allergy. As you know, Lepidocyrtus, has scales too. “
Mertens, J. (2004:in litt.) Belgium:
“In 1976 (or 1977), our Faculty of Medicine was puzzled by a rare case of skin allergy in a woman, living near Ghent. The allergy was caused by the scales of Seira domestica on a cushion of a rotan chair. Whenever the woman used the rotan chair, the allergic skin response occured (and only then). The chair was located in the veranda, which was quite moisty and where the temperature was enjoyable. It turned out that the hollow rotan branches of the chair hosted a population of Seira domestica. During the night, they left their hiding place and crawled all over the chair. The cushion collected many of the lost scales, causing as such the allergic reaction.”
Scott, H.G., Wiseman, J.S. & Stojanovich, C.J. (1962:430) Texas:
“Springtail insects (Orchesella albosa Guthrie, 1903, forma ainslieri Folsom, 1924) were found infesting the heads and pubic areas of a family in Buffalo, Leon County, Texas, in June 1961. No dermatitis was reported due to this infestation, and the source of the insects was not determined. Based upon known habits of this species, some moldy household item (perhaps bedding) was probably involved. Orchesella albosa has never before been reported infesting man or houses. Its chewing mouthparts are probably not capable of biting man.
At this point I’m going to stop referencing cases. There are many, many more on the following links at the end of this post that you will find extremely interesting. I wish I had time to do this post justice but I’m so busy just trying to be a dad, hold down a job, and stay well. However, if anyone is interested in finding out if Collembola can infest human beings the answer is on this post and in the following links.
One could make the argument, okay, so maybe you could dig up 30 or 40 cases of springtails in human tissue using the links below, but that’s not very many. And I would answer that with “Yes, but who is looking? We are told we are DOP without so much as an examination”
If you take the time to look at these links, especially the first two you will begin to realize there are a lot of references of human infestation of springtails (Collembola) in man. As as referred to above in the medical literature “the patient experienced a sharp biting sensation, followed by intolerable itching. There were few marks on the body with an occasional excoriated papule, while the irritation was distributed fairly generally over the trunk and limbs” it sure sounds a lot like us.
The toxic ingredients common to all belong in four major categories: Zinc Oxide, Ethyltoluene Sulfonamide (especially in patients with allergy to sulfa and toluene)
Dr. Amin feels strongly apparently that Toluene plays a major role in NCS and if you look at his site I don’t care what we call it, NCS and Morgellons exhibit the same exact symptoms. Most people when they think of removing fillings think of “Mercury Fillings” but the kind Dr. Amin is refers to here are the newer kind. You know, the new white ones that weren’t supposed to be bad for us, sigh … I’ll be honest, I had quite a bit of dental work done before this all started, crowns and fillings and alike, all the new ceramic type too.
This subject is certainly not new to the Morgellons debate and indeed I have touched on this subject before. However, not the extent that this article is going to go into the evidence and support for the role of an active Collembola infection in Morgellon patients. If you think that you’ve read all there is to know on this subject you are about to be surprised. I believe this is one type of crawling the Morgellons sufferer feels and will talk later about a few very cheap ways to get rid of them (or at least make the crawling such that it isn’t overwhelming) and make your life so much better. This is a long post, but if you want to understand what is causing many of your symptoms I suggest that you stick with me on this. There are thousands of species of Collembola and they are extremely small in size and I read that they suspect some 30,000 species have yet to be identified.
First, let’s define what Collembola are for readers that might not know.
Collembola or springtails are the most numerous and the oldest known of all insects; they are found in the soil under leaf litter; they prefer damp conditions and some species are semiaquatic. About 2.000 species are classified. Springtails are wingless (Apterygota), brown to grey and measure about 5 mm.; they have antennae of different length, an abdomen with six segments and have a ventral tube probably involved in respiration, water absorption and locomotion. The name (springtails) refers to the characteristic method of locomotion leaping using the fourth organ, called Furca wich is doubled under the abdomen when at rest. They feed mainly on decomposing vegetal or animal material but they can feed on seeds, bulbs, roots, algae, fungi, insects or mites eggs. Some species have been observed in strict contact with humans causing persistent and not only occasional infestations.
In my first blog post titled “Does this identification mean anything? I do not know” I talk about Collembola and their possible relationship to Dr. Randy Wymore’s culturing Pseudomonas Putida directly off Morgellons fibers and the fact that Ps. Putida happens to be the favorite food of our Collembola friends. If you haven’t read my first blog post (linked above) I suggest you read it before continuing as it talks about a study in 2004 that discovered Collembola in a group of individuals previously diagnosed (if you can call it that) with delusory parasitosis. It also has some excellent pictures and links to Collembola not found in this post.
If you are familiar with Niels Mayer then you know the battle he has fought with those on Wikipedia regarding Morgellons disease, there is no doubt that a campaign is being run by a group of people to keep Morgellons listed as DOP on Wikipedia. Niels eventually setup Morgellons.Wiki where at least the grown ups are in charge.
If you look at the Wikipedia entry on Collembola you will notice that they attempt to totally discredit the 2004 NPA study. The National Pediculosis Association reported Collembola in skin scrapings collected from 18 of 20 research participants in its study published in the Journal of the New York Entomological Society. Here is the quote from Wikipedia attempting to discredit that study.
Claims of persistent human skin infection by springtails may indicate a neurological problem, or else delusory parasitosis, a psychological rather than entomological problem. Researchers themselves may be subject to psychological phenomena. For example, a publication in 2004 claiming that springtails had been found in skin samples was later determined to be a case of pareidolia; that is, no springtail specimens were actually recovered, but the researchers had digitally enhanced photos of sample debris to create images resembling small arthropod heads, which they then claimed to be springtail remnants.
There is a footnote on that comment but no real proof, in fact, I consider that comment nothing but disinformation probably by the same crew dogging the Morgellons wikipedia entry. Notice they make the claim these people really had pareidolia, here is the definition for that word.
The term pareidolia (pronounced /pærɪˈdoʊliə/) describes a psychological phenomenon involving a vague and random stimulus (often an image or sound) being perceived as significant. Common examples include seeing images of animals or faces in clouds, the man in the moon, and hearing hidden messages on records played in reverse. The word comes from the Greek para- —"beside", "with" or "alongside"- meaning, in this context, something faulty or wrong (as in paraphasia, disordered speech)—and eidolon—"image" (the diminutive of eidos—"image", "form", "shape"). Pareidolia is a type of apophenia.
So, it would appear that Wikipedia thinks these people have even something worse than DOP as far as crazy goes. Notice they even call the researchers basically crazy, that is an outright lie on their part. Where is the evidence to back up this claim that the researchers suffered pareidolia? Nonsense. What a hit piece, I wonder if the folks at NPA even know of this ridiculous claim.
Some Collembola experts disagreed with the NPA’s research findings, insisting that it was impossible for Collembola to live in human skin. Deborah Altschuler, lead author of the NPA paper, likens the scenario of Collembola and humans to the discovery of Helicobacter (H.) pylori otherwise hidden in the stomach lining, and the erroneous yet long held assumption that the stomach was a sterile environment and that peptic ulcers were caused by lifestyle choices. According to Kimball C. Atwood IV, MD., physicians scoffed when first faced with the notion of a bacterial basis for peptic ulcer disease. Altschuler asserts that there is more of a scientific basis for Collembola in humans than the entomologist and physician’s overwhelming acceptance of a psychiatric explanation (Delusions of Parasitosis) for people’s sensations of biting, stinging and crawling in their skin. See this PAGE on the NPA Site for some very interesting information I didn’t include in my blog post, it’s well worth the read.
The NPA says even the experts appear to have missed this 1955 Swedish Medical Journal report in which the well-respected entomologist, anthropologist and author, Felix Bryk, refers to the incidence of Collembola in humans as a plague, making mention of colleagues who during that time had also found Collembola as parasites in humans. Bryk said the Springtail Sira, (today’s spelling Seira), was a human parasite being confirmed for the first time in Sweden. All this prompted him to write a report to the medical literature in which he stated:
The report can be found HERE and you should really take a moment to read it, it’s only a few pages long.
Until now, collembolans or “springtails” have played a miniscule role as parasitic insects on the human body from a entomological/medical standpoint. Rarely, if ever, are they mentioned in the scientific literature. However, the appearance of a previously unknown collembolan as an occasional parasite that for years caused depression in a patient and continues to do so – so far a unique case – has now rightly gotten the attention of scientists.
The female patient, a 60-year-old married housewife had been suffering for two years from a “nervous disorder.” She had consulted various physicians, including dermatologists. However, none were able to determine the cause of the medical problem, which manifested itself as a weak, although, annoying itching, and considered being of a nervous type. In the end, the patient herself managed to detect the culprit, which resided in the genitals and anus, organs that are difficult to access.
Initially, the patient believed that the creatures causing her discomfort were lice. They caused irritation, especially at night. They crawled out of their hiding-places. She vaguely felt how she was being "stung” by these creatures, leaving small red pricks on the skin of her torso all the way up to the arms. In specimens that were killed in hot water, she discovered a glassy, sharp process on the abdominal hind parts, whose “sting” she rightfully attributed to her “nervous condition.
I can certainly relate to the red pin pricks and the glass parts (glitter?) that so many of us see.
A clinical and paraclinical investigation on a 80-year-old woman who claimed a "beetles attack". A 80-year-old woman presented with pruritus (itching), insomnia, anxiety, paleness, weight loss (7 Kg in 6 months), and loss of appetite. In the lumbar region, on the buttocks, on the right posterior hemithorax and interdigitally on both feet she had small ulcers of 0.5-1 cm in diameter, surrounded by an indurated congestive or cyanotic, ovoid area of 1 x 1.5 cm.
Small scars, with furfuraceous, dry and gray exfoliation on round and linear zones of 20-25 x 4-5 cm., were observed in the submammalian region. Linear short subepidermic truncated trajects (tunnels) were also observed.
The microscopic examination of the hypodermic material obtained by scraping, revealed an adult Collembola spp. insect, eggs, cocoons of different colours, piles, a pupa, an exuvium and larvae.
The histological investigation revealed thickening and erosion of the epidermis, and isolated or confluent blood gaps. In the epidermis spaces were present binding up larvae and nymphs. The horny layer was hyperkeratotic and included the adult insect. The blood gaps were probably caused by the histophagy of the insect. Intraepidermic bullae covered by a thin corneous layer and ulcers were also observed.
Histological Findings in the case of the 80 year old woman
(Note that Collembola go through many stages, these are what they found in the woman)
Six egg piles (shown on the right) were observed into the epidermic material. Four to 22 eggs were present in each pile, cemented together with a glue-like secretion.
The piles were elongated, round, with a small, cylindric, blunt and short appendix.
The piles measured 144-306 by 162-378 µm, while eggs (spherical to oval) were 35-140 by 35-123 µm (average 78 by 88 µm).
The remains of an exoskeleton that is left after an arthropod (insect, crustacean or arachnid) has moulted.
The exuvium , the larval or nimphal cuticle, was translucide,
very thin, composed of four distinct zones: a globulous apical pole,
a narrow neck, a brown collar and a big, thin body of the exuvium.
Larvae were divided in three segments: (shown on the right)
a) an anterior, mobile, cylindric one with two subterminally mandibular pieces;
b) a middle ovoid segment covered by a rigid cuticle;
c) a caudal segment, frayed, with three terminal sharp apexes
for the fixation to the substrate.
The pupa was brownish, oval, measuring 2,280 mm by 720 µm.
Taken from the 80 year old woman
This certainly would go a long way in describing all of the debris found in our bodies. All of the information regarding the 80 year old woman can be found HERE and you can click on the photos there for even larger images.
So, we have The National Pediculosis Association (NPA) study identifiying Collembola in 18 of 20 persons previously diagnosed as DOP (otherwise called crazy) including photographs of the Collembola, many professional researchers were involved in this study. If you take the time to look at the study you can see the difficulty involved in finding these little pests. We also have the cases in 1955 in Sweden with many folks being infected by Collembola and the case of the 80 year old woman as well. I suspect these cases are but the tip of the iceberg and these particular sufferers were lucky enough to be able to contact somebody that actually took the time to “look into things”. In other words, they found caring doctors with open minds, an extreme rarity indeed.
Meanwhile, Wikipedia makes the claim the 2004 Study was basically a farce. I know who I believe and it isn’t those controlling the information on Wikipedia. But let’s continue …
When I first came down with Morgellons I would take lots of baths and use different things like sea salt and stuff. I would notice this things moving which floated on the water. But the things were so small you couldn’t see them, you only knew they were there by the tiny ripples around them, in fact, you had to be at a 45 degree angle to really notice them but they were definitely alive and moving. In fact, I was corresponding with a woman on the yahoo finding1cure group just the other day who can flake them off her skin into water and see them exactly as I have described above, only she thinks they’re mites, my strong bet would be that they are Collembola and not mites. Since I still have light crawling I am going to do this experiment capture some of them and put them under the scope and will report my findings at a later time.
My crawling is basically a low boil now, not very noticeable most of the time. The first time I ever got rid of the crawling and realized there was at least a topical aspect to Morgellons was using the ESP Botanicals system. The Green Balm I would use and cover my body head to toe and after a matter of weeks the crawling was nearly gone. My mistake was not sticking with it as once I got feeling better and ran out I stopped. But I was so sick back then I wasn’t thinking clearly. I now have the complete New Hope Two Product line.
Also, Sulfur kills mites and I believe it kills Collembola as well. I learned about Azufre – Sulfur with Lanolin Bar (10% Sulfur) and still use it regularly. If you use this first try it on a small part of your body to make sure it’s not too strong for you, I have no issues with it at all. I use it in the shower, get wet, soap myself up, turn the water off and stand there for 3 or 4 minutes and then rinse. And for $1.95 a bar you can’t go wrong. It comes from Mexico and really, really works.
Finally, wash your clothes only in Dr. Bonners Peppermint Soap I cannot tell you how well this works. It has a strong mint smell to it and mint also kills mites, or anthropods such as Collembola. I have made this recommendation to people that have washed their bed sheets and told me that very night was there best nights sleep in years, it works. Some people use it in the bath but it must be diluted massively, such as only 1/2 cup. I recommend just washing your clothes in it. You can find Dr. Bonners at Whole Food stores and GNC stores.
I make no money off any of these products, nor do I from any products whatsoever.
I believe Collembola infestation is a major player in Morgellons disease despite the naysayers and those that have already become numbed to the subject. The evidence of infestation has been proven beyond a doubt in my mind. And as was stated in the NPA article linked above:
Altschuler asserts that there is more of a scientific basis for Collembola in humans than the entomologist and physician’s overwhelming acceptance of a psychiatric explanation (Delusions of Parasitosis) for people’s sensations of biting, stinging and crawling in their skin.
And finally, I would like to share with you the “other symptoms” the 18 individuals found to have Collembola infections suffered from. Here they are …
These individuals experience itching, stinging/biting, and crawling sensations on or under their skin, which are often associated with excoriations, discoloration, scaling, tunneling or sores. Their conviction that they are infested is reinforced by their observation of particles described as sparkly, crusty, crystal-like, white or black specks and/or fibers. Typically, these individuals have consulted extensively with general physicians, dermatologists, and entomologists (Kushon et al., 1993) who could not find physical cause for their complaints. Despite findings ruling out lice, scabies or other medical causes, patients refuse to accept the diagnosis of delusory parasitosis.
These people basically have Morgellons, only it wasn’t widely known in 2004. I would also bet most of the folks on the bird mite forums have Morgellons and not bird mites. Bird mites are easily spotted with the naked eye, I had them get on me at my initial onset and actually captured two of them.
This blog post is probably long enough at this point. One thing I have not covered is why we are infected with Collembola (why has this infection become so much more widespread all of a sudden). Also, I am BY NO MEANS SAYING Morgellons is merely a Collembola infection. They are in us for a reason (something came first) and they bring with them an interesting component to the mix. These issues will be covered in my Grand Unification Theory which is still forthcoming. I know this topic will bring some debate and that’s just fine. One wonders why there would be such deception on the part of Wikipedia, perhaps their fear is the world finding out why all of sudden we’ve become their dinner, and by the hundreds of thousands, probably millions worldwide.
I beleive the water technique can be used to identify a Collembola infection quite easily and will report my findings in short order. You might take a moment to watch this News Report VIDEO of families who believe they are infested with Collembola.
Before you react in anger over the title of this post it is important for you to know that I am a Morgellons sufferer and I am certainly not a Methamphetamine user nor do I think Morgellons is caused by Meth use. Have an open mind and be willing to learn, you are going to be very surprised I think. I will go where ever the facts go, the only stake I have in this game is the hope of a cure.
If you didn’t read my first blog post I strongly urge you to read it now, before continuing. My first blog post can be found here and it’s called Does this identification mean anything? I do not know. In summary, I discussed Dr. Wymore’s culturing of Pseudomonas Putida from Morgellons fibers and the ramifications which are many. It’s important to know that Pseudomonas Putida can and does turn things like oil and glucose into Toluene, a very toxic chemical. Remember that name, Toluene, it’s key to where we are going. However, if you do not take the time to read my first post you might as well stop reading now because you’re really not interested. If you want to understand this disease invest the 10 minutes it will take to read it.
WARNING SIGNS OF METH USE
Meth users will exhibit numerous signs of use, the most obvious of which are hyperactivity, incessant talking, and wakefulness. The drug produces a false sense of confidence in the user. Users will often have a loss of appetite and become extremely irritable and moody. Prolonged meth abuse can resemble symptoms of schizophrenia characterized by hallucinations, paranoia, and repetitive behavior patterns. Abusers develop delusions of insects under the skin known as “speed bugs” or “meth bugs,” which cause the user to pick at the skin incessantly resulting in open lesions. As the effects of the drug wear off, users may experience drug cravings, depressed moods, lethargy, and prolonged periods of sleep lasting 24 hours or more.
The part in “red” above is not the surprise, it coming though, sit tight. However, surely if you have been following the Morgellons mystery you have heard Morgellons written off as merely a bunch of former or current methamphetamine users. It’s quite common for meth users to totally freak out and claim that there are bugs crawling under their skin. Sound familiar? However, I am about to make a rather shocking claim that there is in fact a connection between Morgellons and Methamphetamine use, but not the connection you might think. Maybe there is a reason Meth users and Morgellon sufferers share these common traits? Hold onto your seat, this is going to get very interesting.
There is a very interesting ingredient that is used by those running meth labs and that ingredient is Toluene. Yes, the very same chemical that Pseudomonas Putida can both create and bioremediate (degrade). I provided links with examples of Pseudomonas Putida creating Toluene in my first blog post. If you have any doubt regarding the connection between Pseudomonas Putida and Toluene click HERE.
Meth “cooks” use most or all of the ingredients below to make methamphetamine. Most all of the ingredients below come with a warning label telling the consumer not to ingest the product.
Ether (starting fluid)
Camp stove fuel
Rock, table or Epsom salt Red Phosphorous
|Toluene (found in brake cleaner)
Red Devil Lye
Lithium from batteries
Now, if that is not enough to make you swear to yourself never to use meth then I don’t know what is. Interestingly however, our friend Toluene shows up in the meth ingredient list (which they get from brake fluid). Could there be a real connection here? Is this yet just another coincidence? Both Morgellons sufferers and Meth users claim to have bugs crawling under their skin and can end up covered in lesions and sores. “But there are so many chemicals in that list above” you say, “It could be any of them, or, meth users could be simply delusional and whacked out due to the overwhelming toxicity of meth”. I don’t think Meth users are delusional when it comes to “meth bugs”. No, they feel the same thing we do because they are ingesting Toluene into their bodies. But in order to prove that we would need to have evidence that Toluene is known to cause such manifestations and symptoms.
Let’s start out with a series of links
Okay, so there’s some information regarding Toluene and it’s ability to cause itching, dermatitus, and other skin ailments, but is that enough evidence?
Now, this find was really kind of “gem” for me. Was I asleep at the switch? I had never heard of this, but it sounds exactly like Morgellons. From “An Overview of Neuro-Cutaneous Syndrome (NCS)” which can be found HERE.
A detailed analysis of the clinical history of a random sample of 50 NCS patients (9 males, 41 females) is reported. Symptoms are classified into six categories, neurological, dermatological (including opportunistic skin infections), systemic, oral, allergic and general. The most common symptoms in each of these categories in the same order are pin prick and crawling sensations, skin lesions and sores, respiratory and bowel disturbances, gum disease, sensitivities to light, noise and mold, and fatigue and insomnia. Symptoms were relatively similar in both sexes. These results are tabulated and their biological foundation explained. The misdiagnosis of NCS by medical professionals is discussed. NCS symptoms in toothless patients and those on recreational drugs are described. Over 360 dental toxins are placed in four major categories and their mode of action explained. Incubation period varied between a few hours to 28 years. Our protocol for rehabilitation is included. All patients following and completing our rehabilitation protocol recovered.
You will really want to visit the link the NCS site after finishing this post, and we’re almost done, but there’s more evidence here, and that is Toluene and it’s relationship to NCS, amazing, it is mentioned at least four times in the article such as “Toluene is a known potent nerve toxin”. Take a look at this quote from the article.
Neurological symptoms are the most prevalent and most diagnostic of NCS. All NCS patients experience skin irritation manifesting as pin prick or crawling and movement sensations; usually both (Table 1). These sensations are caused by damage to nerve cells caused by such dental materials as Toluene2,3 and methacrylates.7 Damage to the nervous system also manifest in loss of memory, brain fog, poor concentration and vision problems in many NCS patients. Males appear to be more susceptible to burning sensations and body tremors than females. The pin prick and crawling sensations were most severe (Table 1) and are considered the primary diagnostic symptoms of NCS even in absence of all other symptoms.
One other thing I haven’t mentioned is that Meth users suffer terrible tooth decay, the pictures are to gruesome to even post and that many Morgellons Sufferers also suffer sudden onset tooth decay, NCS also talks about this. The NCS site even talks about Collembola.
Opportunistic infections with springtails (Collembola) from NCS patients’ sores, especially scalp sores, are not uncommon (Table 2). Arthropods are attracted to open sores, especially those infected with micro-organisms, for feeding, moisture and possibly nesting. Facial sores of one NCS patient living in an old musty home in a wooded area in Oklahoma included a tick, an ants, a caterpillar, thrips, oribatid mite, and parts of a wasp, cranefly and other insects.4 Other arthropods collected from open facial sores of NCS patients include fleas, beetles, winged flies and midges, and spiders2,3. It should be noted that all kinds of opportunistic infections represent only aggravating but not causal factors of NCS sores.
This is either an incredible set of coincidences or there really is something here. What have we learned?
From my First Post
From this Post
We are vastly moving beyond the point where we can believe this is all sheer coincidence. If you look at the material safety sheet it can cause all kinds of things, including heart problems, central nervous system problems, and even death, not to mention all the facts that point to it being the cause of our condition. Could NCS really be Morgellons?
In our case however, if it is the Putida, we are different than the Meth users in this regard because Putida is a soil based organism and can be strong attractant to Collembola and other bugs, we not only have the nerve damage from the Toluene that makes us feel like we have bugs, but potentially, many of us also do have real bugs.
Finally, I’ll leave you with a quote from this page I found, I’m not sure if it is true, but have no reason to think it is not. From “America’s Next Top Bacteria”
Pseudomonas putida is a bacteria that was discovered fairly recently. 1991 is the earliest report of this bacteria. In 1992, French scientists found a bacteria that fed specifically on oil. This was the first report of any type of oil eating microbes. Later, in 2001, Elf Aquitaine researchers found this bacteria. They used it as a more efficient way to decontaminate landfills. This was the first large experiment recorded using pseudomonas putida. The experiment lasted a year ad went of without a hitch. The experiment was very successful. Then in 2002, a small group of scientist from The United States Department of Energy discovered a very similar bacteria that removes pollutants from coal. This bacteria was used to produce a cleaner burning coal.
Funny the timing of 1991 which is relatively new and in agreement with the Morgellons timeline and in France of all places, where Morgellons is first referenced in medical history during the 1600’s.
I guess this could all be simply cleared up by finding out if in fact we have toluene in our blood or skin or even Putida for that matter. Who knows, some environmental Toluene poisoning from coal burning? Perhaps it’s not doctors we need, but Toxocologists and Bioengineers to track this down, if you read any of the documents in the Bioengineering fields you can see how advanced the technology is, they of all people ought to be able to figure out what is the matter with us.