Archive for the ‘Neurocutaneous Syndrome’ Category

CBS News airing program on Omar Amin Phd and Neuro-cutaneous Syndrome

A Special thank you goes out to Pamela Mae of http://www.morgellonsfocusonhealth.com for alerting me to this important news story.

CBS News airing program on Omar Amin Phd and NCS on Tuesday night at 10pm Neurocutaneous Syndrome and Morgellons

This is the summary Pamela Provided to me in the Email.


Very nice. Good Press. Sound Science.

We know that each case with Morgellons is slightly different. But all present with Hyper-toxicity and Fungal involvement. I do not believe that Omar Amin Phd is saying ‘All’ Morgellons patients have ‘Only’ Hyper-toxicity from Dental products.

Because Omar Amin Phd is a Parasitiologist, I believe he is aware that because of the Toxic state of NCS or Morgellons patients that the body has become unable to move the toxins out.

The body has then developed Fungal involvement,in some cases bacterias,and in other cases an external parasite. As is evidenced in the work by Nancy Guberti CHN,by her  ‘Functional Medicine Testing’ done on Morgellons patients.

It is my own personal belief that  Dr Fry’s,’ Protomyxzoa’ which is a blood based newly discovered protozoan ,also needs to be eventually dealt with,and which comes into remission when all of the above are controlled dealt with or eradicated.

Our health and welfare are not based on the CDC, the Government,or upon Foundations which really say or do nothing.

It is driven by the Lord our God,and the wisdom and it’s responsibility given to good individuals who will move sound science to help those in need.

Congratulations ParasiteTesting.com and to you Omar Amin Phd.

Blessings
Pamela Mae


STORY FOLLOWS …

Scottsdale doctor claims to find cause of skin crawling disease

See http://www.kpho.com/story/16666981/cdc

SCOTTSDALE, AZ (CBS5) -

“It will make you crazy because that’s all you can think about,” said Stacy Hillman, who describes 2011 as the year from hell. “I was suicidal,” she said.

Last March, Stacy started feeling pin pricks on her skin and a few weeks later, lesions covered her entire body.

She tells CBS 5 News, “It felt like there was some type of bug, thousands of bugs, crawling all over my face.”

Stacy and her husband, Jeff, spent thousands of dollars going from doctor to doctor, trying to get help.

Jeff remembers those visits. “They basically would look at her like she was crazy.”

Stacy shared that skin-crawling sensation with thousands of people all over the country.

It’s common name is Morgellon’s syndrome, which the CDC doesn’t recognize as legitimate. Critics say it’s a mental issue.

Stacy fires back, “Pray you don’t ever have it for a day or a week, and then talk to me and tell me if it’s all in your head.”

The Hillman’s found relief in Dr. Omar Amin in Scottsdale.

He’s a parasitologist, but since so many sought his help believing they were infected by bugs, he wanted answers.

“I’m a hard-core, old-fashioned scientist,” said Amin.

Amin believes the problem actually comes down to dental material, that is not compatible with the body’s immune system.

The exposure to those toxins causes nerve damage, which makes it feel like the skin is crawling.

Amin says, “the nerve cells will misfire. You have no normal nerve impulse anymore, and that misfiring will cause the sensations of movement and pinprick.”

The lymphatic system tries to eliminate those toxins through the skin, which breaks out in sores and invites other biological organisms to nest, like spores which grow long-stemmed fungus.

The concept is new to science and easy to dismiss, but Amin warns thousands are at risk.

“Everybody who has dental work, and that’s just about everybody who lives in this culture of ours, is an open game,” he said.

Amin calls the disorder, NCS, for neurocutaneous syndrome, and sees patients from all over the world.

The Hillmans came to seek treatment from Los Angeles. Stacy is taking homeopathic remedies, and her dentist has started to replace her fillings with more compatible material based on her individual blood tests.

If you would like more information on Amin and his Parisitology Clinic in Scottsdale, visit his website here: http://www.parasitetesting.com

Another Theory for Sufferers to Consider

Crawling Sensations, Fibers and Other Noted Morgellons Syndrome Symptoms: Another Theory for Sufferers to Consider

by Joseph Keleher

I am not a medical professional. Let me repeat that. I am not, never have been and have no desire to ever become a medical professional. I might best be labeled a reluctant participant, as I suffered from Morgellons Syndrome Symptoms.

I have done my best to document what I went through (see Keleher 2008 “Hell and Back Again”). I wrote about connections to mercury and these horrific symptoms. I’ve angered medical professionals and sufferers. I’ve lost some old friends. I’ve found some new friends. I’ve written, emailed, and spoke on the phone with others who suffer. I’ve cried a bunch and still do (I don’t exactly know why).

I know it is possible to get well. I know what I suffered from and what I think most are suffering from is mercury as a neurotoxin. It is treatable with chelation and changes in lifestyle.

What I share are my thoughts. Yes, it is speculation. It is theory related to symptoms I had and you may still have. In reading be warned there are many “I think”s and “I believe”s. It’s all theory; it may be right or it may be wrong. Still, I feel compelled to share.

The Cause

I believe the recent growing numbers of those suffering from Morgellons symptoms is due to two ingredients- toluene and mercury.

Toluene is found in glues, methamphetamines and cocaine. I believe the toluene found in dental adhesives, and connected to symptoms by Dr. Omar Amin, correlate with a surge of sufferers of these symptoms (and possibly via prenatal passage the exponentially growing number of children with autism, ADD and ADHD).

I think toulene interacts with mercury to allow passage of this toxin into the nervous system. Toluene may dissipate after carrying mercury into the system leaving deposits of mercury. The primary source of mercury in the mouth (though there are other sources causing mercury increases including some skin creams, coal burning power plants, and historic mining activities) decreases as secondary deposits in the nerves increase.

The associated symptoms of this neurotoxin can be viewed as occurring in stages with new symptoms added as secondary deposits increase. The stages, based partly on my own experience, but also drawing from the experience of many others, might look like: 1) depression and panic attacks (typical symptoms of neurotransmitter blockage), 2) bloating, heart palpitations, and exhaustion, 3) crawling sensations, 4) fibers and skin lesions….and so on.

All of the symptoms noted with Morgellons, except crawling sensations and fibers, have been shown to be associated with mercury poisoning; because of this, I want to share how I think these two symptoms form.

Crawling Sensations

In the earliest onset of the “bugs”, I had faint flea-like zips across my scalp. I believe this was mercury pathways forming within the nerve cell network under my skin.

As the secondary levels of mercury increased, the sensations of crawling began. My crawling always started each night at the rear, right occipital area of my scalp (in journaling, at that time, I referred to the spot on my scalp as the “Mother Ship”); this area correlates with the low spot of my skull when resting (I have a preference for sleeping on my right with my head cocked back). The extreme density of mercury I believe was pooling.

Crawling typically began as the sun was setting and there was a change in temperature. Within weeks, the crawling expanded to my groin and bottom of my feet. Eventually my whole body had the sensations. It was during this time that I was diagnosed with sheet mites and scabies (and found out about something called Morgellons).

I believe as levels of mercury further increased, the crawling became more pronounced and expanded throughout my body. The sensation was especially disturbing when it began in my ear canals and nostrils.

I think the crawling is due to the expansion and contracting of mercury within the network of nerves. I picture it as one spider-shaped cell after another filling and twitching as mercury moves through (causing a kind of “cartoon effect” movement); one sufferer described this observed movement as certain proof of parasitic infestation. I still get a faint twitch occasionally at the base of my feet (but it feels different and I suspect it relates to nerve repairs and not mercurial movement).

If the Morgellons symptoms occur in stages related to increases in mercury, then I arrived at one of the middle stages. I never found fibers projecting from my skin or had any lesions. I’m thankful for this.

Fibers

I think the fibers are the result of repeated cell damage repairs from mercury expansion and contrasting through the nerve system. A weak spot may have mercury break through and this will “scab” over. The fibers eventually poke through as they have nowhere else to go.

Final Words

I know there are many theories out there. Mine is one among many. Take it as such.

Morgellons is an especially cruel condition. In addition to the symptoms, the medical community has not been especially kind. The sensationalism of the media doesn’t help. Isolation and lose of connections to other people is difficult. There is one thing though that especially irritates me; I cannot stand that there are people taking advantage of the vulnerability of many sufferers. For those snake oil sales people making money off this condition, someday you will get yours; it is true what we do to others we do to ourselves.

For those suffering, be careful of the sharks, keep faith that answers will come soon, and always, always keep an “attitude of gratitude” for all you have. You are not alone and things will get better! I wish each and ever one of you a clear path towards health.


Thank you so much Joseph for another excellent and thought provoking article. Your thoughts really make me wonder if the NAC I take is a major player in my feeling better? Here are two other excellent articles written by Joseph that the reader might also want to examine.

Morgellons Disease – A Patient’s Perspective
http://www.arizonahomeopathic.org/alternative-medicine/morgellons-disease-a-patients-perspective/

Keleher 2008 “Hell and Back Again”
http://members.cox.net/llyee2/NCS_article_by_joe.pdf

Neurocutaneous Syndrome (NCS)

I received permission from Dr. Omar M. Amin to republish this article. In some of my future blog posts I will be revisiting the subject of toxicity again. I have made reference to NCS in several posts in the past especially in connection with Toluene and other compounds.

 

On the Diagnosis and Management of Neurocutaneous Syndrome (NCS)
A toxicity disorder from dental sealants

Omar M. Amin, B.Sc., M.Sc., Ph.D.*
__________________________________________________________
*Parasitology Center, Inc., 11445 E. Via Linda, # 2-419, Scottsdale, AZ 85259-2638  USA
Phone: 480-767-2522; Fax: 480-767-5855;  E-mail: OmarAmin@aol.com
Web address: http://www.parasitetesting.com/

 

Abstract:

Neurocutaneous syndrome (NCS), a newly discovered toxicity disorder, is characterized by neurological sensations, pain, depleted energy and memory loss as well as itchy cutaneous lesions which may invite various opportunistic infections. Components in the calcium hydroxide dental sealants Dycal, Life and Sealapex have been identified as sources of the observed symptoms. Sulfonamide and neurological toxicity issues are discussed and three case histories are presented. Additional notes on zinc oxide, Fynal, IRM and Sultan U/P sealers are also included. Diagnostic and management protocols at the Parasitology Center, Inc. (PCI) are proposed.
 

Introduction

The original description of the neurocutaneous syndrome (NCS)1 was introductory in nature.1 Examination of many NCS patients and a careful study of their symptoms, exposures, clinical conditions and histories made it possible to identify the underlying cause of the syndrome and proceed with its management.

 

Materials and Methods

Patients were personally evaluated and their clinical history, records, symptomology and exposures carefully examined. Specimens provided or collected at the Parasitology Center, Inc. (PCI) were studied. An NCS status was only determined based on symptoms and determination that one or more of the suspect sealers have been used on prior dates. Sensitivity to sulfa and elevated levels of sulfa in the blood were used as a confirmation of sulfonamide toxicity. Continuing patients follow our recommendations for dental rehabilitation, extraction of suspect liner(s), and replacement with ethyltoluene sulfonomide (ETS) and zinc oxide free sealants. A list of vitamin/mineral supplements for patient use during the transitional period and another list of substitute sealants are provided.  Patients are followed up to monitor and insure the resolution of  symptoms.

 

Results and Discussion

 

The Neurocutaneous Syndrome

The disorder is double faceted with dermatological and neurological symptoms compatible with classical sulfa toxicity. The latter is characterized by changes in blood values, photosensitive reactions, allergic vasculitis sores, bacterial flora changes, and redness of the skin, which may lead to liver and kidney failure.2 The neurological aspects are characterized by pin-prick and/or creeping, painful and irritating movement sensations, often interpreted as parasite movements in various body tissues and/or cavities.. Movement sensations are either unipolar or bipolar and may proceed horizontally or vertically. They may manifest as variably shaped bruises or waves of elevated ripples or channels. In no case was the movement sensation related to parasites1. Neurological symptoms may also include loss of memory, brain fog, lack of concentration and control of voluntary movements.

NCS_fig1

Fig. 1. Early NCS sores on the thigh of KM. She was born in 1964, treated with Dycal in two teeth in 1982 and in one tooth in 2002. Neurological symptoms in upper quadrant started in 1997. Cutaneous symptoms began in Spring 2002 preceded by extensive treatment with topical sulfa preparations for possible mite infestation. Dycal was removed in December, 2002 and recovery is in progress.

The cutaneous aspects include small itchy sores (Fig.1), inflamed often elevated pimples (Figs.2,3), and fully inflamed and painful open/amorphous mucoid lesions that often enlarge and coalese (Fig.4). Histopathological sections of lesions (Fig.5) show superficial and deep perivascular infiltrate of lymphocytes, accompanied by interstitial deposits of granular mucin material. Eosonophils are usually present within the inflammatory infiltrate and foci of epidermolytic hyporkeratosis are often identified within the epidermis (Fig.5). Lesions may also be on the scalp where they may be associated with infestation of springtails (Collembola). 1 In many cases, lesions are associated with edematous reaction usually in the arms and legs (Fig.6). Blood vessels may also become enlarged and elevated, and head may become hot and turn red. The gum tissue and the teeth and oral mucoid secretions may turn gray and become compromised first and stay compromised the longest. The above creeping sensation is clearly distinguished from these caused by nematodes such as Toxocara canis3 or Dioctophyme sp.4

General symptoms usually include fatigue, compromised immune system, psychological trauma and loss of self- esteem. The depressed immune status in most patients appears to pre-empt them for opportunistic infections.

 

Compounding Factors

NCS_fig2

Fig. 2. Elevated sores on the forehead of KM (Fig.1); note the hot red color of the skin.

NCS_fig3a

Fig. 3. Diffuse NCS sores covering the whole body that was treated with Dycal in 1985 (Case no. 1)

While NCS itself is not a contagious condition, superimposed opportunistic infections on open sores may be. Initial infection with fungus or bacteria appear to attract subsequent infestations with many arthropod species, especially springtails (Collembola: Insecta).1,5,6,7 Black specks associated with such infections appear to be metabolic waste (fecal elements) of these organisms or mycelial masses of certain fungal species. Staphylococcus aureus, S. haemolyticus,  Streptomyces spp., Candida albicans and Madurella spp. among others, have been identified from cultured swabs taken from sores of various NCS patients. These opportunistic infections have been shown to aggravate the cutaneous symptoms of NCS patients. The Madurella infections are usually associated with black grains of mycelial masses that may be related to the black specks and fibers observed by some NCS patients. The healing of certain patients lesions9 was observed to be proportional to the exit of remaining fibers from lesions.3 Patients experiencing complete remission remain susceptible to fungal promoting conditions in damp, shaded, moldy places.

Arthropods identified from sores include fleas, caterpillars, wasps, ants, beetles, winged flies, midges, thrips, ticks, mites, spiders, and springtails.1,4 Springtails may have close association with sores in many NCS patients but they, and other opportunistic infections, are not causal factors of NCS sores.

     

The Sealants

The three major calcium hydroxide sealants causing NCS (Dycal, Life and Sealapex) considered 9 include only about 50% calcium hydroxide in the catalyst (Table1). Of the components common to all three sealants, ethyltoluene sulfonamide as well as zinc oxide are considered most toxic. Toluene is a known potent nerve toxin.10 The sulfonamide component of this compound causes a sensitivity allergic- toxic reaction ultimately manifesting as the vascular mucoid sores characteristic of the NCS, especially in sulfa sensitive patients.


NCS_fig4

NCS_fig5

NCS_fig6

Fig. 4. Mucoid NCS/lesions on the face of MM.  She was born in 1950, poisoned with Fynal in six teeth in 1981 and in one tooth in 1986 as well as with Life in two teeth in 1985 and 1988.

Fig. 5. Histopathological section of one of the roughly 300 sores covering the body of SK. She was born in 1956 and reacted with typical NCS symptoms to a  zinc oxide cement (combined with Durelon) underneath a total veneer job in 1982. The section shows hyperkeratosis like perivascular dermatitis with eosinophils.

Fig. 6. Cutaneous sores and swelling in the right hand and arm of DB. Born in 1965, DB had 10 amalgam restorations in 1982 and 1983 using Life. She started experiencing symptoms including ulcerated rash all over the body, unilateral edema and pin-prick and subcutaneous movement sensations in 2001-2002. Life is being removed and recovery is in progress.

Zinc oxide was shown to be genotoxic11, cytotoxic12,13, killing microphages14, and causing chronic and fibrous inflammatory reaction15,16 ulcerations16 and osteosclerosis.17 Additionally, the toxic effects of zinc oxide and calcium hydroxide were shown to be similar.18,19 Calcium hydroxide was shown to cause periapical inflammation, typical granuloma and partial lack of healing.20 Titanium dioxide and Barium ions (Table 1) were also shown to provoke strong foreign body and bio-incompatible reactions in live tissue.21,22

Cytotoxicity of Dycal, Life and Sealapex was clearly demonstrated invivo and invitro in various tissues.23 Sealapex was shown to cause severe inflammatory infiltration15,24,25 and edema25 accompanied by subcutaneous tissue necrosis15,26 and progressive differentiation and reaction of monocytes, macrophages and epithelial cells27. The final phase of the inflammation is characterized by an intense granulomatus reaction especially in epithelial cells causing various intensities of irritation.28The cytotoxicity29,30 and neurotoxicity31 of Sealapex was well demonstrated in various mammalian systems.

As with Sealapex, Dycal was also shown to cause hemorrhage and acute to consistent inflammatory cells16,32,33 necrosis,16,32,33 tissue loss,33 karyorrhexis,16 neurotoxicity.34 and formation of serous exudates.16 Life has been the least researched sealant. It, however, has the same toxic ingredients, i.e., ethyltoleune sulfonamide and zinc oxide, as Sealapex and Dycal and has been associated with classical NCS symptoms in some of our patients, e.g., DB (Fig.6) and MM (Fig.4).

Sealants not containing ethyltoluene sulfonamide but including zinc oxide and eugenol have also been associated with NCS cases.These include Fynal(>75% zinc oxide), IRM and Sultan U/P (<50% zinc oxide). Fynal was associated with the cases of MM (Fig.4).  Similarly, IRM (by Dentsply caulk) and Sultan U/P (by Sultan Chemists) were associated with classical NCS symptoms in some of our patients.

 

chart

Case Histories

Case #1.

    A white female born in 1951. In 1985 she underwent dental repairs, which included the use of Dycal in 20 teeth. The lady is allergic to sulfonomides, with IGE values reaching 5000. Every dental treatment was followed by aggressive skin reactions of allergic and toxicological nature (Fig.3). All tests for parasites were negative. Her symptoms fulminated into full blown typical sulfa toxicity reactions including oozing skin and nasal sores with bloody scabs and smelly discharge and an infection with S. aureus ( Fig.7). Other symptoms included loss of memory, kidney pain and urgency, sensitivity to light and electricity fields, pin-prick and moving sensations under the skin, and swelling. After each treatment, the white female felt totally knocked out with breathing and talking difficulties. She subsequently developed intestinal problems and her skin sores flared up with unbearable and unresolved itching. Photosensitive reactions presented as blotchy skin ( Fig.7) with severe burning sensations in the face, throat and chest.

       Dycal was removed in 1991-1992 and initially replaced with Harvard cement. The lady was confined to bed with whole body musculo-skeletal system pain, bowel disturbances and signs of polyneuropathy. Shortly after the removal of the Dycal in February 1992, most of her sores and rashes disappeared and she could tolerate sunlight (Fig.8).

NCS_fig7a

NCS_fig8a

Fig. 7. Case no. 1 before treatment; note the hot red face.

Fig. 8. after recovery.

 

Case # 2.

       Born in Chicago in 1965, JM was a healthy active Caucasian woman until she started experiencing her first symptoms in 1991. By then, she already had 17 fillings. No sealants were used in one filling; Dycal was used in the other 16. Her earliest symptoms appeared as skin break outs on the face and neck, which was recurrent over the following 9 years, accompanied by body tremors, sleeplessness and joint pain with occasional vomiting of black bile. Thrush appeared in the mouth and around the lips. Pain at the teeth roots persisted throughout the nineties associated with rapid major decay. A sensation of prickling pain with a pressure and movement under the skin, urticaria and skin ulcerations would last for weeks or months. JMs body showed random swelling with red marks in serpentine-like shapes. The swellings eventually bottlenecked at the knees and ankles. The chest burned and hurt with strange fits of coughing. JM then started losing hair as she experienced night fevers and sweats, and peeling of the skin.

       During the early 1990s JM was medicated with various antibiotics, antiparasitics and herbal remedies. She experienced some anti-inflammatory relief and occasional temporary clearing of ulcers after which ulcers returned and lasted longer. In 1998, massive ulcers appeared on JMs face at the nasiolobial area and on the skin ( Fig.9). A CBC in 1999 was unremarkable except for a high level of Alpha 1- Globulin of 0.5 (Normal range 0.2-0.4) and low levels of IgA of 99 (normal range 60-400) and IgG of 724(normal range 700-1500). The right ocular cavity was severely painful and JM was beginning to lose her eyesight.

     A major dental repair was completed in 2001 when Dycal was removed from all 16 teeth. Initially, JM experienced a few episodes of sickness, sweats, and vomiting. After the fourth visit, her eyebrow area had a dramatic reduction in swelling, sensation of movement and in the red-hot congestion of her face. JMs teeth were subsequently rebuilt with gold onlays section by section. By the end of the total repair, Nov.2001, JM has regained her normal skin (Fig.10) with no movement sensations or pain anywhere in her body. This state of total resolution has lasted to date without regression or relapses.

NCS_fig9

NCS_fig10

Fig. 9. Case no. 2 (JM) before treatment; note the lesion
on the right cheek and the hot red face.

Fig. 10. JM after recovery.


Case #3
.

       LG, a medium- built white American born in 1957, was in perfect health until September 18, 1998 when she had a filling in her tooth no. 18 using Dycal as a liner. She experienced severe headache within 2 hours. By 6:00 pm she was vomiting and delirious with the headache persisting. Her blood pressure then was monitored at 169/108 and remained high for the following three years despite repeated attempts to control it with Atenenol and Diazide. LG never experienced high blood pressure or headaches before. An MRI scan was negative. In 1999 LGs health deteriorated progressively with arthritis- like symptoms in her back, heart palpitations, mitral valve prolapse, fatigue, abnormal pap-smears including pre-cancerous cell abnormalities, night sweats, missed periods, and severe depression.  By March 2001, LG, who normally weighed 120 lbs has lost 20 lbs.

       In April 2001 lesions started appearing on LGs face, which quickly became red-hot.  Her legs became swollen and painfully burning. By May 2001, LG had several open lesions (6 mm to 2 cm in diameter) with some surrounding erythema, on her face and scalp. Her cheek pulsated as the facial lesions seemed to track to the chin (Fig.11) where the most fulminating lesion was; nearest to her teeth. The face was burning hot. Springtails (Collembola) and fibers were recovered from these sites. At that time, she showed low lymphocytes of 15.0% (normal 20-43%), high granulocytes of 77.1% (normal 51-74%) and high rheumatoid factor of 22.6 (normal <20 IU/ml). She also tested negative for all communicable diseases then. Her weight dropped to 92 lbs as she started experiencing movement sensations under the skin of her arms, face and scalp. Grayish pustular secretions oozed and moved down from the bloody lesions on the scalp and face. The lesion then extended to her legs.

       In January 2002, LG was diagnosed with NCS by OMA. She was allergic to sulfa and sulfonamide compounds. Following our protocol, LG had the filling and the Dycal liner removed from tooth #18 in April 2002. These were replaced with Starflow and Aria (a combination of Bisgma, Tegdma, Lidma and catalysts). Our recommended vitamin supplementation program was initiated then. By May 2002, all symptoms were resolved (Fig.12). Constitutional and neurological functions as well as psychological, emotional and energy levels were restored to normalcy.

NCS_fig11

NCS_fig12

Fig. 11. Case no. 3 (LG) before treatment.

Fig. 12. LG after recovery; note the return of the natural
baby skin back after healing of all facial lesions.

 

Conclusion

The toxicity of Dycal, Life and Sealapex has been well demonstrated in invivo and invitro studies of various animal and human models by many workers. The toxicity assumed cytotoxic, genotoxic, neurotoxic, phototoxic, necrotic, and inflammatory manifestations compatible with the pathology and symptoms observed in NCS patients. Ethyltoluene sulfonamide, common to all three sealants, is considered the primary cause of the NCS. The toluene component, a known nerve toxin, is believed to be responsible, at least in part, for the neurological symptoms. Neurological abnormalities are related to nerve damage associated with vasomotoric reactions due to a direct influence on the peripheral nerve endings.35 The sulfonamide component is the cause of the cutaneous symptoms, especially in sulfa-sensitive patients who usually had elevated sulfonamide/sulfa levels in blood tests and allergy to sulfa in skin sensitivity tests. The relationship between sulfonamide and phototoxicity has been well established.29 Resolving the symptoms (effect) by removing the sealants (cause) in patients undergoing treatments, confirms this cause-effect relationship.

The nature of causation of NCS precludes contagious transmission. Any similarities of symptoms among partners within the same household are traceable to the transmission of opportunistic infections, especially fungi.

It is recommended not to rehabilitate more than two or three teeth per month. The patient is given a list of vitamins and other supplements to take during the procedure and for the following few weeks until symptoms are completely resolved. After reaching the state of normalcy, the patient may still retain some sensitivity to moldy places lacking sun and fresh air circulation.

After additional test results become available and a satisfactory diagnosis of an NCS case is made at the Parasitology Center, Inc. (PCI), arrangements for dental rehabilitation are made and patient prognosis is monitored.

Acknowlegment

      I am grateful to Marie Erixon, Nordea, Sweden for her contributions to the better understanding of issues related to NCS.

References

1. Amin OM. Neuro-cutaneous Syndrome (NCS); a new disorder. Explore 2001; 10: 55-56.
2. Ockert K. Filling caused serious reactions. Trandlakartidningen 1994; 86: 470. (in Swedish).
3. Garcia LS. Diagnostic Medical Parasitology. Wash, DC: Am Soc Microbiol Press, 2001.
4. Urano Z, Hasegawa H, Katsumata T, Toriyama K, Aoki Y. Dioctophymatid nematode larva found from human skin with creeping eruption. J Parasitol 2001;87: 462-465.
5. Amin OM. Facial cutaneous dermatitis associated with arthropod presence. Explore 1996; 7: 62-64.
6. Frye FL. In search for the haphazardly elusive: a follow-up report on an investigation into the possible role of collembolans in human dermatitis. Vet Invert Soc Newsletter 1997; 13: 10-13.
7. Janssens F. Checklist of the Collembola: Collembola in association with man. http//www.collembola.org/publicat/sidney.htm 1999-2003; 10pp, and per comm..
8. Mahon CR, Manuselis G Jr. Diagnostic Microbiology. Philadelphia: WB Saunders Co, 1995.
9. Draheim RN, Murray AJ. Compressive strength of two calcium hydroxide bases. J Prothet Dent 1985; 54: 365-366.
10. Burry MB. Neurodevelopmental toxicity of toluene.M. Sc. Thesis: Seattle, Univ Wash, 2001.
11. Tai KW, Huang FM, Huang MS, Chang YC. Assessment of the genotoxicity of resin and zinc-oxide eugenol-based root canal sealers, using an in vitro mammalian test system. J Biomed Mater Res 2002; 59: 73-77.
12. Wright KJ, Barbosa SV, Araki K, Spangberg LS. In vitro antimicrobial cytotoxic effects of Kri 1 paste and zinc oxide eugenol used in primary tooth pulpectomies. Pediatr Dent 1994; 16: 102-106.
13. Pissiotis E, Spangberg LS. Toxicity of pulpisad using four different cell types. Int Endod J 1991; 24: 249-257.
14. Sadeghein A, Bolhari B, Sarafnejad A. A comparison of the effects of three endodontic sealers on adherence of mouse peritoneal macrophages. J Calif Den Assoc 2001; 29: 673-677.
15. Soares I, Goldberg F, Massone EJ, Soares IM. Periapical tissue response to two calcium hydroxide-containing endodontic sealers. J Endod 1990; 16: 166-169.
16. McShane CJ, Stimson PG, Bugg JL, Jennings RE. Tissue reactions to Dycal. J Dent Childr 1970; 37: 466-474.
17. Erausquin J. Periapical tissue reaction to root canal fillings with zinc, titanium, lead, and aluminum oxides. Oral Surg Oral Med Oral Pathol 1970; 30: 545-554.
18. Berman DS. Pulpal healing following experimental pulpotomy. Brit Dent J 1958; 105: 7-16.
19. Berman DS, Massler M. Experimental pulpotomies in rat molars. J Dent Res 1958; 37: 229-242.
20. Weinstein R, Goldman M. Apical hard-tissue deposition in adult teeth of monkeys with use of calcium hydroxide. Oral Surg Oral Med Oral Pathol 1977; 43: 627-630.
21. Bennatti-netto C, Bramante CM, Ber-Bert A, Lia RCC. Reacao do tecido conjuntivo subcutaneo de rato ante a implantacao dos materials components do cimento AH-26. Rev Bras Odontol 1982; 39: 11-20.
22. Smith JW, Leeb IJ, Torney DL. A comparison of calcium hydroxide and barium hydroxide as agents for inducing apical closure. J Endod 1984; 10: 64-70.
23. Topalian M. Effecto Citotoxico de los cementos selladores utilizados en endodoncia sobre et Tejido periapical. Endodocia-Caracas 2002, http://www.carlosboveda.com; 48pp, and per comm.
24. Buntak-Kobler D, Prpic-Mehicic, Najzar-Fleger D, Katunaric M, Talan-Hranilovic J, Suman L. Cytotoxicity of Ca(OH)2 endodontic sealers on connective, muscle and bone tissues. Acta Stomatol Croat 1993; 27: 175-180.
25. Sonat B, Dalat D, Gunhan O. Periapical tissue reaction to root fillings with Sealapex. Intern Endod J 1990; 23: 46-52.
26. Bezerra LA, Leonardo MR, Faccioli MR, Faccioli LH, Figueiredo F. Inflammatory response to calcium hydroxide based root canal sealers. J Endod 1997; 23: 86-90.
27. Tronstad L, Barnett F, Flax M. Solubility and biocompatibility of calcium hydroxide-containing root canal sealers. Endod Dent Traumatol 1988; 4: 152-159.
28. Zmener O, Guglielmotti MB, Cabrini RL. Biocompatibility of two calcium hydroxide-based endodontic sealers: a quantitative study in the subcutaneous connective tissue of the rat. J Endod 1988; 14: 229-232.
29. Beltes B, Koulaouzidou E, Kotoula V, Kortsaris AH. In vitro evaluation of the cytotoxicity of calcium hydroxide-based root canal sealers. Endod Dent Traumatol 1995; 11: 245-249.
30. Guertsen W, Leinenbach F, Krage T, Leyhausen G. Cytotoxicity of four root canal sealers in permanent 3T3 cells and primary human periodontal ligament fibroblast cultures. Oral Surg Oral Med Oral Pathol Oral Radiol 1998; 85: 592-597.
31. Serper A, Ucer O, Onur R, Etikan I. Comparative neurotoxic effects of root canal filling materials on rat sciatic nerve. J Endod 1998; 24: 592-594.
32. Good DL. Effects of materials used in pediatric dentistry on the pulp: a review of the literature. J Calif Dent Assoc 1999; 27: 861-867.
33. Heys DR, Heys RJ, Cox CF, Avery JK. The response of four calcium hydroxides on monkey pulp. J Oral Pathol 1980; 9: 372-379.
34. Norrsells N. Aven svenska tandlakare tillats nu sedan EU-intradet att anvanda den effektiva N2-metoden for rotfyllig. Med denna metod kan 500 miljoner kr sparas arligen at patientena och lidandet minskas. Endod Sverige 2002; 5p.
35. Hensten-Pettersen A. Skin and mucosal reactions associated with dental materials. Eur J Oral Sci 1998; 106: 707-712.   

       ——————————————————————————————————

See also: Amin, O. M. 2004. Dental Sealant Toxicity:  Neurocutaneous Syndrome (NCS), a dermatological and neurological disorder.  Holistic Dental Association Journal (No. 1, Jan.):  1-15  http://www.holisticdental.org/.

See also: Amin, O. M. 2004. On the diagnosis and management of neurocutaneous syndrome, a toxicity disorder from dental sealants. California Dental Association Journal 32 (9): 657-663.

Joe’s Morgellons/ Mercury Article

I was directed to this article by Sidney and I highly recommend that all of you read it. It is the most complete analysis of the Morgellons of the past (1500’s and 1600’s) that I have seen. Terrific article Joe. I have two main thoughts right now having read the article (which I will re-read again later today – it’s that good).

  1. It seems that Joe did a lot of work and research concerning the old accounts of Morgellons and I am absolutely astounded that in the none of the medical literature can be found the mention of “Skin Crawling or Biting Sensations”.  Also, Morgellons was found almost primarily in infants and young children and occasionally women. I am reeling from these two observations right now and seriously questioning whether Morgellons of the past is our Morgellons. I have very serious questions as to how our crawling and biting sensations, which are among the most bothersome symptoms, were never mentioned in 200 years of medical history?
  2. While reeling from the first part of Joe’s article the second part did seem to explain how part one above can be true. Joe connects Morgellons (of the past) to Mercury contamination and explains why infants, children and women were more likely to be exposed. Joe is or rather “was” a Morgellons sufferer who was helped by Dr. Amin, someone I have quoted often on my blog. Dr. Amin works with what he calls Neurocutaneous Syndrome (NCS) which I have also written about. The interesting thing about the mercury aspect is that I grind my teeth at night (I need to go back to using my byte guard again) and that would no doubt release quite a bit of mercury as I have mercury fillings. I wonder if that is why NAC is helping me so much. I still have doubts about mercury and newer dental fillings being the cause of this however. But one cannot doubt that Joe’s better now and was helped by Detoxing. I wonder if Joe outlines his symptoms and his treatment anywhere?

So here is the article ==>  Joe’s Morgellons/ Mercury Article

Please, read the article and offer me your thoughts on Joe’s blog post. I am extremely curious to know your thoughts as my original assumption was that Morgellons is caused by a toxic condition. I hope to see some comments on this post.


[UPDATED]

Hell and Back Again

An Account of Morgellon’s Disease and Its Cure from a Former Sufferer

See Joes Personal Story Here ==> http://members.cox.net/llyee2/NCS_article_by_joe.pdf

Follow

Get every new post delivered to your Inbox.

Join 388 other followers