Archive for the ‘Toluene’ Category

“It sort of blew our minds,” Robinson said.

For the record I am not posting this as Chemtrail article, that is something that I consider on the “fringe” but don’t let that offend you, we all have to make our own choices. I try to keep this site rooted in the believable so as to lend credibility to our effort. That being said I often notice the skies clear in the morning and then totally clouded over by jet contrails by evening and wonder “Just how much polution we are being exposed to?” I know it wasn’t like this when I was a child.

Interestingly, our friend Toluene shows up here yet again.

PITTSBURGH – A ground breaking study from a U.S. university has shed light on how the sun transforms jet engine exhaust, potentially creating toxic particles, reported Friday.

Researchers have discovered that drops of oil created by idling aircraft engines can over time turn into tiny particles that can easily penetrate the lungs and brain.

The surprising discovery has been detailed in the latest edition of the Atmospheric Chemistry and Physics Journal.

In the first study of its kind, experts from the Carnegie Mellon University in Pittsburgh, Pa., collected pollution from an idling commercial plane as it operated at different loads.

When the jet operated at full power the emissions were composed of mainly solid particles, however when it idled on the runway they took on a different form—microscopic droplets.

When the exhaust was exposed to sunlight in a “smog chamber” a chemical reaction took place that saw the formation of toxic particles from the interaction between the oil and gases.

It was found that sunlight can generate 35 times more particles than were originally emitted from the jet’s engine and 10 times what had typically been predicted.

These particles can include compounds such as benzene and toluene, which are known to impact health.

“Smog chamber experiments indicate that photo-oxidation of aircraft emissions produces significant amounts of secondary PM [particulate matter], which, under typical summertime conditions, exceed the primary emissions within minutes of the exhaust leaving the engine.

Allen Robinson of Carnegie Mellon University said the results were “unbelievable.”

“It sort of blew our minds,” Robinson said.

The research is a step further in understanding how aircraft emissions can impact air quality.

Gulf Disaster, “Natural Barrier of Our Skin is Gone”

I think you’ll find this very interesting and can give you some insight on what is the cause of our condition. Watch the video …

The Intel Hub
Alex Thomas

We have heavily documented the sickness that has been caused by BP and their use of the toxic dispersant known as Corexit. We have produced photographs, eye witness accounts [See the June/July archives of our radio show], and multiple blood tests. The problem is that every time we receive more evidence it doesn’t indicate that the crisis is beginning to get better, rather it indicates that it is much worse.

Kindra Arnesen has battled BP and their toxic spraying from the beginning and has been a ray of hope for all. She has helped to expose the many cover ups perpetrated by BP and their puppets within the American government.

Last week, a radio broadcast by The Coastal Heritage Society of Louisiana, complete with a radio host broadcasting live from the hospital, created a tremendous uproar throughout the internet. Florida Oil Spill Law posted the audio which was subsequently picked up The Intel Hub and as well as many others. The audio indicated that all the members of the CHSL radio show were sickened, with health problems ranging from a staph infection to neurotoxin poisoning.

Naturally the radio broadcast left many unanswered questions and Project Gulf Impact has attempted to answer at least some of them. Gulf Impact has worked diligently to expose the disaster and this video is one of the most important they have ever produced.

This video really speaks for itself. Kindra, along with many other gulf coast residents have had their natural skin barrier DESTROYED.

Doctors in the area will not deal with these clear medical problems. Have they been bribed or do they simply not want to put their livelihood on the line for the sake of someone else s health? Some have tried to help while others have seemingly misdiagnosed people on purpose.




Who would allow such a sickening chemical to be used so close to the human population? Why are the people of the gulf being used as guinea pigs in an experiment sanctioned by elements of our own government?

Please Spread this article to everyone you know. This is extremely important information. The Gulf disaster is NOT over, in fact, the actual spraying of Corexit hasn’t even stopped.

Another Theory for Sufferers to Consider

Crawling Sensations, Fibers and Other Noted Morgellons Syndrome Symptoms: Another Theory for Sufferers to Consider

by Joseph Keleher

I am not a medical professional. Let me repeat that. I am not, never have been and have no desire to ever become a medical professional. I might best be labeled a reluctant participant, as I suffered from Morgellons Syndrome Symptoms.

I have done my best to document what I went through (see Keleher 2008 “Hell and Back Again”). I wrote about connections to mercury and these horrific symptoms. I’ve angered medical professionals and sufferers. I’ve lost some old friends. I’ve found some new friends. I’ve written, emailed, and spoke on the phone with others who suffer. I’ve cried a bunch and still do (I don’t exactly know why).

I know it is possible to get well. I know what I suffered from and what I think most are suffering from is mercury as a neurotoxin. It is treatable with chelation and changes in lifestyle.

What I share are my thoughts. Yes, it is speculation. It is theory related to symptoms I had and you may still have. In reading be warned there are many “I think”s and “I believe”s. It’s all theory; it may be right or it may be wrong. Still, I feel compelled to share.

The Cause

I believe the recent growing numbers of those suffering from Morgellons symptoms is due to two ingredients- toluene and mercury.

Toluene is found in glues, methamphetamines and cocaine. I believe the toluene found in dental adhesives, and connected to symptoms by Dr. Omar Amin, correlate with a surge of sufferers of these symptoms (and possibly via prenatal passage the exponentially growing number of children with autism, ADD and ADHD).

I think toulene interacts with mercury to allow passage of this toxin into the nervous system. Toluene may dissipate after carrying mercury into the system leaving deposits of mercury. The primary source of mercury in the mouth (though there are other sources causing mercury increases including some skin creams, coal burning power plants, and historic mining activities) decreases as secondary deposits in the nerves increase.

The associated symptoms of this neurotoxin can be viewed as occurring in stages with new symptoms added as secondary deposits increase. The stages, based partly on my own experience, but also drawing from the experience of many others, might look like: 1) depression and panic attacks (typical symptoms of neurotransmitter blockage), 2) bloating, heart palpitations, and exhaustion, 3) crawling sensations, 4) fibers and skin lesions….and so on.

All of the symptoms noted with Morgellons, except crawling sensations and fibers, have been shown to be associated with mercury poisoning; because of this, I want to share how I think these two symptoms form.

Crawling Sensations

In the earliest onset of the “bugs”, I had faint flea-like zips across my scalp. I believe this was mercury pathways forming within the nerve cell network under my skin.

As the secondary levels of mercury increased, the sensations of crawling began. My crawling always started each night at the rear, right occipital area of my scalp (in journaling, at that time, I referred to the spot on my scalp as the “Mother Ship”); this area correlates with the low spot of my skull when resting (I have a preference for sleeping on my right with my head cocked back). The extreme density of mercury I believe was pooling.

Crawling typically began as the sun was setting and there was a change in temperature. Within weeks, the crawling expanded to my groin and bottom of my feet. Eventually my whole body had the sensations. It was during this time that I was diagnosed with sheet mites and scabies (and found out about something called Morgellons).

I believe as levels of mercury further increased, the crawling became more pronounced and expanded throughout my body. The sensation was especially disturbing when it began in my ear canals and nostrils.

I think the crawling is due to the expansion and contracting of mercury within the network of nerves. I picture it as one spider-shaped cell after another filling and twitching as mercury moves through (causing a kind of “cartoon effect” movement); one sufferer described this observed movement as certain proof of parasitic infestation. I still get a faint twitch occasionally at the base of my feet (but it feels different and I suspect it relates to nerve repairs and not mercurial movement).

If the Morgellons symptoms occur in stages related to increases in mercury, then I arrived at one of the middle stages. I never found fibers projecting from my skin or had any lesions. I’m thankful for this.


I think the fibers are the result of repeated cell damage repairs from mercury expansion and contrasting through the nerve system. A weak spot may have mercury break through and this will “scab” over. The fibers eventually poke through as they have nowhere else to go.

Final Words

I know there are many theories out there. Mine is one among many. Take it as such.

Morgellons is an especially cruel condition. In addition to the symptoms, the medical community has not been especially kind. The sensationalism of the media doesn’t help. Isolation and lose of connections to other people is difficult. There is one thing though that especially irritates me; I cannot stand that there are people taking advantage of the vulnerability of many sufferers. For those snake oil sales people making money off this condition, someday you will get yours; it is true what we do to others we do to ourselves.

For those suffering, be careful of the sharks, keep faith that answers will come soon, and always, always keep an “attitude of gratitude” for all you have. You are not alone and things will get better! I wish each and ever one of you a clear path towards health.

Thank you so much Joseph for another excellent and thought provoking article. Your thoughts really make me wonder if the NAC I take is a major player in my feeling better? Here are two other excellent articles written by Joseph that the reader might also want to examine.

Morgellons Disease – A Patient’s Perspective

Keleher 2008 “Hell and Back Again”

Neurocutaneous Syndrome (NCS)

I received permission from Dr. Omar M. Amin to republish this article. In some of my future blog posts I will be revisiting the subject of toxicity again. I have made reference to NCS in several posts in the past especially in connection with Toluene and other compounds.


On the Diagnosis and Management of Neurocutaneous Syndrome (NCS)
A toxicity disorder from dental sealants

Omar M. Amin, B.Sc., M.Sc., Ph.D.*
*Parasitology Center, Inc., 11445 E. Via Linda, # 2-419, Scottsdale, AZ 85259-2638  USA
Phone: 480-767-2522; Fax: 480-767-5855;  E-mail:
Web address:



Neurocutaneous syndrome (NCS), a newly discovered toxicity disorder, is characterized by neurological sensations, pain, depleted energy and memory loss as well as itchy cutaneous lesions which may invite various opportunistic infections. Components in the calcium hydroxide dental sealants Dycal, Life and Sealapex have been identified as sources of the observed symptoms. Sulfonamide and neurological toxicity issues are discussed and three case histories are presented. Additional notes on zinc oxide, Fynal, IRM and Sultan U/P sealers are also included. Diagnostic and management protocols at the Parasitology Center, Inc. (PCI) are proposed.


The original description of the neurocutaneous syndrome (NCS)1 was introductory in nature.1 Examination of many NCS patients and a careful study of their symptoms, exposures, clinical conditions and histories made it possible to identify the underlying cause of the syndrome and proceed with its management.


Materials and Methods

Patients were personally evaluated and their clinical history, records, symptomology and exposures carefully examined. Specimens provided or collected at the Parasitology Center, Inc. (PCI) were studied. An NCS status was only determined based on symptoms and determination that one or more of the suspect sealers have been used on prior dates. Sensitivity to sulfa and elevated levels of sulfa in the blood were used as a confirmation of sulfonamide toxicity. Continuing patients follow our recommendations for dental rehabilitation, extraction of suspect liner(s), and replacement with ethyltoluene sulfonomide (ETS) and zinc oxide free sealants. A list of vitamin/mineral supplements for patient use during the transitional period and another list of substitute sealants are provided.  Patients are followed up to monitor and insure the resolution of  symptoms.


Results and Discussion


The Neurocutaneous Syndrome

The disorder is double faceted with dermatological and neurological symptoms compatible with classical sulfa toxicity. The latter is characterized by changes in blood values, photosensitive reactions, allergic vasculitis sores, bacterial flora changes, and redness of the skin, which may lead to liver and kidney failure.2 The neurological aspects are characterized by pin-prick and/or creeping, painful and irritating movement sensations, often interpreted as parasite movements in various body tissues and/or cavities.. Movement sensations are either unipolar or bipolar and may proceed horizontally or vertically. They may manifest as variably shaped bruises or waves of elevated ripples or channels. In no case was the movement sensation related to parasites1. Neurological symptoms may also include loss of memory, brain fog, lack of concentration and control of voluntary movements.


Fig. 1. Early NCS sores on the thigh of KM. She was born in 1964, treated with Dycal in two teeth in 1982 and in one tooth in 2002. Neurological symptoms in upper quadrant started in 1997. Cutaneous symptoms began in Spring 2002 preceded by extensive treatment with topical sulfa preparations for possible mite infestation. Dycal was removed in December, 2002 and recovery is in progress.

The cutaneous aspects include small itchy sores (Fig.1), inflamed often elevated pimples (Figs.2,3), and fully inflamed and painful open/amorphous mucoid lesions that often enlarge and coalese (Fig.4). Histopathological sections of lesions (Fig.5) show superficial and deep perivascular infiltrate of lymphocytes, accompanied by interstitial deposits of granular mucin material. Eosonophils are usually present within the inflammatory infiltrate and foci of epidermolytic hyporkeratosis are often identified within the epidermis (Fig.5). Lesions may also be on the scalp where they may be associated with infestation of springtails (Collembola). 1 In many cases, lesions are associated with edematous reaction usually in the arms and legs (Fig.6). Blood vessels may also become enlarged and elevated, and head may become hot and turn red. The gum tissue and the teeth and oral mucoid secretions may turn gray and become compromised first and stay compromised the longest. The above creeping sensation is clearly distinguished from these caused by nematodes such as Toxocara canis3 or Dioctophyme sp.4

General symptoms usually include fatigue, compromised immune system, psychological trauma and loss of self- esteem. The depressed immune status in most patients appears to pre-empt them for opportunistic infections.


Compounding Factors


Fig. 2. Elevated sores on the forehead of KM (Fig.1); note the hot red color of the skin.


Fig. 3. Diffuse NCS sores covering the whole body that was treated with Dycal in 1985 (Case no. 1)

While NCS itself is not a contagious condition, superimposed opportunistic infections on open sores may be. Initial infection with fungus or bacteria appear to attract subsequent infestations with many arthropod species, especially springtails (Collembola: Insecta).1,5,6,7 Black specks associated with such infections appear to be metabolic waste (fecal elements) of these organisms or mycelial masses of certain fungal species. Staphylococcus aureus, S. haemolyticus,  Streptomyces spp., Candida albicans and Madurella spp. among others, have been identified from cultured swabs taken from sores of various NCS patients. These opportunistic infections have been shown to aggravate the cutaneous symptoms of NCS patients. The Madurella infections are usually associated with black grains of mycelial masses that may be related to the black specks and fibers observed by some NCS patients. The healing of certain patients lesions9 was observed to be proportional to the exit of remaining fibers from lesions.3 Patients experiencing complete remission remain susceptible to fungal promoting conditions in damp, shaded, moldy places.

Arthropods identified from sores include fleas, caterpillars, wasps, ants, beetles, winged flies, midges, thrips, ticks, mites, spiders, and springtails.1,4 Springtails may have close association with sores in many NCS patients but they, and other opportunistic infections, are not causal factors of NCS sores.


The Sealants

The three major calcium hydroxide sealants causing NCS (Dycal, Life and Sealapex) considered 9 include only about 50% calcium hydroxide in the catalyst (Table1). Of the components common to all three sealants, ethyltoluene sulfonamide as well as zinc oxide are considered most toxic. Toluene is a known potent nerve toxin.10 The sulfonamide component of this compound causes a sensitivity allergic- toxic reaction ultimately manifesting as the vascular mucoid sores characteristic of the NCS, especially in sulfa sensitive patients.




Fig. 4. Mucoid NCS/lesions on the face of MM.  She was born in 1950, poisoned with Fynal in six teeth in 1981 and in one tooth in 1986 as well as with Life in two teeth in 1985 and 1988.

Fig. 5. Histopathological section of one of the roughly 300 sores covering the body of SK. She was born in 1956 and reacted with typical NCS symptoms to a  zinc oxide cement (combined with Durelon) underneath a total veneer job in 1982. The section shows hyperkeratosis like perivascular dermatitis with eosinophils.

Fig. 6. Cutaneous sores and swelling in the right hand and arm of DB. Born in 1965, DB had 10 amalgam restorations in 1982 and 1983 using Life. She started experiencing symptoms including ulcerated rash all over the body, unilateral edema and pin-prick and subcutaneous movement sensations in 2001-2002. Life is being removed and recovery is in progress.

Zinc oxide was shown to be genotoxic11, cytotoxic12,13, killing microphages14, and causing chronic and fibrous inflammatory reaction15,16 ulcerations16 and osteosclerosis.17 Additionally, the toxic effects of zinc oxide and calcium hydroxide were shown to be similar.18,19 Calcium hydroxide was shown to cause periapical inflammation, typical granuloma and partial lack of healing.20 Titanium dioxide and Barium ions (Table 1) were also shown to provoke strong foreign body and bio-incompatible reactions in live tissue.21,22

Cytotoxicity of Dycal, Life and Sealapex was clearly demonstrated invivo and invitro in various tissues.23 Sealapex was shown to cause severe inflammatory infiltration15,24,25 and edema25 accompanied by subcutaneous tissue necrosis15,26 and progressive differentiation and reaction of monocytes, macrophages and epithelial cells27. The final phase of the inflammation is characterized by an intense granulomatus reaction especially in epithelial cells causing various intensities of irritation.28The cytotoxicity29,30 and neurotoxicity31 of Sealapex was well demonstrated in various mammalian systems.

As with Sealapex, Dycal was also shown to cause hemorrhage and acute to consistent inflammatory cells16,32,33 necrosis,16,32,33 tissue loss,33 karyorrhexis,16 neurotoxicity.34 and formation of serous exudates.16 Life has been the least researched sealant. It, however, has the same toxic ingredients, i.e., ethyltoleune sulfonamide and zinc oxide, as Sealapex and Dycal and has been associated with classical NCS symptoms in some of our patients, e.g., DB (Fig.6) and MM (Fig.4).

Sealants not containing ethyltoluene sulfonamide but including zinc oxide and eugenol have also been associated with NCS cases.These include Fynal(>75% zinc oxide), IRM and Sultan U/P (<50% zinc oxide). Fynal was associated with the cases of MM (Fig.4).  Similarly, IRM (by Dentsply caulk) and Sultan U/P (by Sultan Chemists) were associated with classical NCS symptoms in some of our patients.



Case Histories

Case #1.

    A white female born in 1951. In 1985 she underwent dental repairs, which included the use of Dycal in 20 teeth. The lady is allergic to sulfonomides, with IGE values reaching 5000. Every dental treatment was followed by aggressive skin reactions of allergic and toxicological nature (Fig.3). All tests for parasites were negative. Her symptoms fulminated into full blown typical sulfa toxicity reactions including oozing skin and nasal sores with bloody scabs and smelly discharge and an infection with S. aureus ( Fig.7). Other symptoms included loss of memory, kidney pain and urgency, sensitivity to light and electricity fields, pin-prick and moving sensations under the skin, and swelling. After each treatment, the white female felt totally knocked out with breathing and talking difficulties. She subsequently developed intestinal problems and her skin sores flared up with unbearable and unresolved itching. Photosensitive reactions presented as blotchy skin ( Fig.7) with severe burning sensations in the face, throat and chest.

       Dycal was removed in 1991-1992 and initially replaced with Harvard cement. The lady was confined to bed with whole body musculo-skeletal system pain, bowel disturbances and signs of polyneuropathy. Shortly after the removal of the Dycal in February 1992, most of her sores and rashes disappeared and she could tolerate sunlight (Fig.8).



Fig. 7. Case no. 1 before treatment; note the hot red face.

Fig. 8. after recovery.


Case # 2.

       Born in Chicago in 1965, JM was a healthy active Caucasian woman until she started experiencing her first symptoms in 1991. By then, she already had 17 fillings. No sealants were used in one filling; Dycal was used in the other 16. Her earliest symptoms appeared as skin break outs on the face and neck, which was recurrent over the following 9 years, accompanied by body tremors, sleeplessness and joint pain with occasional vomiting of black bile. Thrush appeared in the mouth and around the lips. Pain at the teeth roots persisted throughout the nineties associated with rapid major decay. A sensation of prickling pain with a pressure and movement under the skin, urticaria and skin ulcerations would last for weeks or months. JMs body showed random swelling with red marks in serpentine-like shapes. The swellings eventually bottlenecked at the knees and ankles. The chest burned and hurt with strange fits of coughing. JM then started losing hair as she experienced night fevers and sweats, and peeling of the skin.

       During the early 1990s JM was medicated with various antibiotics, antiparasitics and herbal remedies. She experienced some anti-inflammatory relief and occasional temporary clearing of ulcers after which ulcers returned and lasted longer. In 1998, massive ulcers appeared on JMs face at the nasiolobial area and on the skin ( Fig.9). A CBC in 1999 was unremarkable except for a high level of Alpha 1- Globulin of 0.5 (Normal range 0.2-0.4) and low levels of IgA of 99 (normal range 60-400) and IgG of 724(normal range 700-1500). The right ocular cavity was severely painful and JM was beginning to lose her eyesight.

     A major dental repair was completed in 2001 when Dycal was removed from all 16 teeth. Initially, JM experienced a few episodes of sickness, sweats, and vomiting. After the fourth visit, her eyebrow area had a dramatic reduction in swelling, sensation of movement and in the red-hot congestion of her face. JMs teeth were subsequently rebuilt with gold onlays section by section. By the end of the total repair, Nov.2001, JM has regained her normal skin (Fig.10) with no movement sensations or pain anywhere in her body. This state of total resolution has lasted to date without regression or relapses.



Fig. 9. Case no. 2 (JM) before treatment; note the lesion
on the right cheek and the hot red face.

Fig. 10. JM after recovery.

Case #3

       LG, a medium- built white American born in 1957, was in perfect health until September 18, 1998 when she had a filling in her tooth no. 18 using Dycal as a liner. She experienced severe headache within 2 hours. By 6:00 pm she was vomiting and delirious with the headache persisting. Her blood pressure then was monitored at 169/108 and remained high for the following three years despite repeated attempts to control it with Atenenol and Diazide. LG never experienced high blood pressure or headaches before. An MRI scan was negative. In 1999 LGs health deteriorated progressively with arthritis- like symptoms in her back, heart palpitations, mitral valve prolapse, fatigue, abnormal pap-smears including pre-cancerous cell abnormalities, night sweats, missed periods, and severe depression.  By March 2001, LG, who normally weighed 120 lbs has lost 20 lbs.

       In April 2001 lesions started appearing on LGs face, which quickly became red-hot.  Her legs became swollen and painfully burning. By May 2001, LG had several open lesions (6 mm to 2 cm in diameter) with some surrounding erythema, on her face and scalp. Her cheek pulsated as the facial lesions seemed to track to the chin (Fig.11) where the most fulminating lesion was; nearest to her teeth. The face was burning hot. Springtails (Collembola) and fibers were recovered from these sites. At that time, she showed low lymphocytes of 15.0% (normal 20-43%), high granulocytes of 77.1% (normal 51-74%) and high rheumatoid factor of 22.6 (normal <20 IU/ml). She also tested negative for all communicable diseases then. Her weight dropped to 92 lbs as she started experiencing movement sensations under the skin of her arms, face and scalp. Grayish pustular secretions oozed and moved down from the bloody lesions on the scalp and face. The lesion then extended to her legs.

       In January 2002, LG was diagnosed with NCS by OMA. She was allergic to sulfa and sulfonamide compounds. Following our protocol, LG had the filling and the Dycal liner removed from tooth #18 in April 2002. These were replaced with Starflow and Aria (a combination of Bisgma, Tegdma, Lidma and catalysts). Our recommended vitamin supplementation program was initiated then. By May 2002, all symptoms were resolved (Fig.12). Constitutional and neurological functions as well as psychological, emotional and energy levels were restored to normalcy.



Fig. 11. Case no. 3 (LG) before treatment.

Fig. 12. LG after recovery; note the return of the natural
baby skin back after healing of all facial lesions.



The toxicity of Dycal, Life and Sealapex has been well demonstrated in invivo and invitro studies of various animal and human models by many workers. The toxicity assumed cytotoxic, genotoxic, neurotoxic, phototoxic, necrotic, and inflammatory manifestations compatible with the pathology and symptoms observed in NCS patients. Ethyltoluene sulfonamide, common to all three sealants, is considered the primary cause of the NCS. The toluene component, a known nerve toxin, is believed to be responsible, at least in part, for the neurological symptoms. Neurological abnormalities are related to nerve damage associated with vasomotoric reactions due to a direct influence on the peripheral nerve endings.35 The sulfonamide component is the cause of the cutaneous symptoms, especially in sulfa-sensitive patients who usually had elevated sulfonamide/sulfa levels in blood tests and allergy to sulfa in skin sensitivity tests. The relationship between sulfonamide and phototoxicity has been well established.29 Resolving the symptoms (effect) by removing the sealants (cause) in patients undergoing treatments, confirms this cause-effect relationship.

The nature of causation of NCS precludes contagious transmission. Any similarities of symptoms among partners within the same household are traceable to the transmission of opportunistic infections, especially fungi.

It is recommended not to rehabilitate more than two or three teeth per month. The patient is given a list of vitamins and other supplements to take during the procedure and for the following few weeks until symptoms are completely resolved. After reaching the state of normalcy, the patient may still retain some sensitivity to moldy places lacking sun and fresh air circulation.

After additional test results become available and a satisfactory diagnosis of an NCS case is made at the Parasitology Center, Inc. (PCI), arrangements for dental rehabilitation are made and patient prognosis is monitored.


      I am grateful to Marie Erixon, Nordea, Sweden for her contributions to the better understanding of issues related to NCS.


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33. Heys DR, Heys RJ, Cox CF, Avery JK. The response of four calcium hydroxides on monkey pulp. J Oral Pathol 1980; 9: 372-379.
34. Norrsells N. Aven svenska tandlakare tillats nu sedan EU-intradet att anvanda den effektiva N2-metoden for rotfyllig. Med denna metod kan 500 miljoner kr sparas arligen at patientena och lidandet minskas. Endod Sverige 2002; 5p.
35. Hensten-Pettersen A. Skin and mucosal reactions associated with dental materials. Eur J Oral Sci 1998; 106: 707-712.   


See also: Amin, O. M. 2004. Dental Sealant Toxicity:  Neurocutaneous Syndrome (NCS), a dermatological and neurological disorder.  Holistic Dental Association Journal (No. 1, Jan.):  1-15

See also: Amin, O. M. 2004. On the diagnosis and management of neurocutaneous syndrome, a toxicity disorder from dental sealants. California Dental Association Journal 32 (9): 657-663.

Morgellons and More Cases of Collembola Infesting Man

I really do feel that Collembola do play a role in our itching and crawling sensations. In fact, I believe they are responsible for the majority of my crawling and biting sensations at this point. If I am correct, and I’m not sure I am, are they here to feed on fungi and or bacteria in my body? As I stated in  my very first post “It would seem the skin and body of the Morgellons sufferer has become like a rotting log, or the very least as favorable a home to both soil bacteria and soil based pests that they are both perfectly at home living in us”. However, not covered in this post are the fungi and bacteria that are found in the gut of the Collembola, could they set this whole thing in motion in us? Some are quite pathogenic. Just thinking out loud here.

I have a FIR (Far Infrared) heating pad that I purchased through Dr. Staninger and when I break out in these red pimple like things the FIR heatpad makes them go away and my skin returns to healthy looking very quickly, but it wasn’t cheap, around $350.00 if I recall. I don’t urge you to run out and buy one as it might not do the same for you and I know how much so many of you have already spent on things that haven’t worked. I have been considering lately as to whether or not the heating pad is killing them.

This post is a follow up to my first post regarding Collembola titled “Collembola – A Major Role in Morgellons Despite the Disinformation” and I hope you find this post interesting as well. I have found even more interesting information regarding Collembola. Here is a quote from

They are generally small and some species of Neelidae (Collembola) are among the smallest hexapods in the world at just over 0.2 mm long (about the same size of the period at the end of this sentence) while the largest Collembola are in the family Uchidanuridae which can reach 10 mm in length. Most species live for a year or less, however some live considerably longer and the record for long life in the laboratory is 67 months for a specimen of Pseudosinella decipiens.

Most Collembola feed on the fungi and bacteria found in rotting organic matter but many arboreal (living in trees) and epidaphic (living on the surface of the soil) species also feed on algae. Some feed on other plant materials and in some places particularly Australia Sminthurus viridus is a pest of lucerne crops. A few other species are carnivorous feeding on Nematodes and other Collembola.

Note that above text mentions that some species of Collembola can be as small as .2 mm.  Just so we are clear on things .2 mm is very small, .2 mm = 0.000787 of an inch !!!!! There are an estimated 30,000 to 100,000 undiscovered species of Collembola.



So How Small Can Insects Be Anyway?

fly The smallest insects are fairy flies, which are insects that parasitize other insects’ eggs by laying their eggs inside them. Fairy flies are only 1/5 of a millimeter in length. Many beetles are less than one millimetre in length, and the North American Feather-winged Beetle Nanosella fungi, at 0.25mm, is a serious contender for the title of smallest insect in the world. Other insect orders which contain extremely small members are the Diptera (True Flies) and the Collembola (Springtails). The “feather-winged” beetles and the “battledore-wing fairy flies” are smaller than some species of protozoa (single cell creatures). Megaphragma caribea a hymenopteran parasite from Guadeloupe, measuring out at a huge 0.17 mm long is in contention for the smallest insect.

See for more information. We are talking about flies here that you probably couldn’t see with your naked eye, amazing.


Okay, Back to Collembola

My first post talked about the National Pediculosis Association’s (NPA) study where they identified Collembola in 18 of 20 individuals studied, and also the 1955 cases is Sweden (there were many), and finally the 80 year old Woman in Romania. I would like to thank Sidney, one of our fellow readers and somebody that knows a lot about this subject. She pointed out to me that there were some mistakes in the identification in the samples of the 80 year old woman. She has first hand knowledge of this case.


rotifersSidney stated:


When Neculai removed the Collembola and Rotifers from the body of the 80 year old Romanian woman please bear in mind Neculai was not an entomologist, but a veterinarian pathologist and head of the department of veterinary medicine at his university. This was not a case of “sample contamination.” The Collembola was removed from the Romanian woman’s tissue by a needle aspiration biopsy. The corrections I mentioned in my email refer to the PUPA and LARVAL stage which Collembola do not have. Rotifers were also found within the Romanian woman’s skin (actual photos shown left), along with bundles of fibers.


I didn’t include the pictures of the fibers that Sidney sent to me but suffice it to say they look like our typical fiber bundles. So, some of the images that were put forth as various stages of the adult Collembola on my first post were actually found to be Rotifers.  If you really want to know more about Rotifers see this article “Epizoic and parasitic rotifers”. There is a interesting quote from the parasitic rotifer article (but you should click on the link and read the entire abstract).


There appear to be few records of epizoic or parasitic rotifers among vertebrates, apart from Encentrum kozminskii on carp, Limnias ceratophylli on the Amazonian crocodile, Melanosuchus niger, and an unidentified Bdelloid apparently living as a pathogenic rotifer in Man.

So, I’ll bet you can sense yet another blog post coming on Rotifers in the future and you would be correct, but for this post we are going to stay on the topic of Collembola.


More Cases of Human Collembola Infestation

collembola1 Let’s start with Dr. Omar, M. Amin, Ph.D., founder of PCI, who is a Professor of Parasitology and who works with something he calls Neuro-cutaneous Syndrome (NCS) which to me at least, seems to be exactly the same thing as Morgellons, though he might differ with me on that. The photo shown on the left is of a springtial (Collembola) that was taken from an actual patient. If you really want to dive into some of Dr. Amin’s information see this PAGE and follow the links on NCS. And if you hit this PAGE look at Table #2 and notice how many were documented to have springtails (another name for Collembola).

Here is a quote regarding the Collembola shown above from Dr. Amin’s site.


Scalp lesions also occur in patients with neurological symptoms and are usually associated with arthropod infestation.  JH (a tall, healthy, well-nourished, middle aged white American female from Arizona) had a number of such lesions (Fig. 4) from which springtails (Collembola: Insecta: Arthropoda) (Fig. 5) were collected by myself in December, 1995.  There is only two other published reports of springtails from humans (Hunter et al., 1960; Scott et al., 1962).


Very interesting. Dr. Amin mentions that there are only two other published reports of springtails in humans. However, he doesn’t mention any of the cases in my first blog post, nor did I mention the cases he is referring to (Hunter et al., 1960; Scott et al., 1962). But as you are about to find out, we are just getting started. We are very grateful for your work Dr. Amin, you provide an amazing amount of information to aid in our search on your site.


Collembola causing itching, biting, irritation, and papules …

Pescott, R.T.M. (1942:68-69) Australia:
“In 1939, specimens of springtails were received from a Melbourne specialist who stated that they were causing skin troubles on a female patient. The insect in question was the species Entomobrya multifasciata Tull., a European species originally described in 1871, but which is now cosmopolitan in its distribution. Womersley (3) records it as being common in cultivated areas in the Australian States. The symptoms of this case were as follows : the patient experienced a sharp biting sensation, followed by intolerable itching. There were few marks on the body with an occasional excoriated papule, while the irritation was distributed fairly generally over the trunk and limbs, but was most marked around the waist. Several specimens of the insect responsible for the condition were found on the patient’s body. She received no active treatment, but her clothes and bedclothes were sterilised and this was sufficient to destroy the insect and thereby remove the irritation.
On considering the origin of this infection, it appeared that the patient had recently moved into a new house where the garden was in the process of being made. The insects had apparently migrated to the patient when the grass, weeds and soil outside were disturbed.”

“In 1941, specimens of another springtail were received from a military hospital in Victoria, where skin irritations were occuring among the nursing staff. The species concerned was Entomobrya tenuicauda Schott., a native insect originally described in 1917 from Queensland, later recorded by Womersley (3) from Western Australia and Tasmania, and now from Victoria. In this instance, the presence of the insect produced on several nurses raised lumps very similar to mosquito bites, and which later were very irritable. In one instance there was also a good deal of reddening of the calf of the leg. These conditions lasted for somewhat less than twenty-four hours in each case, but reoccurred the next day, probably from more ‘bites’. On analyzing this case, it appears certain that the insects were introduced into the hospital with flowers, and from there moved on the affected nurses during their normal routine duties.”

Womersley suggested that the easily detached, long ciliated hairs of Entomobrya species undoubtedly would cause skin irritations.  Pescott concludes that severe skin irritation can be caused by certain species of Collembola: “Typical symptoms are a biting sensation, followed by intense irritation and the production of small pimple-like bodies.

Mackie, T.T., Hunter, G.W. & Brooke Worth, C. (1945:541-542) Australia:
“The Collembola are primarily phytophagous and are not usually thought of as medically important insects. Two Australian species, however, Entomobrya multifasciata Tullb. and E. tenuicauda Schott have recently been recorded as attacking man, the patients complaining of a sharp, biting sensation followed by irritation and papules similar to mosquito bites, with pruritus.”

Cited from Scott, H.G., Wiseman, J.S. & Stojanovich, C.J. (1962:430):
Entomobrya nivalis (cosmopolitan) and Entomobrya tenuicauda (Australasian) have been reported as causing a pruritic dermatitis in man.”

Cited from Ebeling, W. (1975):
“They [Collembola] have never been incriminated in the transmission of any human disease, but Entomobrya nivalis L., a cosmopolitan species, has been reported to cause an itching type of dermatitis in man, …”

Martini, M. (1952:354) cited from Bryk, F. (1955:1824) :
“Very discomforting mosquito-like skin irritations attributable to collembolans of the genus Entomobrya attempting to bite. “

Mertens, J. in Christiansen, K. (1998 in 2001:in litt.) Belgium:
” Several years ago our Faculty of Medicine once offered me ‘strange small insects’, which were considered as being responsible for causing allergic reactions on the skin of a woman. Those insects were Seira domestica. I could prove that the scales of Seira on the cushioned seats caused the allergy. As you know, Lepidocyrtus, has scales too. “

Mertens, J. (2004:in litt.) Belgium:
“In 1976 (or 1977), our Faculty of Medicine was puzzled by a rare case of skin allergy in a woman, living near Ghent. The allergy was caused by the scales of Seira domestica on a cushion of a rotan chair. Whenever the woman used the rotan chair, the allergic skin response occured (and only then). The chair was located in the veranda, which was quite moisty and where the temperature was enjoyable. It turned out that the hollow rotan branches of the chair hosted a population of Seira domestica. During the night, they left their hiding place and crawled all over the chair. The cushion collected many of the lost scales, causing as such the allergic reaction.”

Scott, H.G., Wiseman, J.S. & Stojanovich, C.J. (1962:430) Texas:
“Springtail insects (Orchesella albosa Guthrie, 1903, forma ainslieri Folsom, 1924) were found infesting the heads and pubic areas of a family in Buffalo, Leon County, Texas, in June 1961. No dermatitis was reported due to this infestation, and the source of the insects was not determined. Based upon known habits of this species, some moldy household item (perhaps bedding) was probably involved. Orchesella albosa has never before been reported infesting man or houses. Its chewing mouthparts are probably not capable of biting man.


At this point I’m going to stop referencing cases. There are many, many more on the following links at the end of this post that you will find extremely interesting. I wish I had time to do this post justice but I’m so busy just trying to be a dad, hold down a job, and stay well. However, if anyone is interested in finding out if Collembola can infest human beings the answer is on this post and in the following links.

One could make the argument, okay, so maybe you could dig up 30 or 40 cases of springtails in human tissue using the links below, but that’s not very many. And I would answer that with “Yes, but who is looking? We are told we are DOP without so much as an examination”


References (there is a wealth of case history on these links)

If you take the time to look at these links, especially the first two you will begin to realize there are a lot of references of human infestation of springtails (Collembola) in man. As as referred to above in the medical literature the patient experienced a sharp biting sensation, followed by intolerable itching. There were few marks on the body with an occasional excoriated papule, while the irritation was distributed fairly generally over the trunk and limbs it sure sounds a lot like us.


FOOTNOTE:  Dental Products Causing Neuro-Cutaneous Syndrome (NCS) Symptoms in NCS Patients


The toxic ingredients common to all belong in four major categories: Zinc Oxide, Ethyltoluene Sulfonamide (especially in patients with allergy to sulfa and toluene)

Dr. Amin feels strongly apparently that Toluene plays a major role in NCS and if you look at his site I don’t care what we call it, NCS and Morgellons exhibit the same exact symptoms. Most people when they think of removing fillings think of “Mercury Fillings” but the kind Dr. Amin is refers to here are the newer kind. You know, the new white ones that weren’t supposed to be bad for us, sigh …  I’ll be honest, I had quite a bit of dental work done before this all started, crowns and fillings and alike, all the new ceramic type too.

Grand Unification Theory – Sneak Peak

I haven’t had time to write up the entire “Grand Unification Theory” yet but I’ll give you a sneak peak. I happened to be doing more research when I stumbled upon something called the Toluene Pathway Map which shows how Toluene is broken down during biodegradation. Notice that Pseudomonas Putida is included in the map. Interestingly at the very bottom of the biodegradation map was something called Acetaldehyde which I immediately recognized from Jill’s post on BTEX. Toluene will eventually break down to Acetaldehyde . As I began to research Acetaldehyde it seemed to be linked to what I call the “Skin Crawling Family Tree”, that is, those that experience skin crawling sensations. Even more revealing the aldehydes were directly linked to some of the exact sensations that we feel, more on that later in this post.

The “Skin Crawling Family Tree”


All of the groups above suffer from Skin Crawling Sensations and the feeling of Bugs Crawling Under the Skin. What if we there was a common link between all of these groups? That is what this post is all about, I believe there is a common link, and that link is Acetaldehyde.

Meth Users

If you have been following my blog then you know how I have connected Morgellons to Toluene. If you haven’t read the previous posts click on the All Posts page and catch up, it should prove well worth your effort. In my Mr. Morgellons meet Mr. Meth, Your Distant Cousin I documented that Toluene is used in Meth production and they experience severe bug crawling sensations known as “Meth Bugs” or “Crank Bugs”. We know that Toluene breaks down into Acetaldehyde.

Cocaine Users

It turns out Cocaine users can also experience bug crawling sensations. From the article Cocaine and the Destruction of the Rainforests there is a quote

Annually, according to Peruvian forest engineer Marc J. Dourojeanni, coca growers dump 15 million gallons of kerosene, 8 million gallons of sulphuric acid, 1.6 million gallons of acetone, 1.6 million gallons of the solvent toluene, 16,000 tons of lime and 3,200 tons of carbide into the valley’s watershed.

For more on Cocaine and how it’s made see this DOCUMENT. We know that Toluene breaks down into Acetaldehyde.

Candida Sufferers

Acetaldehyde is the main waste product when Candida die-off occurs. For an interesting article see The Candida/Aldehyde detox pathway and the Molybdenum Connection for more information. The damage that Acetaldehyde causes to the body is tremendous and behind many other common diseases. Also, another good article is Candida Albicans and Acetaldehyde Toxin from which you will find this quote.

These effects of acetylaldehyde are multiplied many times over when candida die off occurs

Skin crawling sensations are common compliant for Candida sufferers. Also, this amazing quote:

The primary detoxification mechanism for scavenging unmetabolized acetaldehyde is sulfur-containing antioxidants [see Figure A]. The two most important are cysteine, a conditionally essential amino acid, and glutathione, a cysteine-containing tripeptide (a three-amino-acid polymer) [see Figure B]. Cysteine and glutathione are active against acetaldehyde (and formaldehyde) because they contain a reduced (unoxidized) form of sulfur called a sulfhydryl group, which contains a sulfur atom bonded to a hydrogen atom (abreviated SH).

Sulfhydryl groups interact with aldehydes to render tham incapable of forming cross links. This “mops up” or scavenges any stray acetaldehyde that is not properly metabolized into acetate (acetic acid) [see Figure A]. Although this is a powerful aldehyde detoxification mechanism, it is easily overwhelmed by the relatively large amounts of alcohol that are typically consumed with alcoholic beverages as compared to the amounts of alcohol and acetaldehyde that are produced through normal metabolism. Fortunately, sulfhydryl antioxidants can easily be fortified through dietary supplementation.

In one experiment with rodents [Sprince et al., 1974], a LD-90 dose of acetaldehyde (the dose that would normally kill 90% of the animals) was completely blocked by pretreatment of the animals with cysteine and vitamins B-1 and C. In other words, none of the cysteine-treated animals succumbed to the lethal dose of acetaldehyde! N-Acetylcysteine (NAC) protected almost as well as cysteine.

In another rodent experiment [Busnel & Lehman, 1980], alcohol’s ability to inhibit swimming after the alcohol had been completely metabolized was blocked by vitamin C. What this and the previous study suggests is that the pharmacologic and toxic effect of alcohol are different. The pharmacological effect (i.e., intoxication or drunkenness) is not inhibited by vitamin C or cysteine, but the toxic effect (e.g., the hangover, nervous irritability, swimming difficulty) is inhibited. This suggests that, with alcohol, you can “have your cake and eat it too.”

If that doesn’t sound exactly what we are going through with Morgellons I don’t know what does, notice how sulfur works so well.

Alcoholics going through DT’s

If you do some searching you will see that some alcoholics go through DTs so bad they experience the sensation of bugs crawling under their skin. It is my suggestion that this occurs when the massive Candida colonies they are maintaining go through die off 3 to 5 days after the alcohol flow is stopped. See Living with Alcohol for it’s link to Acetaldehyde.

Morgellons Sufferers

Like I have stated in previous blog posts many Morgellons patients have found high Toluene levels in their blood work and its metabolites such as Hippuric acid are found as well. Toluene will also biodegrade down the Benzoate path thus our glass crystals also (covered in another post). Much more can be said here. Of course, as this breaks down into the Acetaldehyde path and we suffer skin crawling just as the Candida sufferer does, it’s not the fungus in our case, it’s the Acetaldehyde being biodegraded from the Toluene in our bodies. I believe that Acetaldehyde causes the skin crawling sensation and not the actual fungus.


Formaldehyde (Tiny insects crawling over the eyes and nose?)

Let’s talk about a close cousin of Acetaldehyde and that is Formaldehyde. There is an amazing research paper out there on this, take a look at this quote:

Skin reactions: …chemical can be both irritating and allergy-causing…(EPA).  A slight sensation of tiny insects crawling over the eyes, nose and pharynx  (formication) is felt at 2-3 ppm.  (Zurlo N, via OSH, NZ.)  Contact with the vapour or solution causes skin to become white, rough, hard and anaesthetic due to superficial coagulation necrosis. With long exposure, dermatitis and hypersensitivity frequently result.  Prolonged exposure may also cause cracking of skin and ulceration, especially around the fingernails.

I wont bother to post all the links but I could find a thousand posts from Morgellons sufferers stating that they are sure they have bugs or worms crawling all over and in their eyes and around there nose, it’s a very, very common symptom. I too have felt that sensation on my eyes, like a bunch of mites crawling across the surface. Depending on what is laying around in our body I believe that Formaldehyde is often times created internally as well as Acetaldehyde.



This is just a very quick write up. I feel strongly that aldehydes such as Acetaldehyde  and Formaldehyde are the dominant causes of our symptoms. I’m glossing over a ton of evidence here such as the Putida and other things, I believe Toluene is being created in our bodies and am sure these are causing my symptoms.

Interestingly N-acetylcysteine (NAC) is great for binding to and ridding the body of Acetaldehyde  and so is Molybdenum, and Pantothenic Acid and Pantethine. However remember, I am no doctor. Also, cysteine is readily and abundantly found in human hair which is why I believe we suffer hair loss as the body is using all it can to bind to and rid us of the Acetaldehyde and I believe Formaldehyde is created also based on what chemicals we have laying around in our bodies at the time.

I think that all of the above groups in the “Skin Crawling Family Tree” suffer skin crawling due to the aldehydes being created in their bodies. I believe this could easily be proven. Something is creating Toluene in sufficient quantities in the human body to cause the Morgellons condition. We know Ps. Putida can create Toluene from glucose and that many alleviate their symptoms by cutting out most of the sugar from their diet. Is there a link here? There is a scientific paper out there which I will have to purchase to see the full text but I believe it links Acetaldehyde directly to skin crawling sensations in humans, we know it’s close cousin Formaldehyde already has been proven to do so.

Morgellons is an ongoing chemical reaction happening in our bodies with volatile organic compounds (VOC) such as Toluene. This is either being brought on by bioremediation bacteria which can create Toluene or the mere fact that Toluene is has now so thoroughly penetrated our environment. I hope this blog post spurs you on to do some research on Acetaldehyde and see if what I am saying is true. And if you find even more relevant information please post a comment on this thread.

Morgellons “Grand Unification Theory” Coming Soon …

Before we begin please remember that I could be wrong, do not get your hopes to high. Many have thought they too had this all figured out.

Who would have thought that when I asked Jill to make her guest post that she would lead me to the missing piece of information that would tie everything together? In fact, to her credit, Jill had this all gathered up, I am just going to take it to the next level and explain it all.

In my next post I am going present some information (following my current line of thinking) to you that is going to literally take your breath away when you read it. I believe the evidence I am about to present will make believers out of skeptics and cause those invested heavily in other theories to reconsider. The good news is if I’m correct, Morgellons isn’t some horrible hideous incredible disease (though the symptoms are) and even better news is that I believe there is a way out of this mess for all of us, and it will not take pharmaceuticals.

I’m terribly excited over what I have stumbled upon, or rather, been lead to. I’m sorry to post such a comment and know that many of you will be anxious to hear what I have to say, I promise you I will post the information as soon as I can. Also, the evidence I have isn’t conspiratorial or anything like that, it’s just something that ties Morgellons to all of our symptoms in such a way that it will be hard to ignore (and I believe this is an understatement).

Jet Fuel Germs, Clouds, and Bees – A Potpourri

In my last post titled “Could Morgellons be an Ongoing Chemical Reaction in our Bodies?” I spoke a little about jet fuel and how 100% octane jet fuel contains approximately 14% Toluene by weight. But what I didn’t know about was something called Biocides that are used in jet fuel to kill bacteria, yeast, and fungi. Now, I shouldn’t really be surprised after all of this research I’ve been doing lately.  If you’ve been reading along then you know about Pseudomonas Putida (the very versatile oil eating bacteria capable of all kinds of things) and so on but I never though that such things growing in the tanks and fuel lines of jets would be a real problem, but it appears that it is. I’m not sure this is of any significance but thought I would post about it just in case. This idea of germs in jet fuel was given to me by Clark, thanks Clark !

Twenty-seven (27) individual species of bugs can occur in diesel fuel

Now, diesel fuel is not jet fuel but I thought this list of “things” that can grow in it is interesting. This information can be found HERE.

Bacteria utilize hydrocarbons and reproduce ‘asexually’ by binary fission; swelling in size as they feed, they then separate into two cells. In this way microbes double their numbers every 20 minutes, one spore becomes 262,144 in 6 hours.  Typical bacteria known to utilise hydrocarbons are Pseudomonas aeruginosa, other Pseudomonas species, Flavobacterium spp., Acinetobacter spp., Alcaligenes spp., Micrococcus spp., Arthobacter spp., Corynebacterium spp., Brevibacterium spp., Klebsiella app.

Yeast bud onto the parent cell, then eventually separate. Reproduction takes several hours and yeast prefer acidy environments. Typical yeasts growing on hydrocarbons are Candida spp., Saccharomyces spp., Torula spp., Torulopsis spp., Hansenula spp.

Fungus grow in the form of branched hyphae, a few microns in diameter, forming thick, tough, intertwined mycelial mats at fuel/water interfaces. Typical moulds which degrade hydrocarbons are Penicillium spp., Aspergillus spp., Fusarium spp., Monilia spp., Botrytis spp, Cunninghammella spp., Scopulariopsis spp., Cladisporium resinae,  Hormonicus resinae.


If you recall my very first blog post titled “Does this identification mean anything? I do not know” focused on Pseudomonas Putida, one of the two forms of bacteria Dr. Wymore cultured off actual Morgellon fibers. The other one was Corynebacterium efficiens and I noticed that Corynebacterium spp appears in the bacteria list above, probably of no significance however.

What does fascinate me is all these things growing in fuel, including jet fuel. At first I wondered if these things could be raining down upon us but it’s probably very unlikely that they could survive even in fuel that might not be burning clean, however, anything is possible.


Biocides for Jet Fuel

From Fuel Quality Services, Inc.

Microbial contamination in aviation fuel systems dates back to the 1950’s. For over half a century fungi and bacteria have consumed kerosene type fuels and have demonstrated their ability to survive the freeze and thaw cycles of the aircraft fuel systems during flight. Microbes enter aircraft fuel systems as a result of poor housekeeping, contaminated hydrant systems, separators and storage tanks. Microbial contamination undoubtedly creates biomats that clog scavenge systems, coats fuel quality indicator system (FQIS) probes, and leads to structural corrosion.

I’m sure these folks make a fine product, I only present this information here to demonstrate that jet fuel, like diesel fuel is effected by bacteria, yeast, and fungi.


Are Bacteria Floating Around In and Falling From the Sky?

When I was originally researching Pseudomonas Putida I stumbled across some information about how Putida can actually be found high in our atmosphere among  the clouds. I’m not saying this is coming from jet fuel by any means, probably by simple evaporation processes.

Fluorescent pseudomonads isolated from Hebridean cloud and rain water produce biosurfactants but do not cause ice nucleation

Microorganisms were discovered in clouds over 100 years ago but information on bacterial community structure and function is limited. Clouds may not only be a niche within which bacteria could thrive but they might also influence dynamic processes using ice nucleating and cloud condensing abilities. Cloud and rain samples were collected from two mountains in the Outer Hebrides, NW Scotland, UK. Cloud samples were dominated by a mixture of fluorescent Pseudomonas spp., some of which have been reported to be ice nucleators.


I just thought this was interesting and would document it.


Bees, Collapsing Colony Disorder (or CCD), and TNT

I have been following the CCD syndrome these past few years and thought I would throw this into the mix. If you haven’t heard about CCD here’s a quick definition from wikipedia on CCD.

Colony collapse disorder (or CCD) is a phenomenon in which worker bees from a beehive or Western honey bee colony abruptly disappear. While such disappearances have occurred throughout the history of apiculture, the term colony collapse disorder was first applied to a drastic rise in the number of disappearances of Western honey bee colonies in North America in late 2006.


Here is a quote from the article Solving the Mystery of the Vanishing Bees on the seriousness of the situation.

One of us (vanEngelsdorp) performed autopsies on Hackenberg’s remaining insects and found symptoms never observed before, such as scar tissue in the internal organs. Initial tests also detected some of the usual suspects in bee disease. In the gut contents we found spores of nosema, single-celled fungal parasites that can cause bee dysentery. The spore counts in these and in subsequent samples, however, were not high enough to explain the losses. Molecular analysis of Hackenberg’s bees, performed by the other of us (Cox-Foster), also revealed surprising levels of viral infections of various known types. But no single pathogen found in the insects could explain the scale of the disappearance.

In other words, the bees were all sick, but each colony seemed to suffer from a different combination of diseases. We hypothesized that something had compromised the bees’ immune system, making them susceptible to any number of infections that healthy colonies would normally fend off.  And Hackenberg was right: the prime suspects, varroa mites, were not present in numbers significant enough to explain the sudden die-off.


TNT (Dynamite) – What could this have to do with Bees?

I thought I would bring up a subtle point about honey bees and TNT (dynamite) which is really an acronym for Trinitrotoluene (Tri-nitro-toluene).  Yep, there’s our old friend Toluene again.  Interestingly, they use honey bees to locate mines so they can be deactivated. The thing is, these bees think they are finding food, how interesting.


Look at this question posed by a beekeeper in Africa and his dilemma.

I am wondering if you can help. I am a small bee farmer in South Africa. I specialize in the control and removal of problem swarms. I have removed them from some of the strangest places. I have just had a request from a company to assist them at their factory. They process TNT and the bees seem to be attracted to it in a big way. Workers get stung on occasions while carrying the TNT and should they drop it, well you can just imagine. This has already happend on a few occasions with some serious results. The cardboard boxes which the TNT comes in, attract swarms once it is empty and discarded. 2 or 3 swarms are "caught" this way every week. I don’t have a problem with sorting out this problem, but it interests me to find out what is attracting the bees. This will also help in the solving of the problem.

Considering all of the Toluene pollution that could be found by bees one wonders if they are able to find it and thus mistake it as food and poison the hive? There is some incredibly bizarre stuff about how these bees abandon and hive, and no other bees will go in to raid the food, the bees leave their brood and even abandon the queen.


And finally, one more quote from WHY ARE THE BEES DISAPPEARING?

It is a fact that honeybees have been used to detect land mines because they are attracted to some component of the explosives used in the mines. They actually pick this material up and bring it back to the colony where it can be analyzed by the people doing the study. If bees are attracted to some component of explosives, isn’t it possible they are also attracted to some unknown (or unlisted) component of pesticides? The chemical structure of the highly dangerous explosive, TNT, or TriNitroToluene, which is used in land mines is benzene and a methy group of chemicals.

Benzene and toluene, two components of TNT are also inert ingredients used in many pesticides. I believe the bees are attracted to pesticides containing these inert ingredients as they are to TNT. If so, the bees may be attracted to trees and shrubs that are being sprayed and will be more inclined to pick up the pesticides and bring it back to their colonies where they can contaminate many more individual bees as well as the honey in the hive.


Now, I know this is pretty anecdotal and might give you cause to laugh but how many sufferers have themselves thought “If only I can move, this stuff is all over my house, my yard. I’m out of here?” In fact, I know many that have, and even a few that abandoned their homes to live in tents.  Could the immune systems of the honey bees actually be being destroyed by them finding sources of Toluene and bringing it back to the hive? Is what is happening to the bees similar to what is happening to Morgellons sufferers? You know how they make honey, here’s how (HERE).

Finally, I’ll leave you with a bee joke that will amuse you.

A bee walks into a doctors office and says to the doctor … “Doc, I’m covered in varroa mites, I can feel them crawling all over me and then he opens a matchbox full of them to show the doctor. The doctor smiles and says “Have you been reading that darned internet again …..”

Sorry for such a strange set of topics tonight everyone. Just getting some thoughts down on paper …

Guest Post by Jill on MTBE and Related Info

I asked Jill who has been working on the connection between Morgellons and MTBE and related compounds to prepare a blog post for me, here is her post, I hope you find it valuable. MTBE is is gasoline additive.

Mr. Common Sense,

Good work on the research! Toluene- most likely one of the culprits. Found, as you point out, in various products, same as Benzene and many others. Many VOC’s (volatile organic compounds) are turning out to be a part of the process that is causing/creating emerging disease.

I invite all of you to visit/post my new board. Together- maybe we can get a treatment that works and hopefully a cure soon.

Per your request, I’m posting my info here- 1st time Blogger.

Take the chemicals- the MTBE/TBA (TBA is the daughter product of the MTBE- IOW MTBE DEGRADES
to TBA) both are carcinogens and they are the MOST COMMON POLLUTANT IN THE UNITED STATES WATER SUPPLY


Know- when you are researching MTBE- that BTEX is commonly found along with the MTBE.


Because both are VOCS- (Volatile Organic Compounds) found in gasoline and Petrol products:


From Wikipedia, the free encyclopedia

Fair use:


BTEX is an acronym that stands for benzene, toluene, ethylbenzene, and xylenes. These compounds are some of the volatile organic compounds (VOCs) found in petroleum derivatives such as petrol (gasoline). Toluene, ethylbenzene, and xylenes have harmful effects on the central nervous system.

BTEX compounds are notorious due to the contamination of soil and groundwater with these compounds. This typically occurs near petroleum and natural gas production sites, and petrol stations and other areas with Underground Storage Tanks (USTs) or Above-ground Storage Tanks (ASTs) containing gasoline or other petroleum-related products.

The amount of ‘Total BTEX’, the sum of the concentrations of each of the constituents of BTEX, is sometimes used to aid in assessing the relative risk or seriousness at contaminated locations and the need of remediation of such sites. Naphthalene may also be included in Total BTEX analysis yielding results referred to as BTEXN. In the same way, styrene is sometimes added, making it BTEXS.

There are other substances found in the mix such as ACETALDEHYDE

This component is the key to the process of FERMENTATION in the body -

Fair use


Intestinal Yeast (candida albicans), and other fungal pathogens such as toxigenic molds, produce
the following toxins, which could be responsible for the symptoms and causes of Fibromyalgia.

This is not an all inclusive list.

1. Ethanol- an alcohol of intoxication mutates immune cells
2. Acetaldehyde 6 times more potent than ethanol, cell mutation ********
3. Tyramine interferes with immune function
4. Canditoxin interferes with and reduces immunity
5. Proteinase increases candida potency
6. Glycoprotein toxin interferes with immune function
7. Polysaccharride proteins reduces immunity
8. Histamine reduces immunity
9. Mycotoxins that can cause immune dysfunction and many other health problems


When thinking about ACETALDEHYDE- think about a bad hangover- that is the process that is
caused and the symptoms of the process – initially.


The Acetaldehyde (along with the other chemicals) cause the symptoms of Fibromyalgia (and more) found here: above link:

(Note- the Fibromyalgia symptoms overlap Lyme diseaese and CFS symptoms )

Partial list:

Symptoms of Fibromyalgia:

  • Chronic fatigue
  • Abnormally high pain sensitivity especially in the muscles, and joints (arthritis)
  • Insomnia or unrefreshing sleep
  • Chronic stiffness especially in the shoulders, back and neck
  • Reduced levels of Human Growth Hormone
  • Reduced levels of ATP (Adenosine triphosphate) resulting in low or no energy
  • Hormone and endocrine imbalances
  • Chronic toxin accumulation from the skin, lungs, intestines, blood stream and brain
  • Depressed immune function and activity

*The above symptoms can decrease immunity and allow or cause fibromyalgia to worsen, producing additional symptoms that include:

  • Depression and personality changes(6)
  • PreMenstrual Syndrome (11,)
  • Tension and migraine headaches (12)
  • Muscle trauma, and deconditioning (16)
  • Anxiety, sleep disorders (62), vertigo, apathy, mood swings and memory loss
  • Autoimmune disorders like chemical hypersensitivity, asthma, lupus and arthritis
  • Irritability
  • Mitral valve prolapse
  • Opportunistic bacterial, viral and parasite infections
  • Chronic fatigue immune deficiency
  • Irritable bowel syndrome (IBS) and leaky bowel syndrome
  • Myofacial pain syndrome
  • Hypoglycemia and inability to burn fat
  • Intestinal and food allergies
  • Possibly a traumatic emotional or physical shock triggers the manifestation of fibromyalgia.

**see more symptoms here:


MTBE/TBA/BTEX and additional component/s such as ACETALDEHYDE create a process in the body

ACETALDEHYDE- produces the sugar process that produces Electricity in the body

ACETALDEHYDE- FERMENTS- creating yeast/fungus and causing pain per above

BENZENE- CAUSES CANCER- per the Cleveland Clinic research-

THE GEL some find- is produced by a process created by the cyanobacteria, commonly called blue-green algae-

A process like this one- a supposedly ‘new’ discovery:


University of Texas Professors R. Malcolm Brown and David Nobles Jr. created the cyanobacteria by giving them a set of cellulose-making genes from a non-photosynthetic bacterium called Acetobacter xylinum, which is commonly used for producing vinegar. The new cyanobacteria produce a relatively pure, gel-like form of cellulose that can be easily broken down into readily fermentable sugars. They also secrete sucrose and glucose, which are already prominent sources of ethanol, most commonly in the form of sugarcane and corn, respectively.

Recall that TT stated there was cyanbacteria found- and since I can remember, those in the study of the fibers/etc have stated that CELLULOSE was found.

Put together what the various researchers have FOUND and apply the above process- chemicals in the body from the various sources, the ACETALDYDE which ferments and causes the symptoms of the many EMERGING DISEASE (Lyme,Fibro, CFS, Etc) and you have the answer.

Yes there is more- but this is the basic process – cause and effect

All the symptoms can be found by a study of these chemicals and the process they create in the body.



Jill has some great information which you can find here

Could Morgellons be an Ongoing Chemical Reaction in our Bodies?

First, let me say that I am not a scientist, I am only grasping at straws here. I am not using proper scientific techniques to come to my conclusions and have no real idea if Toluene is the cause of, or if it plays a role in Morgellons. I am only hypothesizing that it does because it seems to be able to cause so many of our manifestations. The real question is can these things I am describing go on “inside of the human body”?

Also, I made a mistake in my last blog post by stating that our fibers couldn’t be the result of nanotubes because things at the nano level are so small they cannot be seen by the naked eye. It was correctly pointed out to me that nanostructures are used to build things which eventually would become large enough to be seen with the naked eye. Thank you for that correction and it’s an important one.


Toluene – Some Definition From Wikipedia

Toluene, also known as methylbenzene or phenylmethane, is a clear, water-insoluble liquid with the typical smell of paint thinners, redolent of the sweet smell of the related compound benzene. It is an aromatic hydrocarbon that is widely used as an industrial feedstock (not animal feed) and as a solvent. Toluene reacts as a normal aromatic hydrocarbon towards electrophilic aromatic substitution. The methyl group makes it around 25 times more reactive than benzene in such reactions. Toluene occurs naturally at low levels in crude oil and is usually produced in the processes of making gasoline via a catalytic reformer, in an ethylene cracker or making coke from coal. Final separation (either via distillation or solvent extraction) takes place in a BTX plant.

Toluene is a common solvent, able to dissolve paints, paint thinners, silicone sealants, many chemical reactants, rubber, printing ink, adhesives (glues), lacquers, leather tanners, and disinfectants. It can also be used as a fullerene indicator, and is a raw material for toluene diisocyanate (used in the manufacture of polyurethane foam) and TNT. It is also used as a cement for fine polystyrene kits (by dissolving and then fusing surfaces) as it can be applied very precisely by brush and contains none of the bulk of an adhesive.

Industrial uses of toluene include dealkylation to benzene, and the disproportionation to a mixture of benzene and xylene in the BTX process. When oxidized it yields benzaldehyde and benzoic acid, two important intermediates in chemistry. It is also used as a carbon source for making Multi-Wall Carbon Nanotubes. Toluene can be used to break open red blood cells in order to extract hemoglobin in biochemistry experiments.

Toluene can be used as an octane booster in gasoline fuels used in internal combustion engines. Toluene at 86% by volume fueled all the turbo Formula 1 teams in the 1980s, first pioneered by the Honda team. The remaining 14% was a "filler" of n-heptane, to reduce the octane to meet Formula 1 fuel restrictions. Toluene at 100% can be used as a fuel for both two-stroke and four-stroke engines; however, due to the density of the fuel and other factors, the fuel does not vaporize easily unless preheated to 70 degrees celsius (Honda accomplished this in their Formula 1 cars by routing the fuel lines through the muffler system to heat the fuel). 

Toluene has also been used in the process of removing the cocaine from coca leaves in the production of Coca-Cola.

Toluene in Airplane Fuel

From TOLUENE – Some Basic Questions.

Question: Why do you think toluene is better than other types of octane boosters?

Answer : Several reasons:

Mindful of the evil reputation of octane boosters in general, toluene is a very safe choice because it is one of the main octane boosters used by oil companies in producing ordinary gasoline of all grades. Thus if toluene is indeed harmful to your engine as feared, your engine would have disintegrated long, long ago since ordinary pump gasoline can contain as much as 50% aromatic hydrocarbons.

Toluene is a pure hydrocarbon (C7H8). i.e. it contains only hydrogen and carbon atoms. It belongs to a particular category of hydrocarbons called aromatic hydrocarbons. Complete combustion of toluene yields CO2 and H2O. This fact ensures that the entire emission control system such as the catalyst and oxygen sensor of your car is unaffected. There are no metallic compounds (lead, magnesium etc), no nitro compounds and no oxygen atoms in toluene. It is made up of exactly the same ingredients as ordinary gasoline. In fact it is one of the main ingredients of gasoline.

Toluene has a RON octane rating of 121 and a MON rating of 107, leading to a (R+M)/2 rating of 114. (R+M)/2 is how ordinary fuels are rated in the US. Note that toluene has a sensitivity rating of 121-107=14. This compares favorably with alcohols which have sensitivities in the 20-30 range. The more sensitive a fuel is the more its performance degrades under load. Toluene’s low sensitivity means that it is an excellent fuel for a heavily loaded engine.

Toluene is denser than ordinary gasoline (0.87 g/mL vs. 0.72-0.74) and contains more energy per unit volume. Thus combustion of toluene leads to more energy being liberated and thus more power generated. This is in contrast to oxygenated octane boosters like ethanol or MTBE which contain less energy per unit volume compared to gasoline. The higher heating value of toluene also means that the exhaust gases contain more kinetic energy, which in turn means that there is more energy to drive turbocharger vanes. In practical terms this is experienced as a faster onset of turbo boost.

Chevron’s published composition of 100 octane aviation fuel shows that toluene comprises up to 14% alone and is the predominant aromatic hydrocarbon. Unfortunately composition specifications for automotive gasoline is harder to pin down due to constantly changing requirements.

We have all woken up to a beautiful blue sky morning only to have it turn totally overcast and hazy, caused only by  traffic patterns from the jumbo jets overhead, by two or three in the afternoon. I sure hope all that Toluene is truly turned into CO2 and H2O during combustion as stated above otherwise it’s raining Toluene. Nevertheless, all it takes is a search on Toluene and “Major Pollutant” to know it is a real problem.

Could Toluene Exposure be the Catalyst in Jumpstarting Morgellons?

The one thing I have noticed in my reading is that Toluene is very reactive to the chemicals around it and it can undergo all kinds of chemical changes that other chemicals like benzene cannot. It seems to be a “wonder chemical” and it is being used for all kinds of things. In this blog post we are going to look at two more symptoms that Morgellon sufferers experience that could also be explained by Toluene exposure. And toluene is everywhere, it is a major pollutant in our world, which is why Pseudomonas putida is being used to clean it up everywhere, from gasoline storage tank leaks and oil spills both on land and in the ocean.

Could Morgellons be in large part an ongoing chemical reaction in our bodies? Does this only happen to those who are carrying a heavy toxic load of Toluene? Or, could something, such as a bacteria that has the ability to create Toluene as a byproduct be entering our bodies and setting things in motion?

Finally, remember, I could be totally wrong here. Even though Toluene does seem to be found in Morgellons patients it is a major pollutant in our world and likely found in nearly everyone at some level.


Toluene, GS-15, and it’s Reaction to Iron = Magnetite

In my last blog post I posted a quote from an article which I will repeat here as well.

Nanotubes grown from pure toluene/ferrocene precursors appear to have a well defined morphology, with thick parallel walls and narrow inner core often filled with catalytic iron nanoparticles.

I find it interesting that the language used here indicates that it was an unexpected surprise that the inner core was filled with iron particles. Then in that very same blog post I go on to indicate how they are making magnetic nanotubes. Here is an interesting article that I found as well

Anaerobic Oxidation of Toluene, Phenol, and p-Cresol by the Dissimilatory Iron-Reducing Organism, GS-15

Growth coincided with Fe(III) reduction. [ring-14C] toluene was oxidized to 14CO2, and the stoichiometry of 14CO2 production and Fe(III) reduction indicated that GS-15 completely oxidized toluene to carbon dioxide with Fe(III) as the electron acceptor. Magnetite was the primary iron end product during toluene oxidation.

Let me try to restate that as best I can into layman’s terms.

Growth coincided with iron reduction. Toluene was oxidized to CO2, and the chemical reaction of CO2 production and Iron reduction indicated that the (bacteria – Geobacter metallireducens) completely oxidized toluene to carbon dioxide with iron as the electron acceptor. Magnetite was the primary iron end product during toluene oxidation.

Many suspect the Morgellon sufferer’s body is acting like a magnet and that magnetism is behind many of the sensations we feel. I find it interesting that Toluene can oxidize into C02 (just as the airplane fuel article above states) and that in the process it turned Iron into Magnetite. Magnetite is the most magnetic of all the naturally occurring minerals on Earth. Could this be happening inside our bodies?


Toluene Oxidation Forming Benzoic Acid Crystals

Many sufferers have no doubt experienced glass like crystals or particles coming through their skin, they can be very painful, some maybe be sharp and pointed or they may come in the form of glitter. Could this also be the result of Toluene? This possibility was pointed out to me by a member on the LymeBusters Morgellons forum.

From the Wikipedia link on Benzoic acid you can find some very interesting information including how its made.

Benzoic acid is produced commercially by partial oxidation of toluene with oxygen.

Take a look at this Benzoic Acid crystal from the wikipedia page.


Again I wonder if the glass like crystals many experience could be the result of some ongoing chemical reaction with Toluene in the human body. I myself have spoken to one person who is doing far better than he was but his main symptom now is these glass like crystals coming through his scalp, very painful he says, and no doubt it must be.

If you look at this LINK under Skin you will see this quote:

Objects described as "shards," hard and crystal-like that are similar to glass that emerge from skin

In fact, if you hit this GOOGLE SEARCH you will see this is not unusual for Morgellons sufferers.


Like I stated at the beginning of this blog post, Toluene seems to be able to do all kinds of things depending on what it comes into contact with. The real questions are could these things be ongoing within the human body? Could a Morgellons sufferer have some extra additive that has jumpstarted this chemical process or thereby taken advantage of our Toluene exposure? Is there something, such as a bacteria in our bodies that is creating Toluene as a byproduct?  Wouldn’t we need an ongoing source of toluene for all this to be happening?

Enough on this Toluene already!! I think it’s time to stop all this Toluene speculation and try to tie together all kinds of scenarios as Kixx has recently done on a reply to my last blog post. For example, “How could Toluene play a role in this when my crawling seemed to be tied to a certain event in time?” I admit I don’t have the answers. But it does seem to me that if someone were to analyze these crystals that come out of Morgellons patients and they turned out to be Benzoic Acid crystals we would have a smoking gun. The frustrating part is that there really is a tremendous amount of evidence that could be provided by Morgellons patients, and other than a few organizations and scientists there doesn’t seem to be anybody listening.

I think I have gone as far down the Toluene road as I can right now. I need to dwell on this for a while and see if I’ve been going down the right path. I do find it interesting that hard glass crystal structures emerging from the skin are a common symptom, and that toluene could indeed very easily create these.


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